Mitochondria: The headquarters in ischemia-induced neuronal death

Central Nervous System Agents in Medicinal Chemistry

Volumn 11, Issue 2, 2011, Pages 98-106

  Jordan, Joaquin ^a   de Groot, Piet W J ^b   Galindo, Maria F ^c  

^a Universidad de Castilla La Mancha   (Spain)

^b UNIVERSITY OF CASTILLA LA MANCHA   (Spain)

^c HOSPITAL GENERAL UNIVERSITARIO DE ALBACETE   (Spain)

Author keywords

Apoptosis; Minocycline; Mitochondrial permeability; Necrosis; Neuroprotection; Pharmacological target; Stroke

Indexed keywords

APOPTOSIS INDUCING FACTOR; APOPTOTIC PROTEASE ACTIVATING FACTOR 1; CALCIUM; CASPASE 9; CYTOCHROME C; ENDONUCLEASE G; MINOCYCLINE; PLACEBO; REACTIVE OXYGEN METABOLITE; TISSUE PLASMINOGEN ACTIVATOR; VOLTAGE DEPENDENT ANION CHANNEL;

APOPTOSIS; BRAIN ISCHEMIA; CALCIUM HOMEOSTASIS; CALCIUM TRANSPORT; CELL DEATH; CELL MEMBRANE PERMEABILITY; CELL STRESS; DRUG HALF LIFE; DRUG TOLERABILITY; ENZYME METABOLISM; HUMAN; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; NERVE CELL; PROTEIN BINDING; PROTEIN SECRETION; REVIEW; SIGNAL TRANSDUCTION; STROKE;

EID: 79959319233     PISSN: 18715249     EISSN: 18756166     Source Type: Journal    
DOI: 10.2174/187152411796011358     Document Type: Review

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