Do axonal defects in tau and amyloid precursor protein transgenic animals model axonopathy in Alzheimer's disease?

Journal of Neurochemistry

Volumn 98, Issue 4, 2006, Pages 993-1006

  Götz, Jürgen ^a   Ittner, Lars M ^a   Kins, Stefan ^b  

^a UNIVERSITY OF SYDNEY   (Australia)

^b UNIVERSITY OF HEIDELBERG   (Germany)

Author keywords

Alzheimer's disease; Amyloid; Animal models; Axonal transport; Axonopathy; Tau

Indexed keywords

AMYLOID BETA PROTEIN[25-35]; AMYLOID PRECURSOR PROTEIN; PRESENILIN 1; TAU PROTEIN;

ALZHEIMER DISEASE; AMYLOID NEUROPATHY; ANIMAL MODEL; DENDRITIC CELL; DOPAMINERGIC NERVE CELL; GENE MUTATION; HISTOPATHOLOGY; HUMAN; MICROTUBULE; MOUSE; NERVE CELL NECROSIS; NERVE CELL PLASTICITY; NERVE FIBER TRANSPORT; NEUROFIBRILLARY TANGLE; NEUROPATHOLOGY; NEUROPATHY; NONHUMAN; PLAQUE FORMING CELL; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; SHORT SURVEY; SURVIVAL RATE; TRANSGENIC MOUSE;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; AXONAL TRANSPORT; AXONS; HUMANS; MICE; MICE, TRANSGENIC; TAU PROTEINS;

ANIMALIA; MUS MUSCULUS;

EID: 33746411112     PISSN: 00223042     EISSN: 14714159     Source Type: Journal    
DOI: 10.1111/j.1471-4159.2006.03955.x     Document Type: Short Survey

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