BCL-2 protects peroxynitrite-treated thymocytes from poly(ADP-ribose) synthase (PARS)-independent apoptotic but not from PARS-mediated necrotic cell death

Free Radical Biology and Medicine

Volumn 29, Issue 8, 2000, Pages 704-713

  Virág, László ^a,b,c   Szabó, Csaba ^a,c  

^a INOTEK CORPORATION   (United States)

^b UNIVERSITY OF DEBRECEN   (Hungary)

^c CINCINNATI CHILDREN'S HOSPITAL MEDICAL CENTER   (United States)

Author keywords

Apoptosis; Bcl 2; Free radicals; Necrosis; Peroxynitrite; Poly(ADP ribose) synthetase; Transglutaminase

Indexed keywords

CARDIOLIPIN; ENZYME INHIBITOR; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; PEROXYNITRITE; PROPIDIUM IODIDE; PROTEIN BCL 2; PROTEIN GLUTAMINE GAMMA GLUTAMYLTRANSFERASE;

ANIMAL CELL; ANIMAL EXPERIMENT; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CYTOTOXICITY; FLOW CYTOMETRY; MALE; MITOCHONDRIAL MEMBRANE; MITOCHONDRION; NECROSIS; NONHUMAN; PRIORITY JOURNAL; THYMOCYTE; TRANSGENIC MOUSE;

ANIMAL; APOPTOSIS; CASPASES; DNA FRAGMENTATION; ENZYME ACTIVATION; GENES, BCL-2; IN VITRO; MICE; MICE, TRANSGENIC; MITOCHONDRIA; NECROSIS; NITRATES; POLY(ADP-RIBOSE) POLYMERASES; PROTO-ONCOGENE PROTEINS C-BCL-2; SUPPORT, U.S. GOV'T, P.H.S.; T-LYMPHOCYTES; TRANSGLUTAMINASES;

MUS MUSCULUS;

EID: 0034667687     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0891-5849(00)00359-2     Document Type: Article

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