Ablation of P/Q-type Ca2+ channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the α(1A)-subunit

Proceedings of the National Academy of Sciences of the United States of America

Volumn 96, Issue 26, 1999, Pages 15245-15250

  Jun, Kisun ^a   Piedras Rentería, Erika S ^a,b   Smith, Stephen M ^a,b   Wheeler, David B ^b   Lee, Seong Beom ^a   Lee, Taehoon G ^a   Chin, Hemin ^c   Adams, Michael E ^b   Scheller, Richard H ^b   Tsien, Richard W ^b   Shin, Hee Sup ^a,d  

^a Pohang University of Science and Technology (POSTECH)   (South Korea)

^b STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c NATIONAL INSTITUTES OF HEALTH   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM CHANNEL; CALCIUM CHANNEL BLOCKING AGENT; CALCIUM ION; COMPLEMENTARY DNA; NIMODIPINE; OMEGA CONOTOXIN GVIA; OMEGA CONOTOXIN MVIIA;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATAXIA; BRAIN SLICE; CALCIUM CURRENT; GENE MUTATION; MEMBRANE CHANNEL; MOUSE; NEUROTRANSMISSION; NONHUMAN; PRIORITY JOURNAL; SYNAPTIC TRANSMISSION;

ANIMALS; ATAXIA; CALCIUM; CALCIUM CHANNELS; CALCIUM CHANNELS, P-TYPE; CALCIUM CHANNELS, Q-TYPE; CEREBELLUM; ELECTRIC CONDUCTIVITY; HIPPOCAMPUS; MICE; MICE, KNOCKOUT; NERVE TISSUE PROTEINS; NERVOUS SYSTEM DISEASES; PURKINJE CELLS; SYNAPTIC TRANSMISSION;

ANIMALIA; ATAXIA;

EID: 0033593005     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.96.26.15245     Document Type: Article

References (53)