-
1
-
-
79956300649
-
Toll-like receptors and their crosstalk with other innate receptors in infection and immunity
-
Kawai T, Akira S. Toll-like receptors and their crosstalk with other innate receptors in infection and immunity. Immunity. 2011;34:637–650.
-
(2011)
Immunity
, vol.34
, pp. 637-650
-
-
Kawai, T.1
Akira, S.2
-
2
-
-
35349016235
-
Recognition of microorganisms and activation of the immune response
-
Medzhitov R. Recognition of microorganisms and activation of the immune response. Nature. 2007;449:819–826.
-
(2007)
Nature
, vol.449
, pp. 819-826
-
-
Medzhitov, R.1
-
3
-
-
61849170035
-
TLRs and innate immunity
-
Beutler BA. TLRs and innate immunity. Blood. 2009;113:1399–1407.
-
(2009)
Blood
, vol.113
, pp. 1399-1407
-
-
Beutler, B.A.1
-
4
-
-
0036606913
-
Lipopolysaccharide recognition: CD14, TLRs and the LPS-activation cluster
-
Triantafilou M, Triantafilou K. Lipopolysaccharide recognition: CD14, TLRs and the LPS-activation cluster. Trends Immunol. 2002;23:301–304.
-
(2002)
Trends Immunol
, vol.23
, pp. 301-304
-
-
Triantafilou, M.1
Triantafilou, K.2
-
5
-
-
79954577277
-
TLR-signaling networks: an integration of adaptor molecules, kinases, and cross-talk
-
Brown J, Wang H, Hajishengallis GN, Martin M. TLR-signaling networks: an integration of adaptor molecules, kinases, and cross-talk. J Dent Res. 2011;90:417–427.
-
(2011)
J Dent Res
, vol.90
, pp. 417-427
-
-
Brown, J.1
Wang, H.2
Hajishengallis, G.N.3
Martin, M.4
-
6
-
-
77952316540
-
An integrated view of humoral innate immunity: pentraxins as a paradigm
-
Bottazzi B, Doni A, Garlanda C, Mantovani A. An integrated view of humoral innate immunity: pentraxins as a paradigm. Annu Rev Immunol. 2010;28:157–183.
-
(2010)
Annu Rev Immunol
, vol.28
, pp. 157-183
-
-
Bottazzi, B.1
Doni, A.2
Garlanda, C.3
Mantovani, A.4
-
7
-
-
0041534400
-
Collectins and ficolins: humoral lectins of the innate immune defense
-
Holmskov U, Thiel S, Jensenius JC. Collectins and ficolins: humoral lectins of the innate immune defense. Annu Rev Immunol. 2003;21:547–578.
-
(2003)
Annu Rev Immunol
, vol.21
, pp. 547-578
-
-
Holmskov, U.1
Thiel, S.2
Jensenius, J.C.3
-
8
-
-
80555149785
-
Pentraxins in humoral innate immunity
-
Inforzato A, Bottazzi B, Garlanda C, Valentino S, Mantovani A. Pentraxins in humoral innate immunity. Adv Exp Med Biol. 2012;946:1–20.
-
(2012)
Adv Exp Med Biol
, vol.946
, pp. 1-20
-
-
Inforzato, A.1
Bottazzi, B.2
Garlanda, C.3
Valentino, S.4
Mantovani, A.5
-
10
-
-
77950187449
-
Crosstalk pathways between Toll-like receptors and the complement system
-
Hajishengallis G, Lambris JD. Crosstalk pathways between Toll-like receptors and the complement system. Trends Immunol. 2010;31:154–163.
-
(2010)
Trends Immunol
, vol.31
, pp. 154-163
-
-
Hajishengallis, G.1
Lambris, J.D.2
-
11
-
-
77950189147
-
Complement activation by CpG in a human whole blood loop system: mechanisms and immunomodulatory effects
-
Mangsbo SM, Sanchez J, Anger K, et al. Complement activation by CpG in a human whole blood loop system: mechanisms and immunomodulatory effects. J Immunol. 2009;183:6724–6732.
-
(2009)
J Immunol
, vol.183
, pp. 6724-6732
-
-
Mangsbo, S.M.1
Sanchez, J.2
Anger, K.3
-
12
-
-
34347400796
-
Regulation of Toll-like receptor-mediated inflammatory response by complement in vivo
-
Zhang X, et al. Regulation of Toll-like receptor-mediated inflammatory response by complement in vivo. Blood. 2007;110:228–236.
-
(2007)
Blood
, vol.110
, pp. 228-236
-
-
Zhang, X.1
-
13
-
-
79952123931
-
Microbial manipulation of receptor crosstalk in innate immunity
-
Hajishengallis G, Lambris JD. Microbial manipulation of receptor crosstalk in innate immunity. Nat Rev Immunol. 2011;11:187–200.
-
(2011)
Nat Rev Immunol
, vol.11
, pp. 187-200
-
-
Hajishengallis, G.1
Lambris, J.D.2
-
15
-
-
24144465889
-
Molecular determinants of crosstalk between nuclear receptors and toll-like receptors
-
Ogawa S, et al. Molecular determinants of crosstalk between nuclear receptors and toll-like receptors. Cell. 2005;122:707–721.
-
(2005)
Cell
, vol.122
, pp. 707-721
-
-
Ogawa, S.1
-
16
-
-
2942754121
-
Cutting edge: physiologic attenuation of proinflammatory transcription by the Gs protein-coupled A2A adenosine receptor in vivo
-
Lukashev D, Ohta A, Apasov S, Chen JF, Sitkovsky M. Cutting edge: physiologic attenuation of proinflammatory transcription by the Gs protein-coupled A2A adenosine receptor in vivo. J Immunol. 2004;173:21–24.
-
(2004)
J Immunol
, vol.173
, pp. 21-24
-
-
Lukashev, D.1
Ohta, A.2
Apasov, S.3
Chen, J.F.4
Sitkovsky, M.5
-
17
-
-
84885442197
-
Inflammatory responses induced by lipopolysaccharide are amplified in primary human monocytes but suppressed in macrophages by complement protein C5a
-
Seow V, et al. Inflammatory responses induced by lipopolysaccharide are amplified in primary human monocytes but suppressed in macrophages by complement protein C5a. J Immunol. 2013;191:4308–4316.
-
(2013)
J Immunol
, vol.191
, pp. 4308-4316
-
-
Seow, V.1
-
18
-
-
70349260730
-
Complement promotes the development of inflammatory T-helper 17 cells through synergistic interaction with Toll-like receptor signaling and interleukin-6 production
-
Fang C, Zhang X, Miwa T, Song WC. Complement promotes the development of inflammatory T-helper 17 cells through synergistic interaction with Toll-like receptor signaling and interleukin-6 production. Blood. 2009;114:1005–1015.
-
(2009)
Blood
, vol.114
, pp. 1005-1015
-
-
Fang, C.1
Zhang, X.2
Miwa, T.3
Song, W.C.4
-
19
-
-
84902458366
-
Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis
-
Maekawa T, et al. Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis. Cell Host Microbe. 2014;15:768–778.
-
(2014)
Cell Host Microbe
, vol.15
, pp. 768-778
-
-
Maekawa, T.1
-
20
-
-
84871927877
-
Crosstalk between Toll like receptors and C5a receptor in human monocyte derived DCs suppress inflammatory cytokine production
-
Zaal A, Lissenberg-Thunnissen SN, van Schijndel G, Wouters D, van Ham SM, ten Brinke A. Crosstalk between Toll like receptors and C5a receptor in human monocyte derived DCs suppress inflammatory cytokine production. Immunobiology. 2013;218:175–180.
-
(2013)
Immunobiology
, vol.218
, pp. 175-180
-
-
Zaal, A.1
Lissenberg-Thunnissen, S.N.2
van Schijndel, G.3
Wouters, D.4
van Ham, S.M.5
ten Brinke, A.6
-
21
-
-
84860902599
-
Evidence for anti-inflammatory effects of C5a on the innate IL-17A/IL-23 axis
-
Bosmann M, Sarma JV, Atefi G, Zetoune FS, Ward PA. Evidence for anti-inflammatory effects of C5a on the innate IL-17A/IL-23 axis. FASEB J. 2011;26:1640–1651.
-
(2011)
FASEB J
, vol.26
, pp. 1640-1651
-
-
Bosmann, M.1
Sarma, J.V.2
Atefi, G.3
Zetoune, F.S.4
Ward, P.A.5
-
22
-
-
9144224761
-
Complement C1q regulates LPS-induced cytokine production in bone marrow-derived dendritic cells
-
Yamada M, et al. Complement C1q regulates LPS-induced cytokine production in bone marrow-derived dendritic cells. Eur J Immunol. 2004;34:221–230.
-
(2004)
Eur J Immunol
, vol.34
, pp. 221-230
-
-
Yamada, M.1
-
23
-
-
70349469876
-
Human genetic deficiencies reveal the roles of complement in the inflammatory network: lessons from nature
-
Lappegård KT, et al. Human genetic deficiencies reveal the roles of complement in the inflammatory network: lessons from nature. Proc Natl Acad Sci USA. 2009;106:15861–15866.
-
(2009)
Proc Natl Acad Sci USA
, vol.106
, pp. 15861-15866
-
-
Lappegård, K.T.1
-
24
-
-
11844295677
-
Role of C5a-C5aR interaction in sepsis
-
Guo RF, Riedemann NC, Ward PA. Role of C5a-C5aR interaction in sepsis. Shock. 2004;21:1–7.
-
(2004)
Shock
, vol.21
, pp. 1-7
-
-
Guo, R.F.1
Riedemann, N.C.2
Ward, P.A.3
-
25
-
-
32044432615
-
Membrane complement regulatory proteins
-
Kim DD, Song WC. Membrane complement regulatory proteins. Clin Immunol. 2006;118:127–136.
-
(2006)
Clin Immunol
, vol.118
, pp. 127-136
-
-
Kim, D.D.1
Song, W.C.2
-
26
-
-
84869761067
-
Local complement-targeted intervention in periodontitis: proof-of-concept using a C5a receptor (CD88) antagonist
-
Abe T, et al. Local complement-targeted intervention in periodontitis: proof-of-concept using a C5a receptor (CD88) antagonist. J Immunol. 2012;189:5442–5448.
-
(2012)
J Immunol
, vol.189
, pp. 5442-5448
-
-
Abe, T.1
-
27
-
-
79251544574
-
The C5a receptor impairs IL-12-dependent clearance of Porphyromonas gingivalis and is required for induction of periodontal bone loss
-
Liang S, et al. The C5a receptor impairs IL-12-dependent clearance of Porphyromonas gingivalis and is required for induction of periodontal bone loss. J Immunol. 2011;186:869–877.
-
(2011)
J Immunol
, vol.186
, pp. 869-877
-
-
Liang, S.1
-
28
-
-
33845462504
-
Cutting edge: TLR2 is required for the innate response to Porphyromonas gingivalis: activation leads to bacterial persistence and TLR2 deficiency attenuates induced alveolar bone resorption
-
Burns E, Bachrach G, Shapira L, Nussbaum G. Cutting edge: TLR2 is required for the innate response to Porphyromonas gingivalis: activation leads to bacterial persistence and TLR2 deficiency attenuates induced alveolar bone resorption. J Immunol. 2006;177:8296–8300.
-
(2006)
J Immunol
, vol.177
, pp. 8296-8300
-
-
Burns, E.1
Bachrach, G.2
Shapira, L.3
Nussbaum, G.4
-
29
-
-
84923792613
-
CD14 and complement crosstalk and largely mediate the transcriptional response to Escherichia coli in human whole blood as revealed by DNA microarray
-
Lau C, et al. CD14 and complement crosstalk and largely mediate the transcriptional response to Escherichia coli in human whole blood as revealed by DNA microarray. PLoS ONE. 2015;10:e0117261.
-
(2015)
PLoS ONE
, vol.10
-
-
Lau, C.1
-
30
-
-
48049089596
-
Combined inhibition of complement and CD14 abolish E. coli-induced cytokine-, chemokine- and growth factor-synthesis in human whole blood
-
Brekke OL, et al. Combined inhibition of complement and CD14 abolish E. coli-induced cytokine-, chemokine- and growth factor-synthesis in human whole blood. Mol Immunol. 2008;45:3804–3813.
-
(2008)
Mol Immunol
, vol.45
, pp. 3804-3813
-
-
Brekke, O.L.1
-
31
-
-
82655173731
-
MyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway
-
Bosmann M, et al. MyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway. FASEB J. 2011;25:4222–4232.
-
(2011)
FASEB J
, vol.25
, pp. 4222-4232
-
-
Bosmann, M.1
-
32
-
-
68849084891
-
Structure and signalling in the IL-17 receptor family
-
Gaffen SL. Structure and signalling in the IL-17 receptor family. Nat Rev Immunol. 2009;9:556–567.
-
(2009)
Nat Rev Immunol
, vol.9
, pp. 556-567
-
-
Gaffen, S.L.1
-
33
-
-
35348821843
-
Function, structure and therapeutic potential of complement C5a receptors
-
Monk PN, Scola AM, Madala P, Fairlie DP. Function, structure and therapeutic potential of complement C5a receptors. Br J Pharmacol. 2007;152:429–448.
-
(2007)
Br J Pharmacol
, vol.152
, pp. 429-448
-
-
Monk, P.N.1
Scola, A.M.2
Madala, P.3
Fairlie, D.P.4
-
34
-
-
0041571471
-
C5L2, a nonsignaling C5a binding Protein
-
Okinaga S, et al. C5L2, a nonsignaling C5a binding Protein. Biochemistry. 2003;42:9406–9415.
-
(2003)
Biochemistry
, vol.42
, pp. 9406-9415
-
-
Okinaga, S.1
-
35
-
-
20444460795
-
Evidence for a functional role of the second C5a receptor C5L2
-
Gao H, et al. Evidence for a functional role of the second C5a receptor C5L2. FASEB J. 2005;19:1003–1005.
-
(2005)
FASEB J
, vol.19
, pp. 1003-1005
-
-
Gao, H.1
-
36
-
-
28844434626
-
An anti-inflammatory function for the complement anaphylatoxin C5a-binding protein, C5L2
-
Gerard NP, et al. An anti-inflammatory function for the complement anaphylatoxin C5a-binding protein, C5L2. J Biol Chem. 2005;280:39677–39680.
-
(2005)
J Biol Chem
, vol.280
, pp. 39677-39680
-
-
Gerard, N.P.1
-
37
-
-
33947110020
-
C5L2 is critical for the biological activities of the anaphylatoxins C5a and C3a
-
Chen NJ, et al. C5L2 is critical for the biological activities of the anaphylatoxins C5a and C3a. Nature. 2007;446:203–207.
-
(2007)
Nature
, vol.446
, pp. 203-207
-
-
Chen, N.J.1
-
38
-
-
43249098418
-
Functional roles for C5a receptors in sepsis
-
Rittirsch D, et al. Functional roles for C5a receptors in sepsis. Nat Med. 2008;14:551–557.
-
(2008)
Nat Med
, vol.14
, pp. 551-557
-
-
Rittirsch, D.1
-
39
-
-
77951250395
-
The C5a receptor (C5aR) C5L2 is a modulator of C5aR-mediated signal transduction
-
Bamberg CE, et al. The C5a receptor (C5aR) C5L2 is a modulator of C5aR-mediated signal transduction. J Biol Chem. 2010;285:7633–7644.
-
(2010)
J Biol Chem
, vol.285
, pp. 7633-7644
-
-
Bamberg, C.E.1
-
40
-
-
84992470815
-
Differential contributions of the complement anaphylotoxin receptors C5aR1 and C5aR2 to the early innate immune response against Staphylococcus aureus infection
-
Horst SA, Itzek A, Klos A, Beineke A, Medina E. Differential contributions of the complement anaphylotoxin receptors C5aR1 and C5aR2 to the early innate immune response against Staphylococcus aureus infection. Pathogens. 2015;4:722–738.
-
(2015)
Pathogens
, vol.4
, pp. 722-738
-
-
Horst, S.A.1
Itzek, A.2
Klos, A.3
Beineke, A.4
Medina, E.5
-
41
-
-
84941779673
-
The novel receptor C5aR2 is required for C5a-mediated human mast cell adhesion, migration, and proinflammatory mediator production
-
Pundir P, MacDonald CA, Kulka M. The novel receptor C5aR2 is required for C5a-mediated human mast cell adhesion, migration, and proinflammatory mediator production. J Immunol. 2015;195:2774–2787.
-
(2015)
J Immunol
, vol.195
, pp. 2774-2787
-
-
Pundir, P.1
MacDonald, C.A.2
Kulka, M.3
-
42
-
-
84943172882
-
Atheroprotective role of C5ar2 deficiency in apolipoprotein E-deficient mice
-
Selle J, et al. Atheroprotective role of C5ar2 deficiency in apolipoprotein E-deficient mice. Thromb Haemost. 2015;114:848–858.
-
(2015)
Thromb Haemost
, vol.114
, pp. 848-858
-
-
Selle, J.1
-
43
-
-
84906063906
-
C5a2 can modulate ERK1/2 signaling in macrophages via heteromer formation with C5a1 and beta-arrestin recruitment
-
Croker DE, et al. C5a2 can modulate ERK1/2 signaling in macrophages via heteromer formation with C5a1 and beta-arrestin recruitment. Immunol Cell Biol. 2014;92:631–639.
-
(2014)
Immunol Cell Biol
, vol.92
, pp. 631-639
-
-
Croker, D.E.1
-
44
-
-
84874623794
-
C5L2: a controversial receptor of complement anaphylatoxin, C5a
-
Li R, Coulthard LG, Wu MC, Taylor SM, Woodruff TM. C5L2: a controversial receptor of complement anaphylatoxin, C5a. FASEB J. 2013;27:855–864.
-
(2013)
FASEB J
, vol.27
, pp. 855-864
-
-
Li, R.1
Coulthard, L.G.2
Wu, M.C.3
Taylor, S.M.4
Woodruff, T.M.5
-
45
-
-
33646908228
-
Phosphoinositide-mediated adaptor recruitment controls Toll-like receptor signaling
-
Kagan JC, Medzhitov R. Phosphoinositide-mediated adaptor recruitment controls Toll-like receptor signaling. Cell. 2006;125:943–955.
-
(2006)
Cell
, vol.125
, pp. 943-955
-
-
Kagan, J.C.1
Medzhitov, R.2
-
46
-
-
77954946392
-
Integrin CD11b negatively regulates TLR-triggered inflammatory responses by activating Syk and promoting degradation of MyD88 and TRIF via Cbl-b
-
Han C, Jin J, Xu S, Liu H, Li N, Cao X. Integrin CD11b negatively regulates TLR-triggered inflammatory responses by activating Syk and promoting degradation of MyD88 and TRIF via Cbl-b. Nat Immunol. 2010;11:734–742.
-
(2010)
Nat Immunol
, vol.11
, pp. 734-742
-
-
Han, C.1
Jin, J.2
Xu, S.3
Liu, H.4
Li, N.5
Cao, X.6
-
47
-
-
84875973489
-
Complement receptor 3 influences toll-like receptor 7/8-dependent inflammation: implications for autoimmune diseases characterized by antibody reactivity to ribonucleoproteins
-
Reed JH, et al. Complement receptor 3 influences toll-like receptor 7/8-dependent inflammation: implications for autoimmune diseases characterized by antibody reactivity to ribonucleoproteins. J Biol Chem. 2013;288:9077–9083.
-
(2013)
J Biol Chem
, vol.288
, pp. 9077-9083
-
-
Reed, J.H.1
-
48
-
-
79955780870
-
A systemic lupus erythematosus-associated R77H substitution in the CD11b chain of the Mac-1 integrin compromises leukocyte adhesion and phagocytosis
-
MacPherson M, Lek HS, Prescott A, Fagerholm SC. A systemic lupus erythematosus-associated R77H substitution in the CD11b chain of the Mac-1 integrin compromises leukocyte adhesion and phagocytosis. J Biol Chem. 2011;286:17303–17310.
-
(2011)
J Biol Chem
, vol.286
, pp. 17303-17310
-
-
MacPherson, M.1
Lek, H.S.2
Prescott, A.3
Fagerholm, S.C.4
-
49
-
-
77955915400
-
Pivotal advance: the pattern recognition receptor ligands lipopolysaccharide and polyinosine-polycytidylic acid stimulate factor B synthesis by the macrophage through distinct but overlapping mechanisms
-
Kaczorowski DJ, et al. Pivotal advance: the pattern recognition receptor ligands lipopolysaccharide and polyinosine-polycytidylic acid stimulate factor B synthesis by the macrophage through distinct but overlapping mechanisms. J Leukoc Biol. 2010;88:609–618.
-
(2010)
J Leukoc Biol
, vol.88
, pp. 609-618
-
-
Kaczorowski, D.J.1
-
50
-
-
84902165493
-
Mucosal toll-like receptor 3-dependent synthesis of complement factor B and systemic complement activation in inflammatory bowel disease
-
Ostvik AE, et al. Mucosal toll-like receptor 3-dependent synthesis of complement factor B and systemic complement activation in inflammatory bowel disease. Inflamm Bowel Dis. 2014;20:995–1003.
-
(2014)
Inflamm Bowel Dis
, vol.20
, pp. 995-1003
-
-
Ostvik, A.E.1
-
51
-
-
84888371687
-
Complement factor B is the downstream effector of TLRs and plays an important role in a mouse model of severe sepsis
-
Zou L, et al. Complement factor B is the downstream effector of TLRs and plays an important role in a mouse model of severe sepsis. J Immunol. 2013;191:5625–5635.
-
(2013)
J Immunol
, vol.191
, pp. 5625-5635
-
-
Zou, L.1
-
52
-
-
78649516502
-
Complement regulates TLR4-mediated inflammatory responses during intestinal ischemia reperfusion
-
Pope MR, Hoffman SM, Tomlinson S, Fleming SD. Complement regulates TLR4-mediated inflammatory responses during intestinal ischemia reperfusion. Mol Immunol. 2010;48:356–364.
-
(2010)
Mol Immunol
, vol.48
, pp. 356-364
-
-
Pope, M.R.1
Hoffman, S.M.2
Tomlinson, S.3
Fleming, S.D.4
-
53
-
-
65249154058
-
Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse heart
-
Singh MV, et al. Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse heart. J Clin Invest. 2009;119:986–996.
-
(2009)
J Clin Invest
, vol.119
, pp. 986-996
-
-
Singh, M.V.1
-
54
-
-
84857327497
-
Modified low density lipoprotein stimulates complement C3 expression and secretion via liver X receptor and Toll-like receptor 4 activation in human macrophages
-
Mogilenko DA, et al. Modified low density lipoprotein stimulates complement C3 expression and secretion via liver X receptor and Toll-like receptor 4 activation in human macrophages. J Biol Chem. 2012;287:5954–5968.
-
(2012)
J Biol Chem
, vol.287
, pp. 5954-5968
-
-
Mogilenko, D.A.1
-
55
-
-
84931268779
-
The complement system and toll-like receptors as integrated players in the pathophysiology of atherosclerosis
-
Hovland A, et al. The complement system and toll-like receptors as integrated players in the pathophysiology of atherosclerosis. Atherosclerosis. 2015;241:480–494.
-
(2015)
Atherosclerosis
, vol.241
, pp. 480-494
-
-
Hovland, A.1
-
56
-
-
0037351927
-
Toll-like receptor-4 (TLR4) signaling augments chemokine-induced neutrophil migration by modulating cell surface expression of chemokine receptors
-
Fan J, Malik AB. Toll-like receptor-4 (TLR4) signaling augments chemokine-induced neutrophil migration by modulating cell surface expression of chemokine receptors. Nat Med. 2003;9:315–321.
-
(2003)
Nat Med
, vol.9
, pp. 315-321
-
-
Fan, J.1
Malik, A.B.2
-
58
-
-
84924630982
-
Polymicrobial synergy and dysbiosis in inflammatory disease
-
Lamont RJ, Hajishengallis G. Polymicrobial synergy and dysbiosis in inflammatory disease. Trends Mol Med. 2015;21:172–183.
-
(2015)
Trends Mol Med
, vol.21
, pp. 172-183
-
-
Lamont, R.J.1
Hajishengallis, G.2
-
59
-
-
84971323731
-
Periodontitis: from microbial immune subversion to systemic inflammation
-
Hajishengallis G. Periodontitis: from microbial immune subversion to systemic inflammation. Nat Rev Immunol. 2015;15:30–44.
-
(2015)
Nat Rev Immunol
, vol.15
, pp. 30-44
-
-
Hajishengallis, G.1
-
60
-
-
84911397225
-
The inflammophilic character of the periodontitis-associated microbiota
-
Hajishengallis G. The inflammophilic character of the periodontitis-associated microbiota. Mol Oral Microbiol. 2014;29:248–257.
-
(2014)
Mol Oral Microbiol
, vol.29
, pp. 248-257
-
-
Hajishengallis, G.1
-
62
-
-
70249118556
-
The oral microbial consortium’s interaction with the periodontal innate defense system
-
Darveau RP. The oral microbial consortium’s interaction with the periodontal innate defense system. DNA Cell Biol. 2009;28:389–395.
-
(2009)
DNA Cell Biol
, vol.28
, pp. 389-395
-
-
Darveau, R.P.1
-
63
-
-
81755166205
-
Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement
-
Hajishengallis G, et al. Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement. Cell Host Microbe. 2011;10:497–506.
-
(2011)
Cell Host Microbe
, vol.10
, pp. 497-506
-
-
Hajishengallis, G.1
-
64
-
-
0037292394
-
Are dental diseases examples of ecological catastrophes?
-
Marsh PD. Are dental diseases examples of ecological catastrophes? Microbiology. 2003;149:279–294.
-
(2003)
Microbiology
, vol.149
, pp. 279-294
-
-
Marsh, P.D.1
-
65
-
-
34249799593
-
Biphasic effect of gingipains from Porphyromonas gingivalis on the human complement system
-
Popadiak K, Potempa J, Riesbeck K, Blom AM. Biphasic effect of gingipains from Porphyromonas gingivalis on the human complement system. J Immunol. 2007;178:7242–7250.
-
(2007)
J Immunol
, vol.178
, pp. 7242-7250
-
-
Popadiak, K.1
Potempa, J.2
Riesbeck, K.3
Blom, A.M.4
-
66
-
-
66549129856
-
Signalling mechanisms for Toll-like receptor-activated neutrophil exocytosis: key roles for interleukin-1-receptor-associated kinase-4 and phosphatidylinositol 3-kinase but not Toll/IL-1 receptor (TIR) domain-containing adaptor inducing IFNβ (TRIF)
-
Brzezinska AA, Johnson JL, Munafo DB, Ellis BA, Catz SD. Signalling mechanisms for Toll-like receptor-activated neutrophil exocytosis: key roles for interleukin-1-receptor-associated kinase-4 and phosphatidylinositol 3-kinase but not Toll/IL-1 receptor (TIR) domain-containing adaptor inducing IFNβ (TRIF). Immunology. 2009;127:386–397.
-
(2009)
Immunology
, vol.127
, pp. 386-397
-
-
Brzezinska, A.A.1
Johnson, J.L.2
Munafo, D.B.3
Ellis, B.A.4
Catz, S.D.5
-
67
-
-
77949316163
-
TLR2-dependent inflammatory response to Porphyromonas gingivalis is MyD88 independent, whereas MyD88 is required to clear infection
-
Burns E, Eliyahu T, Uematsu S, Akira S, Nussbaum G. TLR2-dependent inflammatory response to Porphyromonas gingivalis is MyD88 independent, whereas MyD88 is required to clear infection. J Immunol. 2010;184:1455–1462.
-
(2010)
J Immunol
, vol.184
, pp. 1455-1462
-
-
Burns, E.1
Eliyahu, T.2
Uematsu, S.3
Akira, S.4
Nussbaum, G.5
-
68
-
-
77950187399
-
Microbial hijacking of complement-toll-like receptor crosstalk
-
Wang M, et al. Microbial hijacking of complement-toll-like receptor crosstalk. Sci Signal. 2010;3:ra11.
-
(2010)
Sci Signal
, vol.3
, pp. ra11
-
-
Wang, M.1
-
69
-
-
17444403917
-
C5a negatively regulates toll-like receptor 4-induced immune responses
-
Hawlisch H, Belkaid Y, Baelder R, Hildeman D, Gerard C, Kohl J. C5a negatively regulates toll-like receptor 4-induced immune responses. Immunity. 2005;22:415–426.
-
(2005)
Immunity
, vol.22
, pp. 415-426
-
-
Hawlisch, H.1
Belkaid, Y.2
Baelder, R.3
Hildeman, D.4
Gerard, C.5
Kohl, J.6
-
70
-
-
23844434887
-
G(i)-protein-dependent inhibition of IL-12 production is mediated by activation of the phosphatidylinositol 3-kinase-protein 3 kinase B/Akt pathway and JNK
-
la Sala A, Gadina M, Kelsall BL. G(i)-protein-dependent inhibition of IL-12 production is mediated by activation of the phosphatidylinositol 3-kinase-protein 3 kinase B/Akt pathway and JNK. J Immunol. 2005;175:2994–2999.
-
(2005)
J Immunol
, vol.175
, pp. 2994-2999
-
-
la Sala, A.1
Gadina, M.2
Kelsall, B.L.3
-
71
-
-
0034122721
-
CR3: a general purpose adhesion-recognition receptor essential for innate immunity
-
Ehlers MRW. CR3: a general purpose adhesion-recognition receptor essential for innate immunity. Microbes Infect. 2000;2:289–294.
-
(2000)
Microbes Infect
, vol.2
, pp. 289-294
-
-
Ehlers, M.R.W.1
-
72
-
-
0027530684
-
The I domain is a major recognition site on the leukocyte integrin Mac-1 (CD11b/CD18) for four distinct adhesion ligands
-
Diamond MS, Garcia-Aguilar J, Bickford JK, Corbi AL, Springer TA. The I domain is a major recognition site on the leukocyte integrin Mac-1 (CD11b/CD18) for four distinct adhesion ligands. J Cell Biol. 1993;120:1031–1043.
-
(1993)
J Cell Biol
, vol.120
, pp. 1031-1043
-
-
Diamond, M.S.1
Garcia-Aguilar, J.2
Bickford, J.K.3
Corbi, A.L.4
Springer, T.A.5
-
73
-
-
0033952395
-
Direct anti-inflammatory effect of a bacterial virulence factor: IL-10-dependent suppression of IL-12 production by filamentous hemagglutinin from Bordetella pertussis
-
McGuirk P, Mills KH. Direct anti-inflammatory effect of a bacterial virulence factor: IL-10-dependent suppression of IL-12 production by filamentous hemagglutinin from Bordetella pertussis. Eur J Immunol. 2000;30:415–422.
-
(2000)
Eur J Immunol
, vol.30
, pp. 415-422
-
-
McGuirk, P.1
Mills, K.H.2
-
74
-
-
0023726598
-
Complement receptor type 3 (CR3) binds to an Arg-Gly-Asp-containing region of the major surface glycoprotein, gp63, of Leishmania promastigotes
-
Russell DG, Wright SD. Complement receptor type 3 (CR3) binds to an Arg-Gly-Asp-containing region of the major surface glycoprotein, gp63, of Leishmania promastigotes. J Exp Med. 1988;168:279–292.
-
(1988)
J Exp Med
, vol.168
, pp. 279-292
-
-
Russell, D.G.1
Wright, S.D.2
-
75
-
-
28244498667
-
Peptide mapping of bacterial fimbrial epitopes interacting with pattern recognition receptors
-
Hajishengallis G, Ratti P, Harokopakis E. Peptide mapping of bacterial fimbrial epitopes interacting with pattern recognition receptors. J Biol Chem. 2005;280:38902–38913.
-
(2005)
J Biol Chem
, vol.280
, pp. 38902-38913
-
-
Hajishengallis, G.1
Ratti, P.2
Harokopakis, E.3
-
76
-
-
0031183179
-
Outside-in signaling by lipopolysaccharide through a tailless integrin
-
Ingalls RR, Arnaout MA, Golenbock DT. Outside-in signaling by lipopolysaccharide through a tailless integrin. J Immunol. 1997;159:433–438.
-
(1997)
J Immunol
, vol.159
, pp. 433-438
-
-
Ingalls, R.R.1
Arnaout, M.A.2
Golenbock, D.T.3
-
77
-
-
17444394875
-
Integrin activation by bacterial fimbriae through a pathway involving CD14, Toll-like receptor 2, and phosphatidylinositol-3-kinase
-
Harokopakis E, Hajishengallis G. Integrin activation by bacterial fimbriae through a pathway involving CD14, Toll-like receptor 2, and phosphatidylinositol-3-kinase. Eur J Immunol. 2005;35:1201–1210.
-
(2005)
Eur J Immunol
, vol.35
, pp. 1201-1210
-
-
Harokopakis, E.1
Hajishengallis, G.2
-
78
-
-
67449116290
-
Induction of distinct TLR2-mediated proinflammatory and proadhesive signaling pathways in response to Porphyromonas gingivalis fimbriae
-
Hajishengallis G, Wang M, Liang S. Induction of distinct TLR2-mediated proinflammatory and proadhesive signaling pathways in response to Porphyromonas gingivalis fimbriae. J Immunol. 2009;182:6690–6696.
-
(2009)
J Immunol
, vol.182
, pp. 6690-6696
-
-
Hajishengallis, G.1
Wang, M.2
Liang, S.3
-
79
-
-
33748473184
-
Differential interactions of fimbriae and lipopolysaccharide from Porphyromonas gingivalis with the Toll-like receptor 2-centred pattern recognition apparatus
-
Hajishengallis G, et al. Differential interactions of fimbriae and lipopolysaccharide from Porphyromonas gingivalis with the Toll-like receptor 2-centred pattern recognition apparatus. Cell Microbiol. 2006;8:1557–1570.
-
(2006)
Cell Microbiol
, vol.8
, pp. 1557-1570
-
-
Hajishengallis, G.1
-
80
-
-
33744903384
-
TLR2 transmodulates monocyte adhesion and transmigration via Rac1- and PI3K-mediated inside-out signaling in response to Porphyromonas gingivalis fimbriae
-
Harokopakis E, Albzreh MH, Martin MH, Hajishengallis G. TLR2 transmodulates monocyte adhesion and transmigration via Rac1- and PI3K-mediated inside-out signaling in response to Porphyromonas gingivalis fimbriae. J Immunol. 2006;176:7645–7656.
-
(2006)
J Immunol
, vol.176
, pp. 7645-7656
-
-
Harokopakis, E.1
Albzreh, M.H.2
Martin, M.H.3
Hajishengallis, G.4
-
81
-
-
34848908962
-
Fimbrial proteins of Porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages
-
Wang M, et al. Fimbrial proteins of Porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages. J Immunol. 2007;179:2349–2358.
-
(2007)
J Immunol
, vol.179
, pp. 2349-2358
-
-
Wang, M.1
-
82
-
-
33644985957
-
Rewiring phagocytic signal transduction
-
Lowell CA. Rewiring phagocytic signal transduction. Immunity. 2006;24:243–245.
-
(2006)
Immunity
, vol.24
, pp. 243-245
-
-
Lowell, C.A.1
-
83
-
-
0021080912
-
Receptors for C3b and C3bi promote phagocytosis but not the release of toxic oxygen from human phagocytes
-
Wright SD, Silverstein SC. Receptors for C3b and C3bi promote phagocytosis but not the release of toxic oxygen from human phagocytes. J Exp Med. 1983;158:2016–2023.
-
(1983)
J Exp Med
, vol.158
, pp. 2016-2023
-
-
Wright, S.D.1
Silverstein, S.C.2
-
84
-
-
0032573378
-
Identification of two distinct mechanisms of phagocytosis controlled by different Rho GTPases
-
Caron E, Hall A. Identification of two distinct mechanisms of phagocytosis controlled by different Rho GTPases. Science. 1998;282:1717–1721.
-
(1998)
Science
, vol.282
, pp. 1717-1721
-
-
Caron, E.1
Hall, A.2
-
85
-
-
0035866555
-
Targeting to Fcg receptors, but not CR3 (CD11b/CD18), increases clearance of Bordetella pertussis
-
Hellwig SM, van Oirschot HF, Hazenbos WL, van Spriel AB, Mooi FR, van De Winkel JG. Targeting to Fcg receptors, but not CR3 (CD11b/CD18), increases clearance of Bordetella pertussis. J Infect Dis. 2001;183:871–879.
-
(2001)
J Infect Dis
, vol.183
, pp. 871-879
-
-
Hellwig, S.M.1
van Oirschot, H.F.2
Hazenbos, W.L.3
van Spriel, A.B.4
Mooi, F.R.5
van De Winkel, J.G.6
-
87
-
-
8444251663
-
Transcriptional suppression of interleukin-12 gene expression following phagocytosis of apoptotic cells
-
Kim S, Elkon KB, Ma X. Transcriptional suppression of interleukin-12 gene expression following phagocytosis of apoptotic cells. Immunity. 2004;21:643–653.
-
(2004)
Immunity
, vol.21
, pp. 643-653
-
-
Kim, S.1
Elkon, K.B.2
Ma, X.3
-
88
-
-
0032416392
-
Complement-dependent clearance of apoptotic cells by human macrophages
-
Mevorach D, Mascarenhas JO, Gershov D, Elkon KB. Complement-dependent clearance of apoptotic cells by human macrophages. J Exp Med. 1998;188:2313–2320.
-
(1998)
J Exp Med
, vol.188
, pp. 2313-2320
-
-
Mevorach, D.1
Mascarenhas, J.O.2
Gershov, D.3
Elkon, K.B.4
-
89
-
-
34848855747
-
Complement receptor 3 blockade promotes IL-12-mediated clearance of Porphyromonas gingivalis and negates its virulence in vivo
-
Hajishengallis G, Shakhatreh MA, Wang M, Liang S. Complement receptor 3 blockade promotes IL-12-mediated clearance of Porphyromonas gingivalis and negates its virulence in vivo. J Immunol. 2007;179:2359–2367.
-
(2007)
J Immunol
, vol.179
, pp. 2359-2367
-
-
Hajishengallis, G.1
Shakhatreh, M.A.2
Wang, M.3
Liang, S.4
-
90
-
-
85014620914
-
Differential capacity for complement receptor-mediated immune evasion by Porphyromonas gingivalis depending on the type of innate leukocyte
-
[Epub ahead of print]
-
Hajishengallis G, Krauss JL, Jotwani R, Lambris JD. Differential capacity for complement receptor-mediated immune evasion by Porphyromonas gingivalis depending on the type of innate leukocyte. Mol Oral Microbiol. 2016. doi: 10.1111/omi.12161. [Epub ahead of print].
-
(2016)
Mol Oral Microbiol
-
-
Hajishengallis, G.1
Krauss, J.L.2
Jotwani, R.3
Lambris, J.D.4
-
91
-
-
77749327561
-
Dendritic cell function in allostimulation is modulated by C5aR signaling
-
Peng Q, et al. Dendritic cell function in allostimulation is modulated by C5aR signaling. J Immunol. 2009;183:6058–6068.
-
(2009)
J Immunol
, vol.183
, pp. 6058-6068
-
-
Peng, Q.1
-
92
-
-
58149393171
-
Cyclic AMP plays a critical role in C3a-receptor-mediated regulation of dendritic cells in antigen uptake and T-cell stimulation
-
Li K, et al. Cyclic AMP plays a critical role in C3a-receptor-mediated regulation of dendritic cells in antigen uptake and T-cell stimulation. Blood. 2008;112:5084–5094.
-
(2008)
Blood
, vol.112
, pp. 5084-5094
-
-
Li, K.1
-
93
-
-
0141541343
-
Isoforms of mammalian adenylyl cyclase: multiplicities of signaling
-
Sunahara RK, Taussig R. Isoforms of mammalian adenylyl cyclase: multiplicities of signaling. Mol Interv. 2002;2:168–184.
-
(2002)
Mol Interv
, vol.2
, pp. 168-184
-
-
Sunahara, R.K.1
Taussig, R.2
-
94
-
-
77749285675
-
C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells
-
Weaver DJ Jr, et al. C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells. Eur J Immunol. 2010;40:710–721.
-
(2010)
Eur J Immunol
, vol.40
, pp. 710-721
-
-
Weaver, D.J.1
-
95
-
-
65349090158
-
C1q enhances IFN-g production by antigen-specific T cells via the CD40 costimulatory pathway on dendritic cells
-
Baruah P, et al. C1q enhances IFN-g production by antigen-specific T cells via the CD40 costimulatory pathway on dendritic cells. Blood. 2009;113:3485–3493.
-
(2009)
Blood
, vol.113
, pp. 3485-3493
-
-
Baruah, P.1
-
96
-
-
33846190080
-
Active MAC-1 (CD11b/CD18) on DCs inhibits full T-cell activation
-
Varga G, et al. Active MAC-1 (CD11b/CD18) on DCs inhibits full T-cell activation. Blood. 2007;109:661–669.
-
(2007)
Blood
, vol.109
, pp. 661-669
-
-
Varga, G.1
-
97
-
-
74949140425
-
When two is better than one: macrophages and neutrophils work in concert in innate immunity as complementary and cooperative partners of a myeloid phagocyte system
-
Silva MT. When two is better than one: macrophages and neutrophils work in concert in innate immunity as complementary and cooperative partners of a myeloid phagocyte system. J Leukoc Biol. 2010;87:93–106.
-
(2010)
J Leukoc Biol
, vol.87
, pp. 93-106
-
-
Silva, M.T.1
-
98
-
-
78049417345
-
Complement and periodontitis
-
Hajishengallis G. Complement and periodontitis. Biochem Pharmacol. 2010;80:1992–2001.
-
(2010)
Biochem Pharmacol
, vol.80
, pp. 1992-2001
-
-
Hajishengallis, G.1
-
99
-
-
0037264098
-
Origin and function of the cellular components in gingival crevice fluid
-
Delima AJ, Van Dyke TE. Origin and function of the cellular components in gingival crevice fluid. Periodontol 2000. 2003;31:55–76.
-
(2003)
Periodontol 2000
, vol.31
, pp. 55-76
-
-
Delima, A.J.1
Van Dyke, T.E.2
-
100
-
-
69549098037
-
CD14-Mac-1 interactions in Bacillus anthracis spore internalization by macrophages
-
Oliva C, Turnbough CL Jr, Kearney JF. CD14-Mac-1 interactions in Bacillus anthracis spore internalization by macrophages. Proc Natl Acad Sci USA. 2009;106:13957–13962.
-
(2009)
Proc Natl Acad Sci USA
, vol.106
, pp. 13957-13962
-
-
Oliva, C.1
Turnbough, C.L.2
Kearney, J.F.3
-
101
-
-
15444365158
-
Cross-talk between CD14 and complement receptor 3 promotes phagocytosis of mycobacteria: regulation by phosphatidylinositol 3-kinase and cytohesin-1
-
Sendide K, Reiner NE, Lee JS, Bourgoin S, Talal A, Hmama Z. Cross-talk between CD14 and complement receptor 3 promotes phagocytosis of mycobacteria: regulation by phosphatidylinositol 3-kinase and cytohesin-1. J Immunol. 2005;174:4210–4219.
-
(2005)
J Immunol
, vol.174
, pp. 4210-4219
-
-
Sendide, K.1
Reiner, N.E.2
Lee, J.S.3
Bourgoin, S.4
Talal, A.5
Hmama, Z.6
-
102
-
-
84875066771
-
Fine tuning inflammation at the front door: macrophage complement receptor 3-mediates phagocytosis and immune suppression for Francisella tularensis
-
Dai S, Rajaram MV, Curry HM, Leander R, Schlesinger LS. Fine tuning inflammation at the front door: macrophage complement receptor 3-mediates phagocytosis and immune suppression for Francisella tularensis. PLoS Pathog. 2013;9:e1003114.
-
(2013)
PLoS Pathog
, vol.9
-
-
Dai, S.1
Rajaram, M.V.2
Curry, H.M.3
Leander, R.4
Schlesinger, L.S.5
-
103
-
-
84908131727
-
Complement opsonization of HIV-1 results in decreased antiviral and inflammatory responses in immature dendritic cells via CR3
-
Ellegard R, et al. Complement opsonization of HIV-1 results in decreased antiviral and inflammatory responses in immature dendritic cells via CR3. J Immunol. 2014;193:4590–4601.
-
(2014)
J Immunol
, vol.193
, pp. 4590-4601
-
-
Ellegard, R.1
-
104
-
-
25444465640
-
gC1q receptor ligation selectively down-regulates human IL-12 production through activation of the phosphoinositide 3-kinase pathway
-
Waggoner SN, Cruise MW, Kassel R, Hahn YS. gC1q receptor ligation selectively down-regulates human IL-12 production through activation of the phosphoinositide 3-kinase pathway. J Immunol. 2005;175:4706–4714.
-
(2005)
J Immunol
, vol.175
, pp. 4706-4714
-
-
Waggoner, S.N.1
Cruise, M.W.2
Kassel, R.3
Hahn, Y.S.4
-
105
-
-
37249079121
-
+ T cells via suppression of dendritic cell IL-12 production
-
+ T cells via suppression of dendritic cell IL-12 production. J Leukoc Biol. 2007;82:1407–1419.
-
(2007)
J Leukoc Biol
, vol.82
, pp. 1407-1419
-
-
Waggoner, S.N.1
Hall, C.H.2
Hahn, Y.S.3
-
106
-
-
0029818545
-
Mechanism of suppression of cell-mediated immunity by measles virus
-
Karp CL, et al. Mechanism of suppression of cell-mediated immunity by measles virus. Science. 1996;273:228–231.
-
(1996)
Science
, vol.273
, pp. 228-231
-
-
Karp, C.L.1
-
107
-
-
0141923890
-
Selective suppression of IL-12 production by human herpesvirus 6
-
Smith A, Santoro F, Di Lullo G, Dagna L, Verani A, Lusso P. Selective suppression of IL-12 production by human herpesvirus 6. Blood. 2003;102:2877–2884.
-
(2003)
Blood
, vol.102
, pp. 2877-2884
-
-
Smith, A.1
Santoro, F.2
Di Lullo, G.3
Dagna, L.4
Verani, A.5
Lusso, P.6
-
108
-
-
84956811284
-
Combined inhibition of complement and CD14 improved outcome in porcine polymicrobial sepsis
-
Skjeflo EW, et al. Combined inhibition of complement and CD14 improved outcome in porcine polymicrobial sepsis. Crit Care. 2015;19:415.
-
(2015)
Crit Care
, vol.19
, pp. 415
-
-
Skjeflo, E.W.1
-
109
-
-
84960146246
-
Inhibition of pre-existing natural periodontitis in non-human primates by a locally administered peptide inhibitor of complement C3
-
Maekawa T, et al. Inhibition of pre-existing natural periodontitis in non-human primates by a locally administered peptide inhibitor of complement C3. J Clin Periodontol. 2016; 43:238–249.
-
(2016)
J Clin Periodontol
, vol.43
, pp. 238-249
-
-
Maekawa, T.1
-
110
-
-
84953835876
-
Complement therapeutics in inflammatory diseases: promising drug candidates for C3-targeted intervention
-
Mastellos DC, Ricklin D, Hajishengallis E, Hajishengallis G, Lambris JD. Complement therapeutics in inflammatory diseases: promising drug candidates for C3-targeted intervention. Mol Oral Microbiol. 2016;31:3–17.
-
(2016)
Mol Oral Microbiol
, vol.31
, pp. 3-17
-
-
Mastellos, D.C.1
Ricklin, D.2
Hajishengallis, E.3
Hajishengallis, G.4
Lambris, J.D.5
-
111
-
-
84862749585
-
Complement activation and toll-like receptor-2 signaling contribute to cytokine production after renal ischemia/reperfusion
-
Amura CR, Renner B, Lyubchenko T, Faubel S, Simonian PL, Thurman JM. Complement activation and toll-like receptor-2 signaling contribute to cytokine production after renal ischemia/reperfusion. Mol Immunol. 2012;52:249–257.
-
(2012)
Mol Immunol
, vol.52
, pp. 249-257
-
-
Amura, C.R.1
Renner, B.2
Lyubchenko, T.3
Faubel, S.4
Simonian, P.L.5
Thurman, J.M.6
-
112
-
-
84861137551
-
Complement activation product C5a is a selective suppressor of TLR4-induced, but not TLR3-induced, production of IL-27(p28) from macrophages
-
Bosmann M, Haggadone MD, Hemmila MR, Zetoune FS, Sarma JV, Ward PA. Complement activation product C5a is a selective suppressor of TLR4-induced, but not TLR3-induced, production of IL-27(p28) from macrophages. J Immunol. 2012;188:5086–5093.
-
(2012)
J Immunol
, vol.188
, pp. 5086-5093
-
-
Bosmann, M.1
Haggadone, M.D.2
Hemmila, M.R.3
Zetoune, F.S.4
Sarma, J.V.5
Ward, P.A.6
-
113
-
-
84880828525
-
Novel roles for complement receptors in T cell regulation and beyond
-
Kemper C, Kohl J. Novel roles for complement receptors in T cell regulation and beyond. Mol Immunol. 2013;56:181–190.
-
(2013)
Mol Immunol
, vol.56
, pp. 181-190
-
-
Kemper, C.1
Kohl, J.2
-
114
-
-
84858762160
-
Crosstalk between complement and toll-like receptors
-
Song WC. Crosstalk between complement and toll-like receptors. Toxicol Pathol. 2012;40:174–182.
-
(2012)
Toxicol Pathol
, vol.40
, pp. 174-182
-
-
Song, W.C.1
-
115
-
-
84935041763
-
Complement system part II: role in immunity
-
Merle NS, Noe R, Halbwachs-Mecarelli L, Fremeaux-Bacchi V, Roumenina LT. Complement system part II: role in immunity. Front Immunol. 2015;6:257.
-
(2015)
Front Immunol
, vol.6
, pp. 257
-
-
Merle, N.S.1
Noe, R.2
Halbwachs-Mecarelli, L.3
Fremeaux-Bacchi, V.4
Roumenina, L.T.5
|