Monogenic models of absence epilepsy. windows into the complex balance between inhibition and excitation in thalamocortical microcircuits

Progress in Brain Research

Volumn 213, Issue C, 2014, Pages 223-252

  Maheshwari, Atul ^a   Noebels, Jeffrey L ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

Corticothalamic; Disinhibition; Feedforward inhibition; Gene; Ion channel; T type calcium channels; Tonic inhibition

Indexed keywords

4 AMINOBUTYRIC ACID A RECEPTOR; AMPA RECEPTOR; CALCIUM CHANNEL Q TYPE; CALCIUM CHANNEL T TYPE;

ABSENCE; ARTICLE; EPILEPTOGENESIS; FEEDBACK SYSTEM; FEEDFORWARD DISINHIBITION; FEEDFORWARD INHIBITION; GENE MUTATION; HUMAN; INTERNEURON; LOSS OF FUNCTION MUTATION; MONOGENIC DISORDER; NEGATIVE FEEDBACK; NEOCORTEX; NERVE CELL INHIBITION; NERVE CELL NETWORK; NEUROTRANSMITTER RELEASE; NONHUMAN; PATHOGENESIS; PHENOTYPE; PRIORITY JOURNAL; PYRAMIDAL NERVE CELL; SPIKE WAVE; SYNAPSE; THALAMOCORTICAL TRACT; THALAMUS RETICULAR NUCLEUS; TONIC INHIBITION; ANIMAL; BIOLOGICAL MODEL; BRAIN CORTEX; GENETICS; NERVE TRACT; PATHOPHYSIOLOGY; THALAMUS;

ANIMALS; CEREBRAL CORTEX; EPILEPSY, ABSENCE; HUMANS; MODELS, GENETIC; NEURAL PATHWAYS; THALAMUS;

EID: 84923652505     PISSN: 00796123     EISSN: 18757855     Source Type: Book Series    
DOI: 10.1016/B978-0-444-63326-2.00012-0     Document Type: Chapter

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