PTEN function in mammalian cell size regulation

Current Opinion in Neurobiology

Volumn 12, Issue 5, 2002, Pages 516-522

  Backman, Stéphanie A ^a   Stambolic, Vuk ^a   Mak, Tak W ^a  

^a UNIVERSITY OF TORONTO   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

LIPID PHOSPHATASE; PHOSPHATASE; PHOSPHATIDYLINOSITOL 3,4,5 TRISPHOSPHATE 3 PHOSPHATASE; PROTEIN KINASE B; UNCLASSIFIED DRUG;

CELL DIFFERENTIATION; CELL GROWTH; CELL SIZE; CELL SURVIVAL; CONTROLLED STUDY; DISEASE ASSOCIATION; DROSOPHILA; GENE DELETION; GROWTH REGULATION; HAMARTOMA; LHERMITTE DUCLOS DISEASE; NERVE CELL; NEUROLOGIC DISEASE; NONHUMAN; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN FUNCTION; REGULATORY MECHANISM; REVIEW; SIGNAL TRANSDUCTION; TUMOR SUPPRESSOR GENE;

ANIMALS; CELL DIFFERENTIATION; CELL DIVISION; CELL SIZE; HAMARTOMA SYNDROME, MULTIPLE; HUMANS; NEURONS; PHOSPHORIC MONOESTER HYDROLASES; PTEN PHOSPHOHYDROLASE; TUMOR SUPPRESSOR PROTEINS;

EID: 0036801311     PISSN: 09594388     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0959-4388(02)00354-9     Document Type: Review

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65. 3 represents a critical step in the interpretation of PI3K/PTEN signals.
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