The complex interplay between autophagy, apoptosis, and necrotic signals promotes T-cell homeostasis

Immunological Reviews

Volumn 236, Issue 1, 2010, Pages 95-109

  Walsh, Craig M ^a   Edinger, Aimee L ^b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Apoptosis; Autophagy; Homeostasis; Programmed necrosis; T cells; Tolerance

Indexed keywords

CASPASE 8; DEATH RECEPTOR; FAS ASSOCIATED DEATH DOMAIN PROTEIN; MITOCHONDRIAL PROTEIN; PROTEIN BCL 2; PROTEIN SERINE THREONINE KINASE; PROTEIN ULK 1; PROTEIN ULK 2; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR RIPK 1; TUMOR NECROSIS FACTOR RECEPTOR ASSOCIATED DEATH DOMAIN PROTEIN; UNCLASSIFIED DRUG;

ADAPTIVE IMMUNITY; ANTIGEN PRESENTATION; APOPTOSIS; AUTOPHAGY; CELL DEATH; CELL EXPANSION; CELL MATURATION; CELL PROLIFERATION; CELL SURVIVAL; EFFECTOR CELL; ENZYME ACTIVITY; HOMEOSTASIS; IMMUNE RESPONSE; IMMUNOLOGICAL TOLERANCE; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; REVIEW; SIGNAL TRANSDUCTION; T LYMPHOCYTE; T LYMPHOCYTE ACTIVATION;

ANIMALS; APOPTOSIS; AUTOPHAGY; HOMEOSTASIS; HUMANS; MODELS, IMMUNOLOGICAL; NECROSIS; SIGNAL TRANSDUCTION; T-LYMPHOCYTES;

EID: 77953783023     PISSN: 01052896     EISSN: 1600065X     Source Type: Journal    
DOI: 10.1111/j.1600-065X.2010.00919.x     Document Type: Review

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