Biochemistry: A pore way to die

Nature

Volumn 434, Issue 7033, 2005, Pages 578-579

  Halestrap, Andrew ^a  

^a UNIVERSITY OF BRISTOL   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

CELLS; LIVING SYSTEMS STUDIES; PATHOLOGY;

MICE; MITOCHONDRIA; PATHOLOGICAL DAMAGES;

BIOCHEMISTRY;

ADENINE NUCLEOTIDE TRANSLOCASE; ADENOSINE TRIPHOSPHATE; CALCIUM; CYCLOPHILIN; CYCLOPHILIN D; CYCLOSPORIN A; CYTOCHROME C; OXYGEN RADICAL; PROTEIN BCL 2; UNCLASSIFIED DRUG;

BIOCHEMISTRY;

APOPTOSIS; CONFORMATIONAL TRANSITION; HEART INJURY; HEART MUSCLE ISCHEMIA; HEART MUSCLE NECROSIS; MEMBRANE PERMEABILITY; MITOCHONDRIAL MEMBRANE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; REPERFUSION INJURY; SHORT SURVEY; ANIMAL; CHEMISTRY; CYTOLOGY; DRUG EFFECT; GENETICS; HEART MUSCLE CELL; METABOLISM; MITOCHONDRION; MITOCHONDRION SWELLING; NECROSIS; NOTE; PATHOLOGY; PHYSIOLOGY;

ANIMALS; APOPTOSIS; CALCIUM; CYCLOPHILINS; MICE; MITOCHONDRIA; MITOCHONDRIAL ADP, ATP TRANSLOCASES; MITOCHONDRIAL SWELLING; MYOCARDIAL ISCHEMIA; MYOCARDIAL REPERFUSION INJURY; MYOCYTES, CARDIAC; NECROSIS; OXIDATIVE STRESS;

EID: 15844404722     PISSN: 00280836     EISSN: None     Source Type: Journal    
DOI: 10.1038/434578a     Document Type: Short Survey

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