Quercetin attenuates cell apoptosis of oxidant-stressed SK-N-MC cells while suppressing up-regulation of the defensive element, HIF-1α

Neuroscience

Volumn 277, Issue , 2014, Pages 780-793

  Roshanzamir, F ^a   Yazdanparast, R ^a  

^a UNIVERSITY OF TEHRAN   (Iran)

Author keywords

HIF 1; Neurodegenerative diseases; Notch; Oxidative stress; Quercetin

Indexed keywords

CARBONYL DERIVATIVE; CATALASE; GLUTATHIONE; GLUTATHIONE PEROXIDASE; HYDROGEN PEROXIDE; HYPOXIA INDUCIBLE FACTOR 1ALPHA; NOTCH1 RECEPTOR; PROTEIN BAX; PROTEIN BCL 2; PROTEIN P53; QUERCETIN; REACTIVE OXYGEN METABOLITE; SMALL INTERFERING RNA; TRANSCRIPTION FACTOR FKHRL1; TRANSCRIPTION FACTOR HES 1; BAX PROTEIN, HUMAN; FORKHEAD TRANSCRIPTION FACTOR; FOXO3 PROTEIN, HUMAN; HIF1A PROTEIN, HUMAN; NEUROPROTECTIVE AGENT; NOTCH1 PROTEIN, HUMAN;

APOPTOSIS; ARTICLE; CELL DEATH; CELL SURVIVAL; CELL VIABILITY; CONTROLLED STUDY; CYTOTOXICITY; DOWN REGULATION; DRUG MECHANISM; DRUG STRUCTURE; ENZYME ACTIVITY; GENE SILENCING; HUMAN; HUMAN CELL; LIPID PEROXIDATION; MOLECULAR INTERACTION; NEUROBLASTOMA CELL LINE; NEUROPROTECTION; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN DOMAIN; SIGNAL TRANSDUCTION; SK N MC CELL LINE; UPREGULATION; DRUG EFFECTS; METABOLISM; PHYSIOLOGY; PROTEIN CARBONYLATION; TUMOR CELL LINE; CELL COUNT; DEGENERATIVE DISEASE; DRUG DESIGN; INTRACELLULAR SIGNALING;

APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CATALASE; CELL LINE, TUMOR; CELL SURVIVAL; FORKHEAD TRANSCRIPTION FACTORS; GLUTATHIONE; GLUTATHIONE PEROXIDASE; HUMANS; HYDROGEN PEROXIDE; HYPOXIA-INDUCIBLE FACTOR 1, ALPHA SUBUNIT; LIPID PEROXIDATION; NEUROPROTECTIVE AGENTS; PROTEIN CARBONYLATION; QUERCETIN; REACTIVE OXYGEN SPECIES; RECEPTOR, NOTCH1; SIGNAL TRANSDUCTION; TUMOR SUPPRESSOR PROTEIN P53; UP-REGULATION;

EID: 84907029474     PISSN: 03064522     EISSN: 18737544     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2014.07.036     Document Type: Article

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