A RASopathy gene commonly mutated in cancer: The neurofibromatosis type 1 tumour suppressor

Nature Reviews Cancer

Volumn 15, Issue 5, 2015, Pages 290-301

  Ratner, Nancy ^a   Miller, Shyra J ^a  

^a CINCINNATI CHILDREN'S HOSPITAL MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIC AMP; IMATINIB; NEUROFIBROMIN; RAS PROTEIN; SELUMETINIB; GUANOSINE TRIPHOSPHATASE ACTIVATING PROTEIN;

ASTROCYTOMA; CANCER GRADING; CANCER RESISTANCE; GENE MUTATION; GENE STRUCTURE; HUMAN; MODIFIER GENE; MUTATIONAL ANALYSIS; NEUROFIBROMATOSIS TYPE 1; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; REGULATORY MECHANISM; REVIEW; SIGNAL TRANSDUCTION; TUMOR SUPPRESSOR GENE; GENETIC PREDISPOSITION; GENETICS; MUTATION; ONCOGENE RAS;

GENES, RAS; GENES, TUMOR SUPPRESSOR; GENETIC PREDISPOSITION TO DISEASE; GTPASE-ACTIVATING PROTEINS; HUMANS; MUTATION; NEUROFIBROMATOSIS 1; NEUROFIBROMIN 1; SIGNAL TRANSDUCTION;

EID: 84928583796     PISSN: 1474175X     EISSN: 14741768     Source Type: Journal    
DOI: 10.1038/nrc3911     Document Type: Review

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