Mechanisms of tumor cell necrosis

Current Pharmaceutical Design

Volumn 16, Issue 1, 2010, Pages 56-68

  Proskuryakov, Sergey Y ^a   Gabai, Vladimir L ^b  

^a MEDICAL RADIOLOGICAL RESEARCH CENTER   (Russian Federation)

^b BOSTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Anti cancer therapy; Inflammation; Mitochondria; Oxidative stress; Programmed cell death; RIP1 kinase; Tumor progression

Indexed keywords

ADENOSINE DIPHOSPHATE RIBOSE; ADENOSINE TRIPHOSPHATE; ANTIMYCIN A1; ARSENIC TRIOXIDE; BENZYLOXYCARBONYLVALYLALANYLASPARTYL FLUOROMETHYL KETONE; CALPAIN; CATHEPSIN; CERAMIDE; CISPLATIN; DNA; GAMMA INTERFERON; HEAT SHOCK PROTEIN; HONOKIOL; IMATINIB; INTERLEUKIN 1BETA; LIPOPOLYSACCHARIDE; NUCLEASE; PHOSPHOLIPASE; PROTEIN BCL 2; PROTEIN KINASE; PROTEINASE; REACTIVE NITROGEN SPECIES; REACTIVE OXYGEN METABOLITE; RESVERATROL; ROTENONE; SHIKONIN; TAMOXIFEN; TUMOR NECROSIS FACTOR; TUMOR NECROSIS FACTOR RELATED APOPTOSIS INDUCING LIGAND;

ANTINEOPLASTIC ACTIVITY; APOPTOSIS; CELL DEATH; CELL HYPOXIA; CELL MEMBRANE; DNA DAMAGE; HUMAN; IMMUNE RESPONSE; MITOCHONDRIAL PERMEABILITY; NONHUMAN; OXIDATIVE STRESS; PHOTODYNAMIC THERAPY; PRIORITY JOURNAL; REVIEW; TUMOR CELL;

ANIMALS; ANTINEOPLASTIC AGENTS; APOPTOSIS; CELL LINE, TUMOR; HUMANS; INFLAMMATION MEDIATORS; NECROSIS; NEOPLASMS; SIGNAL TRANSDUCTION;

EID: 77949454722     PISSN: 13816128     EISSN: None     Source Type: Journal    
DOI: 10.2174/138161210789941793     Document Type: Review

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