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Volumn 112, Issue 3, 2002, Pages 707-716
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The mechanisms of neuronal death produced by mitochondrial toxin 3-nitropropionic acid: The roles of N-methyl-D-aspartate glutamate receptors and mitochondrial calcium overload
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Author keywords
Caspase; Mitochondrial membrane potential; Mitochondrial permeability transition; Reactive oxygen species
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Indexed keywords
3 NITROPROPIONIC ACID;
CALCIUM ION;
CASPASE 3;
DIZOCILPINE;
N METHYL DEXTRO ASPARTIC ACID RECEPTOR;
REACTIVE OXYGEN METABOLITE;
RUTHENIUM RED;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
BRAIN MITOCHONDRION;
CALCIUM CELL LEVEL;
CELL SURVIVAL;
CONTROLLED STUDY;
DRUG EFFECT;
ENZYME ACTIVATION;
ENZYME INACTIVATION;
MEMBRANE DEPOLARIZATION;
MITOCHONDRIAL MEMBRANE;
NERVE CELL CULTURE;
NERVE CELL NECROSIS;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
ANIMALS;
CALCIUM;
CELL DEATH;
ELECTROPHYSIOLOGY;
EMBRYO;
INTRACELLULAR MEMBRANES;
MITOCHONDRIA;
NEURONS;
NEUROTOXINS;
NITRO COMPOUNDS;
OSMOLAR CONCENTRATION;
PERMEABILITY;
PROPIONIC ACIDS;
RATS;
RATS, WISTAR;
REACTIVE OXYGEN SPECIES;
RECEPTORS, N-METHYL-D-ASPARTATE;
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EID: 0037025058
PISSN: 03064522
EISSN: None
Source Type: Journal
DOI: 10.1016/S0306-4522(02)00097-0 Document Type: Article |
Times cited : (45)
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References (51)
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