-
1
-
-
44049086446
-
Cooperating gene mutations in acute myeloid leukemia: A review of the literature
-
Renneville A, Roumier C, Biggio V, et al. Cooperating gene mutations in acute myeloid leukemia: A review of the literature. Leukemia 2008;22:915-931.
-
(2008)
Leukemia
, vol.22
, pp. 915-931
-
-
Renneville, A.1
Roumier, C.2
Biggio, V.3
-
2
-
-
84155165116
-
Prognostic implications of genetic aberrations in acute myelogenous leukemia with normal cytogenetics
-
Ghanem H, Tank N, Tabbara IA. Prognostic implications of genetic aberrations in acute myelogenous leukemia with normal cytogenetics. Am J Hematol 2012;87:69-77.
-
(2012)
Am J Hematol
, vol.87
, pp. 69-77
-
-
Ghanem, H.1
Tank, N.2
Tabbara, I.A.3
-
3
-
-
84055218304
-
Acute myeloid leukemia: 2012 Update on diagnosis, risk stratification, and management
-
Estey EH. Acute myeloid leukemia: 2012 Update on diagnosis, risk stratification, and management. Am J Hematol 2012;87:89-99.
-
(2012)
Am J Hematol
, vol.87
, pp. 89-99
-
-
Estey, E.H.1
-
4
-
-
69149100639
-
Acquired copy number alterations in adult acute myeloid leukemia genomes
-
Walter MJ, Payton JE, Ries RE, et al. Acquired copy number alterations in adult acute myeloid leukemia genomes. Proc Natl Acad Sci USA 2009;106:12950-12955.
-
(2009)
Proc Natl Acad Sci USA
, vol.106
, pp. 12950-12955
-
-
Walter, M.J.1
Payton, J.E.2
Ries, R.E.3
-
5
-
-
59449095868
-
Frequent genomic abnormalities in acute myeloid leukemia/myelodysplastic syndrome with normal karyotype
-
Akagi T, Ogawa S, Dugas M, et al. Frequent genomic abnormalities in acute myeloid leukemia/myelodysplastic syndrome with normal karyotype. Haematologica 2009;94:213-223.
-
(2009)
Haematologica
, vol.94
, pp. 213-223
-
-
Akagi, T.1
Ogawa, S.2
Dugas, M.3
-
6
-
-
77955737728
-
NF1 inactivation in adult acute myelogenous leukemia
-
Parkin B, Ouillette P, Wang Y, et al. NF1 inactivation in adult acute myelogenous leukemia. Clin Cancer Res 2010;16:4135-4147.
-
(2010)
Clin Cancer Res
, vol.16
, pp. 4135-4147
-
-
Parkin, B.1
Ouillette, P.2
Wang, Y.3
-
7
-
-
34047235283
-
Neurofibromatosis 1, and Not TP53, seems to be the main target of chromosome 17 deletions in de novo acute myeloid leukemia
-
author reply, 1152-1153
-
Suela J, Largo C, Ferreira B, et al. Neurofibromatosis 1, and Not TP53, seems to be the main target of chromosome 17 deletions in de novo acute myeloid leukemia. J Clin Oncol 2007;25:1151-1152; author reply, 1152-1153.
-
(2007)
J Clin Oncol
, vol.25
, pp. 1151-1152
-
-
Suela, J.1
Largo, C.2
Ferreira, B.3
-
8
-
-
84859647446
-
Deletion of the tumor-suppressor gene NF1 occurs in 5% of myeloid malignancies and is accompanied by a mutation in the remaining allele in half of the cases
-
Haferlach C, Grossmann V, Kohlmann A, et al. Deletion of the tumor-suppressor gene NF1 occurs in 5% of myeloid malignancies and is accompanied by a mutation in the remaining allele in half of the cases. Leukemia 2012;26:834-839.
-
(2012)
Leukemia
, vol.26
, pp. 834-839
-
-
Haferlach, C.1
Grossmann, V.2
Kohlmann, A.3
-
9
-
-
33748450145
-
Disclosure of candidate genes in acute myeloid leukemia with complex karyotypes using microarray-based molecular characterization
-
Rücker FG, Bullinger L, Schwaenen C, et al. Disclosure of candidate genes in acute myeloid leukemia with complex karyotypes using microarray-based molecular characterization. J Clin Oncol 2006;24:3887-3894.
-
(2006)
J Clin Oncol
, vol.24
, pp. 3887-3894
-
-
Rücker, F.G.1
Bullinger, L.2
Schwaenen, C.3
-
10
-
-
77953929724
-
Neurofibromatosis type 1: Siagnosis and recent advances
-
Upadhyaya M. Neurofibromatosis type 1: Siagnosis and recent advances. Expert Opin Med Diagn 2010;4:307-322.
-
(2010)
Expert Opin Med Diagn
, vol.4
, pp. 307-322
-
-
Upadhyaya, M.1
-
11
-
-
0037961574
-
Von Recklinghausen's neurofibromatosis: Neurofibromatosis type 1
-
Reynolds R, Browning G, Nawroz I, et al. Von Recklinghausen's neurofibromatosis: Neurofibromatosis type 1. Lancet 2003;361:1552-1554.
-
(2003)
Lancet
, vol.361
, pp. 1552-1554
-
-
Reynolds, R.1
Browning, G.2
Nawroz, I.3
-
12
-
-
0030947237
-
Homozygous inactivation of the NF1 gene in bone marrow cells from children with neurofibromatosis type 1 and malignant myeloid disorders
-
Side L, Taylor B, Cayouette M, et al. Homozygous inactivation of the NF1 gene in bone marrow cells from children with neurofibromatosis type 1 and malignant myeloid disorders. N Engl J Med 1997;336:1713-1720.
-
(1997)
N Engl J Med
, vol.336
, pp. 1713-1720
-
-
Side, L.1
Taylor, B.2
Cayouette, M.3
-
13
-
-
76549092856
-
Mitotic recombination and compound-heterozygous mutations are predominant NF1-inactivating mechanisms in children with juvenile myelomonocytic leukemia and neurofibromatosis type 1
-
Steinemann D, Arning L, Praulich I, et al. Mitotic recombination and compound-heterozygous mutations are predominant NF1-inactivating mechanisms in children with juvenile myelomonocytic leukemia and neurofibromatosis type 1. Haematologica 2010;95:320-323.
-
(2010)
Haematologica
, vol.95
, pp. 320-323
-
-
Steinemann, D.1
Arning, L.2
Praulich, I.3
-
14
-
-
77952420655
-
AML with CBFB-MYH11 rearrangement demonstrate RAS pathway alterations in 92% of all cases including a high frequency of NF1 deletions
-
Haferlach C, Dicker F, Kohlmann A, et al. AML with CBFB-MYH11 rearrangement demonstrate RAS pathway alterations in 92% of all cases including a high frequency of NF1 deletions. Leukemia 2010;24:1065-1069.
-
(2010)
Leukemia
, vol.24
, pp. 1065-1069
-
-
Haferlach, C.1
Dicker, F.2
Kohlmann, A.3
-
15
-
-
77950274995
-
Which AML subsets benefit from leukemic cell priming during chemotherapy? Long-term analysis of the ALFA-9802 GM-CSF study
-
Thomas X, Raffoux E, Renneville A, et al. Which AML subsets benefit from leukemic cell priming during chemotherapy? Long-term analysis of the ALFA-9802 GM-CSF study. Cancer 2010;116:1725-1732.
-
(2010)
Cancer
, vol.116
, pp. 1725-1732
-
-
Thomas, X.1
Raffoux, E.2
Renneville, A.3
-
16
-
-
77649202987
-
Randomized study of intensified anthracycline doses for induction and recombinant interleukin-2 for maintenance in patients with acute myeloid leukemia age 50 to 70 years: Results of the ALFA-9801 study
-
Pautas C, Merabet F, Thomas X, et al. Randomized study of intensified anthracycline doses for induction and recombinant interleukin-2 for maintenance in patients with acute myeloid leukemia age 50 to 70 years: Results of the ALFA-9801 study. J Clin Oncol 2010;28:808-814.
-
(2010)
J Clin Oncol
, vol.28
, pp. 808-814
-
-
Pautas, C.1
Merabet, F.2
Thomas, X.3
-
17
-
-
79958072848
-
Impact of post-remission therapy in patients aged 65-70 years with de novo acute myeloid leukemia: A comparison of two concomitant randomized ALFA trials with overlapping age inclusion criteria
-
Itzykson R, Gardin C, Pautas C, et al. Impact of post-remission therapy in patients aged 65-70 years with de novo acute myeloid leukemia: A comparison of two concomitant randomized ALFA trials with overlapping age inclusion criteria. Haematologica 2011;96:837-844.
-
(2011)
Haematologica
, vol.96
, pp. 837-844
-
-
Itzykson, R.1
Gardin, C.2
Pautas, C.3
-
19
-
-
79955708249
-
Comparison of three targeted enrichment strategies on the SOLiD sequencing platform
-
Hedges DJ, Guettouche T, Yang S, et al. Comparison of three targeted enrichment strategies on the SOLiD sequencing platform. PLoS One 2011;6: e18595.
-
(2011)
PLoS One
, vol.6
-
-
Hedges, D.J.1
Guettouche, T.2
Yang, S.3
-
20
-
-
43249089608
-
Leukemia-associated NF1 inactivation in patients with pediatric T-ALL and AML lacking evidence for neurofibromatosis
-
Balgobind BV, Van Vlierberghe P, van den Ouweland AMW, et al. Leukemia-associated NF1 inactivation in patients with pediatric T-ALL and AML lacking evidence for neurofibromatosis. Blood 2008;111:4322-4328.
-
(2008)
Blood
, vol.111
, pp. 4322-4328
-
-
Balgobind, B.V.1
Van Vlierberghe, P.2
van den Ouweland, A.M.W.3
-
21
-
-
77950647203
-
High-resolution single-nucleotide polymorphism array-profiling in myeloproliferative neoplasms identifies novel genomic aberrations
-
Stegelmann F, Bullinger L, Griesshammer M, et al. High-resolution single-nucleotide polymorphism array-profiling in myeloproliferative neoplasms identifies novel genomic aberrations. Haematologica 2010;95:666-669.
-
(2010)
Haematologica
, vol.95
, pp. 666-669
-
-
Stegelmann, F.1
Bullinger, L.2
Griesshammer, M.3
-
22
-
-
54349092877
-
Monosomal karyotype in acute myeloid leukemia: A better indicator of poor prognosis than a complex karyotype
-
Breems DA, Van Putten WLJ, De Greef GE, et al. Monosomal karyotype in acute myeloid leukemia: A better indicator of poor prognosis than a complex karyotype. J Clin Oncol 2008;26:4791-4797.
-
(2008)
J Clin Oncol
, vol.26
, pp. 4791-4797
-
-
Breems, D.A.1
Van Putten, W.L.J.2
De Greef, G.E.3
-
23
-
-
79955814184
-
The inv(3)(q21q26)/t(3;3)(q21;q26) is frequently accompanied by alterations of the RUNX1, KRAS and NRAS and NF1 genes and mediates adverse prognosis both in MDS and in AML: A study in 39 cases of MDS or AML
-
Haferlach C, Bacher U, Haferlach T, et al. The inv(3)(q21q26)/t(3;3)(q21;q26) is frequently accompanied by alterations of the RUNX1, KRAS and NRAS and NF1 genes and mediates adverse prognosis both in MDS and in AML: A study in 39 cases of MDS or AML. Leukemia 2011;25:874-877.
-
(2011)
Leukemia
, vol.25
, pp. 874-877
-
-
Haferlach, C.1
Bacher, U.2
Haferlach, T.3
-
24
-
-
79751530369
-
The prognostic impact and stability of Isocitrate dehydrogenase 2 mutation in adult patients with acute myeloid leukemia
-
Chou W, Lei W, Ko B, et al. The prognostic impact and stability of Isocitrate dehydrogenase 2 mutation in adult patients with acute myeloid leukemia. Leukemia 2011;25:246-253.
-
(2011)
Leukemia
, vol.25
, pp. 246-253
-
-
Chou, W.1
Lei, W.2
Ko, B.3
-
25
-
-
84856596417
-
Inactivation of polycomb repressive complex 2 components in myeloproliferative and myelodysplastic/myeloproliferative neoplasms
-
Score J, Hidalgo-Curtis C, Jones AV, et al. Inactivation of polycomb repressive complex 2 components in myeloproliferative and myelodysplastic/myeloproliferative neoplasms. Blood 2012;119:1208-1213.
-
(2012)
Blood
, vol.119
, pp. 1208-1213
-
-
Score, J.1
Hidalgo-Curtis, C.2
Jones, A.V.3
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