Mitochondrial death protein Nix is induced in cardiac hypertrophy and triggers apoptotic cardiomyopathy

Nature Medicine

Volumn 8, Issue 7, 2002, Pages 725-730

  Yussman, Martin G ^a   Toyokawa, Tsuyoshi ^a   Odley, Amy ^a   Lynch, Roy A ^c   Wu, Guangyu ^a,c   Colbert, Melissa C ^c   Aronow, Bruce J ^c   Lorenz, John N ^b   Dorn, Gerald W ^a,c  

^a Department of Internal Medicine ^*  (United States)

^b Molec and Cell Phys   (United States)

^c UNIVERSITY OF CINCINNATI MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; CELL PROTEIN; CYTOCHROME C; GUANINE NUCLEOTIDE BINDING PROTEIN; ISOPROTEIN; PROTEIN NIX; UNCLASSIFIED DRUG;

ANALYTIC METHOD; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CARDIOMYOPATHY; CONTROLLED STUDY; DIMERIZATION; ENZYME ACTIVATION; ENZYME RELEASE; FIBROBLAST; GENETIC ANALYSIS; GENETIC ORGANIZATION; HEART FAILURE; HEART MUSCLE CELL; HEART VENTRICLE HYPERTROPHY; HUMAN; HUMAN CELL; IN VIVO STUDY; MALE; MITOCHONDRION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; REGULATORY MECHANISM;

ANIMALS; APOPTOSIS; CARDIOMYOPATHY, HYPERTROPHIC; CLONING, MOLECULAR; HUMANS; MEMBRANE PROTEINS; MICE; MICE, TRANSGENIC; MITOCHONDRIA; MOLECULAR SEQUENCE DATA; PROTO-ONCOGENE PROTEINS; RECOMBINANT PROTEINS; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; TUMOR SUPPRESSOR PROTEINS;

ANIMALIA;

EID: 0036061644     PISSN: 10788956     EISSN: None     Source Type: Journal    
DOI: 10.1038/nm719     Document Type: Article

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