Mitochondrial-mediated suppression of ROS production upon exposure of neurons to lethal stress: Mitochondrial targeted preconditioning

Advanced Drug Delivery Reviews

Volumn 60, Issue 13-14, 2008, Pages 1471-1477

  Busija, David W ^a   Gaspar, Tamas ^a   Domoki, Ferenc ^a,b   Katakam, Prasad V ^a   Bari, Ferenc ^b  

^a WAKE FOREST UNIVERSITY HEALTH SCIENCES   (United States)

^b UNIVERSITY OF SZEGED   (Hungary)

Author keywords

Anoxia; Apoptosis; ATP sensitive potassium channels; Brain; Excitotoxicity; Ischemia; Mitochondria; Necrosis; Oxygen glucose deprivation

Indexed keywords

CHEMICAL REACTIONS; PHOSPHORYLATION; POTASSIUM;

ANOXIA; APOPTOSIS; ATP-SENSITIVE POTASSIUM CHANNELS; BRAIN; EXCITOTOXICITY; ISCHEMIA; MITOCHONDRIA; NECROSIS; OXYGEN GLUCOSE DEPRIVATION;

CELL MEMBRANES;

1 (2 HYDROXY 5 TRIFLUOROMETHYLPHENYL) 5 TRIFLUOROMETHYL 2(3H) BENZIMIDAZOLONE; ADENOSINE TRIPHOSPHATE; ADENOSINE TRIPHOSPHATE SENSITIVE POTASSIUM CHANNEL; LARGE CONDUCTANCE CALCIUM ACTIVATED POTASSIUM CHANNEL; REACTIVE OXYGEN METABOLITE;

ANOXIA; APOPTOSIS; BRAIN ISCHEMIA; BRAIN MITOCHONDRION; CALCIUM CELL LEVEL; CELL DEATH; CELL PROTECTION; CELL STRESS; EXCITOTOXICITY; HUMAN; NONHUMAN; OXIDATIVE PHOSPHORYLATION; OXIDATIVE STRESS; PATHOPHYSIOLOGY; PRIORITY JOURNAL; RESPIRATORY CHAIN; REVIEW;

ANIMALS; BENZIMIDAZOLES; BENZOPYRANS; BRAIN ISCHEMIA; CELL DEATH; ELECTRON TRANSPORT; HUMANS; IMIDAZOLES; ION CHANNEL GATING; ISCHEMIC PRECONDITIONING; KATP CHANNELS; LARGE-CONDUCTANCE CALCIUM-ACTIVATED POTASSIUM CHANNELS; MITOCHONDRIA; MITOCHONDRIAL MEMBRANES; NEURONS; OXIDATIVE PHOSPHORYLATION; REACTIVE OXYGEN SPECIES;

EID: 51249100204     PISSN: 0169409X     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.addr.2008.03.020     Document Type: Review

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