Molecular mechanisms that differentiate apoptosis from programmed necrosis

Toxicologic Pathology

Volumn 41, Issue 2, 2013, Pages 227-234

  Dorn, Gerald W ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

apoptosis; mitochondrial permeability transition pore; necrosis

Indexed keywords

ADENOSINE TRIPHOSPHATE; PERMETHRIN; PROTEIN BCL 2;

APOPTOSIS; ARTICLE; CALORIC INTAKE; CELL DEATH; CELL DIFFERENTIATION; CELL ENLARGEMENT; CELL LOSS; CELL PROLIFERATION; CHROMATIN CONDENSATION; CONGESTIVE CARDIOMYOPATHY; DEPOLARIZATION; ELECTRON TRANSPORT; ENDOPLASMIC RETICULUM; GENE DELETION; GENE EXPRESSION; GENETIC MANIPULATION; HEART FAILURE; HEART MUSCLE CELL; HEART VENTRICLE HYPERTROPHY; HUMAN; INSULIN RELEASE; MITOCHONDRIAL PERMEABILITY; MITOCHONDRIAL RESPIRATION; MITOCHONDRION; MITOCHONDRION SWELLING; MOLECULAR MECHANICS; MUSCLE CELL; NECROPTOSIS; NICK END LABELING; NON INSULIN DEPENDENT DIABETES MELLITUS; NUTRITIONAL STATUS; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; PROTEIN PROCESSING; TRANSMISSION ELECTRON MICROSCOPY;

ANIMALS; APOPTOSIS; HUMANS; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MOLECULAR BIOLOGY; NECROSIS; SIGNAL TRANSDUCTION;

EID: 84875753808     PISSN: 01926233     EISSN: 15331601     Source Type: Journal    
DOI: 10.1177/0192623312466961     Document Type: Article

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