Differential capacity for complement receptor-mediated immune evasion by Porphyromonas gingivalis depending on the type of innate leukocyte

Molecular Oral Microbiology

Volumn 32, Issue 2, 2017, Pages 154-165

  Hajishengallis, G ^a   Krauss, J L ^b,d   Jotwani, R ^b   Lambris, J D ^c  

^a UNIVERSITY OF PENNSYLVANIA   (United States)

^b UNIVERSITY OF LOUISVILLE   (United States)

^c UNIVERSITY OF PENNSYLVANIA   (United States)

^d WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

C3aR; C5aR1; C5aR2; Complement; CR3; dendritic cells; immune evasion; intracellular killing; Porphyromonas gingivalis

Indexed keywords

COMPLEMENT COMPONENT C3A; COMPLEMENT COMPONENT C3A RECEPTOR; COMPLEMENT COMPONENT C5A; COMPLEMENT COMPONENT C5A RECEPTOR; COMPLEMENT COMPONENT C5A RECEPTOR 1; COMPLEMENT COMPONENT C5A RECEPTOR 2; COMPLEMENT RECEPTOR; G PROTEIN COUPLED RECEPTOR; G PROTEIN COUPLED RECEPTOR 77; UNCLASSIFIED DRUG; CD11B ANTIGEN; CYTOKINE;

ANIMAL CELL; ANTIGEN PRESENTING CELL; ARTICLE; BACTERIAL COUNT; BACTERIAL SURVIVAL; BACTERIAL VIABILITY; BONE MARROW DERIVED DENDRITIC CELL; CELL SURVIVAL; CELL VIABILITY; COMPLEMENT SYSTEM; CONTROLLED STUDY; CYTOKINE PRODUCTION; HUMAN; HUMAN CELL; IMMUNE EVASION; INNATE IMMUNITY; INTRACELLULAR KILLING; LEUKOCYTE; MONOCYTE; MOUSE; MYELOID DENDRITIC CELL; NONHUMAN; PORPHYROMONAS GINGIVALIS; PROTEIN FUNCTION; UPREGULATION; ANIMAL; BONE MARROW CELL; DENDRITIC CELL; FIMBRIA; HOST PATHOGEN INTERACTION; IMMUNOLOGY; METABOLISM; MICROBIOLOGY; PATHOGENICITY; PHYSIOLOGY;

ANIMALS; BONE MARROW CELLS; CYTOKINES; DENDRITIC CELLS; FIMBRIAE, BACTERIAL; HOST-PATHOGEN INTERACTIONS; HUMANS; IMMUNE EVASION; IMMUNITY, INNATE; LEUKOCYTES; MACROPHAGE-1 ANTIGEN; MICE; MONOCYTES; PORPHYROMONAS GINGIVALIS; RECEPTORS, COMPLEMENT; UP-REGULATION;

EID: 85014620914     PISSN: 20411006     EISSN: 20411014     Source Type: Journal    
DOI: 10.1111/omi.12161     Document Type: Article

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