-
1
-
-
0015383455
-
Apoptosis: A basic biological phenomenon with wide-ranging implications in tissue kinetics
-
Kerr JF, Wyllie AH, Currie AR. Apoptosis: a basic biological phenomenon with wide-ranging implications in tissue kinetics. Br J Cancer. 1972;26(4):239-57.
-
(1972)
Br J Cancer.
, vol.26
, Issue.4
, pp. 239-257
-
-
Kerr, J.F.1
Wyllie, A.H.2
Currie, A.R.3
-
2
-
-
0015880897
-
The morphology of various types of cell death in prenatal tissues
-
Schweichel JU, Merker HJ. The morphology of various types of cell death in prenatal tissues. Teratology. 1973;7(3):253-66.
-
(1973)
Teratology.
, vol.7
, Issue.3
, pp. 253-266
-
-
Schweichel, J.U.1
Merker, H.J.2
-
3
-
-
41149118513
-
How dying cells alert the immune system to danger
-
Kono H, Rock KL. How dying cells alert the immune system to danger. Nat Rev Immunol. 2008;8(4):279-89.
-
(2008)
Nat Rev Immunol.
, vol.8
, Issue.4
, pp. 279-289
-
-
Kono, H.1
Rock, K.L.2
-
4
-
-
77955403878
-
A critical cysteine is required for HMGB1 binding to Toll-like receptor 4 and activation of macrophage cytokine release
-
Yang H et al. A critical cysteine is required for HMGB1 binding to Toll-like receptor 4 and activation of macrophage cytokine release. Proc Natl Acad Sci U S A. 2010;107(26): 11942-7.
-
(2010)
Proc Natl Acad Sci U S A.
, vol.107
, Issue.26
, pp. 11942-11947
-
-
Yang, H.1
-
5
-
-
77958184901
-
Manipulation of host cell death pathways during microbial infections
-
LamkanfiM, Dixit VM. Manipulation of host cell death pathways during microbial infections. Cell Host Microbe. 2010;8(1):44-54.
-
(2010)
Cell Host Microbe.
, vol.8
, Issue.1
, pp. 44-54
-
-
Lamkanfi, M.1
Dixit, V.M.2
-
6
-
-
77957105977
-
Molecular mechanisms of necroptosis: An ordered cellular explosion
-
Vandenabeele P et al. Molecular mechanisms of necroptosis: an ordered cellular explosion. Nat Rev Mol Cell Biol. 2010;11(10):700-14.
-
(2010)
Nat Rev Mol Cell Biol.
, vol.11
, Issue.10
, pp. 700-714
-
-
Vandenabeele, P.1
-
7
-
-
0023697978
-
Tumor necrosis factor can induce both apoptic and necrotic forms of cell lysis
-
Laster SM, Wood JG, Gooding LR. Tumor necrosis factor can induce both apoptic and necrotic forms of cell lysis. J Immunol. 1988;141(8):2629-34.
-
(1988)
J Immunol.
, vol.141
, Issue.8
, pp. 2629-2634
-
-
Laster, S.M.1
Wood, J.G.2
Gooding, L.R.3
-
8
-
-
0032482169
-
Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor
-
Vercammen D et al. Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor. J Exp Med. 1998;187(9):1477-85.
-
(1998)
J Exp Med.
, vol.187
, Issue.9
, pp. 1477-1485
-
-
Vercammen, D.1
-
9
-
-
0032494143
-
Dual signaling of the Fas receptor: Initiation of both apoptotic and necrotic cell death pathways
-
Vercammen D et al. Dual signaling of the Fas receptor: initiation of both apoptotic and necrotic cell death pathways. J Exp Med. 1998;188(5):919-30.
-
(1998)
J Exp Med.
, vol.188
, Issue.5
, pp. 919-930
-
-
Vercammen, D.1
-
10
-
-
5944233768
-
Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule
-
Holler N et al. Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule. Nat Immunol. 2000;1(6):489-95.
-
(2000)
Nat Immunol.
, vol.1
, Issue.6
, pp. 489-495
-
-
Holler, N.1
-
11
-
-
56249132503
-
Antigen-mediated T cell expansion regulated by parallel pathways of death
-
Ch'en IL et al. Antigen-mediated T cell expansion regulated by parallel pathways of death. Proc Natl Acad Sci U S A. 2008;105(45):17463-8.
-
(2008)
Proc Natl Acad Sci U S A.
, vol.105
, Issue.45
, pp. 17463-17468
-
-
Ch'en, I.L.1
-
12
-
-
79955484750
-
Mechanisms of necroptosis in T cells
-
Ch'en IL et al. Mechanisms of necroptosis in T cells. J Exp Med. 2011;208(4):633-41.
-
(2011)
J Exp Med.
, vol.208
, Issue.4
, pp. 633-641
-
-
Ch'en, I.L.1
-
13
-
-
80051481307
-
RIP1-dependent and independent effects of necrostatin-1 in necrosis and T cell activation
-
Cho Y et al. RIP1-dependent and independent effects of necrostatin-1 in necrosis and T cell activation. PloS One. 2011;6(8):e23209.
-
(2011)
PloS One.
, vol.6
, Issue.8
, pp. e23209
-
-
Cho, Y.1
-
14
-
-
84055181328
-
Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3-mediated pathway
-
He S et al. Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3-mediated pathway. Proc Natl Acad Sci U S A. 2011;108(50):20054-9.
-
(2011)
Proc Natl Acad Sci U S A.
, vol.108
, Issue.50
, pp. 20054-20059
-
-
He, S.1
-
15
-
-
84858017083
-
Heme induces programmed necrosis on macrophages through autocrine TNF and ROS production
-
Fortes GB et al. Heme induces programmed necrosis on macrophages through autocrine TNF and ROS production. Blood. 2012;119(10):2368-75.
-
(2012)
Blood.
, vol.119
, Issue.10
, pp. 2368-2375
-
-
Fortes, G.B.1
-
16
-
-
84871028547
-
CIAP1 and cIAP2 limit macrophage necroptosis by inhibiting Rip1 and Rip3 activation
-
McComb S et al. cIAP1 and cIAP2 limit macrophage necroptosis by inhibiting Rip1 and Rip3 activation. Cell Death Differ. 2012;19(11):1791-801.
-
(2012)
Cell Death Differ.
, vol.19
, Issue.11
, pp. 1791-1801
-
-
McComb, S.1
-
17
-
-
0028891783
-
Apoptosis, oncosis, and necrosis. An overview of cell death
-
Majno G, Joris I. Apoptosis, oncosis, and necrosis. An overview of cell death. Am J Pathol. 1995;146(1):3-15.
-
(1995)
Am J Pathol.
, vol.146
, Issue.1
, pp. 3-15
-
-
Majno, G.1
Joris, I.2
-
18
-
-
0016723594
-
An endotoxin-induced serum factor that causes necrosis of tumors
-
Carswell EA et al. An endotoxin-induced serum factor that causes necrosis of tumors. Proc Natl Acad Sci U S A. 1975;72(9):3666-70.
-
(1975)
Proc Natl Acad Sci U S A.
, vol.72
, Issue.9
, pp. 3666-3670
-
-
Carswell, E.A.1
-
19
-
-
0347065342
-
A role for tumor necrosis factor receptor-2 and receptor-interacting protein in programmed necrosis and antiviral responses
-
Chan FK et al. A role for tumor necrosis factor receptor-2 and receptor-interacting protein in programmed necrosis and antiviral responses. J Biol Chem. 2003;278(51):51613-21.
-
(2003)
J Biol Chem.
, vol.278
, Issue.51
, pp. 51613-51621
-
-
Chan, F.K.1
-
20
-
-
66449133280
-
Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation
-
Cho YS et al. Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation. Cell. 2009;137(6):1112-23.
-
(2009)
Cell.
, vol.137
, Issue.6
, pp. 1112-1123
-
-
Cho, Y.S.1
-
21
-
-
66749183275
-
Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-α
-
He S et al. Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-α. Cell. 2009;137(6):1100-11.
-
(2009)
Cell.
, vol.137
, Issue.6
, pp. 1100-1111
-
-
He, S.1
-
22
-
-
67650812332
-
RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis
-
Zhang DW et al. RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis. Science. 2009;325(5938):332-6.
-
(2009)
Science.
, vol.325
, Issue.5938
, pp. 332-336
-
-
Zhang, D.W.1
-
23
-
-
0041853690
-
Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes
-
Micheau O, Tschopp J. Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes. Cell. 2003;114(2):181-90.
-
(2003)
Cell.
, vol.114
, Issue.2
, pp. 181-190
-
-
Micheau, O.1
Tschopp, J.2
-
24
-
-
33646034316
-
Activation of IKK by TNFalpha requires site-specific ubiquitination of RIP1 and polyubiquitin binding by NEMO
-
Ea CK et al. Activation of IKK by TNFalpha requires site-specific ubiquitination of RIP1 and polyubiquitin binding by NEMO. Mol Cell. 2006;22(2):245-57.
-
(2006)
Mol Cell.
, vol.22
, Issue.2
, pp. 245-257
-
-
Ea, C.K.1
-
25
-
-
33744951304
-
Ubiquitination of RIP is required for tumor necrosis factor alpha-induced NF-kappaB activation
-
Li H et al. Ubiquitination of RIP is required for tumor necrosis factor alpha-induced NF-kappaB activation. J Biol Chem. 2006;281(19):13636-43.
-
(2006)
J Biol Chem.
, vol.281
, Issue.19
, pp. 13636-13643
-
-
Li, H.1
-
26
-
-
78650300883
-
C-IAP1 and UbcH5 promote K11-linked polyubiquitination of RIP1 in TNF signalling
-
Dynek JN et al. c-IAP1 and UbcH5 promote K11-linked polyubiquitination of RIP1 in TNF signalling. EMBO J. 2010;29(24):4198-209.
-
(2010)
EMBO J
, vol.29
, Issue.24
, pp. 4198-4209
-
-
Dynek, J.N.1
-
27
-
-
79953240109
-
Linear ubiquitination prevents inflammation and regulates immune signalling
-
Gerlach B et al. Linear ubiquitination prevents inflammation and regulates immune signalling. Nature. 2011;471(7340):591-6.
-
(2011)
Nature.
, vol.471
, Issue.7340
, pp. 591-596
-
-
Gerlach, B.1
-
28
-
-
33847251527
-
Ubiquitination of RIP1 regulates an NF-kappaB-independent cell-death switch in TNF signaling
-
O'Donnell MA et al. Ubiquitination of RIP1 regulates an NF-kappaB-independent cell-death switch in TNF signaling. Curr Biol. 2007;17(5):418-24.
-
(2007)
Curr Biol.
, vol.17
, Issue.5
, pp. 418-424
-
-
O'donnell, M.A.1
-
29
-
-
84864389178
-
NEMO inhibits programmed necrosis in an NFkappaB-independent manner by restraining RIP1
-
O'Donnell MA et al. NEMO inhibits programmed necrosis in an NFkappaB-independent manner by restraining RIP1. PloS One. 2012;7(7):e41238.
-
(2012)
PloS One.
, vol.7
, Issue.7
, pp. e41238
-
-
O'donnell, M.A.1
-
30
-
-
57649181391
-
Identification of a molecular signaling network that regulates a cellular necrotic cell death pathway
-
Hitomi J et al. Identification of a molecular signaling network that regulates a cellular necrotic cell death pathway. Cell. 2008;135(7):1311-23.
-
(2008)
Cell.
, vol.135
, Issue.7
, pp. 1311-1323
-
-
Hitomi, J.1
-
31
-
-
79952623655
-
CIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production
-
Vanlangenakker N et al. cIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production. Cell Death Differ. 2011;18(4):656-65.
-
(2011)
Cell Death Differ.
, vol.18
, Issue.4
, pp. 656-665
-
-
Vanlangenakker, N.1
-
32
-
-
0032583164
-
Caspase-independent cell killing by Fas-associated protein with death domain
-
Kawahara A et al. Caspase-independent cell killing by Fas-associated protein with death domain. J Cell Biol. 1998;143(5):1353-60.
-
(1998)
J Cell Biol.
, vol.143
, Issue.5
, pp. 1353-1360
-
-
Kawahara, A.1
-
33
-
-
1542289739
-
Differential signaling to apoptotic and necrotic cell death by Fasassociated death domain protein FADD
-
Vanden Berghe T et al. Differential signaling to apoptotic and necrotic cell death by Fasassociated death domain protein FADD. J Biol Chem. 2004;279(9):7925-33.
-
(2004)
J Biol Chem.
, vol.279
, Issue.9
, pp. 7925-7933
-
-
Vanden Berghe, T.1
-
34
-
-
0033214236
-
Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis
-
Lin Y et al. Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis. Genes Dev. 1999;13(19):2514-26.
-
(1999)
Genes Dev.
, vol.13
, Issue.19
, pp. 2514-2526
-
-
Lin, Y.1
-
35
-
-
34548437549
-
Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain
-
Feng S et al. Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain. Cell Signal. 2007;19(10):2056-67.
-
(2007)
Cell Signal.
, vol.19
, Issue.10
, pp. 2056-2067
-
-
Feng, S.1
-
36
-
-
84856160569
-
Caspase 8 inhibits programmed necrosis by processing CYLD
-
O'Donnell MA et al. Caspase 8 inhibits programmed necrosis by processing CYLD. Nat Cell Biol. 2011;13(12):1437-42.
-
(2011)
Nat Cell Biol.
, vol.13
, Issue.12
, pp. 1437-1442
-
-
O'donnell, M.A.1
-
37
-
-
0037088632
-
Identification of a novel homotypic interaction motif required for the phosphorylation of receptor-interacting protein (RIP) by RIP3
-
Sun X et al. Identification of a novel homotypic interaction motif required for the phosphorylation of receptor-interacting protein (RIP) by RIP3. J Biol Chem. 2002;277(11):9505-11.
-
(2002)
J Biol Chem.
, vol.277
, Issue.11
, pp. 9505-9511
-
-
Sun, X.1
-
38
-
-
33644840693
-
Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury
-
Degterev A et al. Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury. Nat Chem Biol. 2005;1(2):112-9.
-
(2005)
Nat Chem Biol.
, vol.1
, Issue.2
, pp. 112-119
-
-
Degterev, A.1
-
39
-
-
42249102086
-
Identification of RIP1 kinase as a specific cellular target of necrostatins
-
Degterev A et al. Identification of RIP1 kinase as a specific cellular target of necrostatins. Nat Chem Biol. 2008;4(5):313-21.
-
(2008)
Nat Chem Biol.
, vol.4
, Issue.5
, pp. 313-321
-
-
Degterev, A.1
-
40
-
-
84864240409
-
The RIP1/RIP3 necrosome forms a functional amyloid signaling complex required for programmed necrosis
-
Li J et al. The RIP1/RIP3 necrosome forms a functional amyloid signaling complex required for programmed necrosis. Cell. 2012;150(2):339-50.
-
(2012)
Cell.
, vol.150
, Issue.2
, pp. 339-350
-
-
Li, J.1
-
41
-
-
77955271788
-
The molecular regulation of programmed necrotic cell injury
-
Moquin D, Chan FK. The molecular regulation of programmed necrotic cell injury. Trends Biochem Sci. 2010;35(8):434-41.
-
(2010)
Trends Biochem Sci.
, vol.35
, Issue.8
, pp. 434-441
-
-
Moquin, D.1
Chan, F.K.2
-
42
-
-
84858374665
-
The amyloid state of proteins in human diseases
-
Eisenberg D, Jucker M. The amyloid state of proteins in human diseases. Cell. 2012;148(6):1188-203.
-
(2012)
Cell.
, vol.148
, Issue.6
, pp. 1188-1203
-
-
Eisenberg, D.1
Jucker, M.2
-
43
-
-
84862907788
-
Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase
-
Sun L et al. Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase. Cell. 2012;148(1-2):213-27.
-
(2012)
Cell.
, vol.148
, Issue.1-2
, pp. 213-227
-
-
Sun, L.1
-
44
-
-
84859467770
-
Mixed lineage kinase domain-like is a key receptor interacting protein 3 downstream component of TNF-induced necrosis
-
Zhao J et al. Mixed lineage kinase domain-like is a key receptor interacting protein 3 downstream component of TNF-induced necrosis. Proc Natl Acad Sci USA. 2012; 109(14):5322-7.
-
(2012)
Proc Natl Acad Sci USA.
, vol.109
, Issue.14
, pp. 5322-5327
-
-
Zhao, J.1
-
45
-
-
84862909353
-
The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways
-
Wang Z et al. The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways. Cell. 2012;148(1-2):228-43.
-
(2012)
Cell.
, vol.148
, Issue.1-2
, pp. 228-243
-
-
Wang, Z.1
-
46
-
-
42649130014
-
PGAM5 tethers a ternary complex containing Keap1 and Nrf2 to mitochondria
-
Lo SC, Hannink M. PGAM5 tethers a ternary complex containing Keap1 and Nrf2 to mitochondria. Exp Cell Res. 2008;314(8):1789-803.
-
(2008)
Exp Cell Res.
, vol.314
, Issue.8
, pp. 1789-1803
-
-
Lo, S.C.1
Hannink, M.2
-
47
-
-
0036715377
-
Tipping the balance between necrosis and apoptosis in human and murine cells treated with interferon and dsRNA
-
Kalai M et al. Tipping the balance between necrosis and apoptosis in human and murine cells treated with interferon and dsRNA. Cell Death Differ. 2002;9(9):981-94.
-
(2002)
Cell Death Differ.
, vol.9
, Issue.9
, pp. 981-994
-
-
Kalai, M.1
-
48
-
-
77956715781
-
Going up in flames: Necrotic cell injury and inflammatory diseases
-
Challa S, Chan FK. Going up in flames: necrotic cell injury and inflammatory diseases. Cell Mol Life Sci. 2010;67(19):3241-53.
-
(2010)
Cell Mol Life Sci.
, vol.67
, Issue.19
, pp. 3241-3253
-
-
Challa, S.1
Chan, F.K.2
-
49
-
-
0036058439
-
Vaccinia virus CrmE encodes a soluble and cell surface tumor necrosis factor receptor that contributes to virus virulence
-
Reading PC, Khanna A, Smith GL. Vaccinia virus CrmE encodes a soluble and cell surface tumor necrosis factor receptor that contributes to virus virulence. Virology. 2002; 292(2):285-98.
-
(2002)
Virology.
, vol.292
, Issue.2
, pp. 285-298
-
-
Reading, P.C.1
Khanna, A.2
Smith, G.L.3
-
50
-
-
0037415598
-
The poxvirus protein A52R targets Toll-like receptor signaling complexes to suppress host defense
-
Harte MT et al. The poxvirus protein A52R targets Toll-like receptor signaling complexes to suppress host defense. J Exp Med. 2003;197(3):343-51.
-
(2003)
J Exp Med.
, vol.197
, Issue.3
, pp. 343-351
-
-
Harte, M.T.1
-
51
-
-
17144366260
-
Vaccinia virus protein A46R targets multiple Toll-like-interleukin-1 receptor adaptors and contributes to virulence
-
Stack J et al. Vaccinia virus protein A46R targets multiple Toll-like-interleukin-1 receptor adaptors and contributes to virulence. J Exp Med. 2005;201(6):1007-18.
-
(2005)
J Exp Med.
, vol.201
, Issue.6
, pp. 1007-1018
-
-
Stack, J.1
-
52
-
-
0030977847
-
Target protease specificity of the viral serpin Crm A. Analysis of five caspases
-
Zhou Q et al. Target protease specificity of the viral serpin CrmA.Analysis of five caspases. J Biol Chem. 1997;272(12):7797-800.
-
(1997)
J Biol Chem.
, vol.272
, Issue.12
, pp. 7797-7800
-
-
Zhou, Q.1
-
53
-
-
0033861807
-
Induction of necrotic-like cell death by tumor necrosis factor alpha and caspase inhibitors: Novel mechanism for killing virus-infected cells
-
Li M, Beg AA. Induction of necrotic-like cell death by tumor necrosis factor alpha and caspase inhibitors: novel mechanism for killing virus-infected cells. J Virol. 2000; 74(16):7470-7.
-
(2000)
J Virol.
, vol.74
, Issue.16
, pp. 7470-7477
-
-
Li, M.1
Beg, A.A.2
-
54
-
-
0030970013
-
Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptors
-
Thome M et al. Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptors. Nature. 1997;386(6624):517-21.
-
(1997)
Nature.
, vol.386
, Issue.6624
, pp. 517-521
-
-
Thome, M.1
-
55
-
-
0030950228
-
A novel family of viral death effector domain-containing molecules that inhibit both CD-95-and tumor necrosis factor receptor-1-induced apoptosis
-
Hu S et al. A novel family of viral death effector domain-containing molecules that inhibit both CD-95-and tumor necrosis factor receptor-1-induced apoptosis. J Biol Chem. 1997;272(15):9621-4.
-
(1997)
J Biol Chem.
, vol.272
, Issue.15
, pp. 9621-9624
-
-
Hu, S.1
-
56
-
-
12644286556
-
Death effector domain-containing herpesvirus and poxvirus proteins inhibit both Fas-and TNFR1-induced apoptosis
-
Bertin J et al. Death effector domain-containing herpesvirus and poxvirus proteins inhibit both Fas-and TNFR1-induced apoptosis. Proc Natl Acad Sci U S A. 1997;94(4):1172-6.
-
(1997)
Proc Natl Acad Sci U S A.
, vol.94
, Issue.4
, pp. 1172-1176
-
-
Bertin, J.1
-
57
-
-
84856231635
-
Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways
-
Mocarski ES, Upton JW, Kaiser WJ. Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways. Nat Rev Immunol. 2012;12(2):79-88.
-
(2012)
Nat Rev Immunol.
, vol.12
, Issue.2
, pp. 79-88
-
-
Mocarski, E.S.1
Upton, J.W.2
Kaiser, W.J.3
-
58
-
-
47749133767
-
Cytomegalovirus M45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1
-
Upton JW, Kaiser WJ, Mocarski ES. Cytomegalovirus M45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1. J Biol Chem. 2008;283(25):16966-70.
-
(2008)
J Biol Chem.
, vol.283
, Issue.25
, pp. 16966-16970
-
-
Upton, J.W.1
Kaiser, W.J.2
Mocarski, E.S.3
-
59
-
-
58749100879
-
Receptor-interacting protein homotypic interaction motif-dependent control of NF-kappa B activation via the DNA-dependent activator of IFN regulatory factors
-
Kaiser WJ, Upton JW, Mocarski ES. Receptor-interacting protein homotypic interaction motif-dependent control of NF-kappa B activation via the DNA-dependent activator of IFN regulatory factors. J Immunol. 2008;181(9):6427-34.
-
(2008)
J Immunol.
, vol.181
, Issue.9
, pp. 6427-6434
-
-
Kaiser, W.J.1
Upton, J.W.2
Mocarski, E.S.3
-
60
-
-
68249113997
-
DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-kappaB
-
Rebsamen M et al. DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-kappaB. EMBO Rep. 2009;10(8):916-22.
-
(2009)
EMBO Rep.
, vol.10
, Issue.8
, pp. 916-922
-
-
Rebsamen, M.1
-
61
-
-
84858420051
-
DAI/ZBP1/DLM-1 complexes with RIP3 to mediate virus-induced programmed necrosis that is targeted by murine cytomegalovirus vIRA
-
Upton JW, Kaiser WJ, Mocarski ES. DAI/ZBP1/DLM-1 complexes with RIP3 to mediate virus-induced programmed necrosis that is targeted by murine cytomegalovirus vIRA. Cell Host Microbe. 2012;11(3):290-7.
-
(2012)
Cell Host Microbe.
, vol.11
, Issue.3
, pp. 290-297
-
-
Upton, J.W.1
Kaiser, W.J.2
Mocarski, E.S.3
-
63
-
-
10744221040
-
The human cytomegalovirus UL45 gene product is a late, virion-associated protein and influences virus growth at low multiplicities of infection
-
Patrone M et al. The human cytomegalovirus UL45 gene product is a late, virion-associated protein and influences virus growth at low multiplicities of infection. J Gen Virol. 2003;84 (Pt 12):3359-70.
-
(2003)
J Gen Virol.
, vol.84
, Issue.PART 12
, pp. 3359-3370
-
-
Patrone, M.1
-
64
-
-
84255162797
-
RIP kinase-dependent necrosis drives lethal systemic inflammatory response syndrome
-
Duprez L et al. RIP kinase-dependent necrosis drives lethal systemic inflammatory response syndrome. Immunity. 2011;35(6):908-18.
-
(2011)
Immunity.
, vol.35
, Issue.6
, pp. 908-918
-
-
Duprez, L.1
-
65
-
-
84865973897
-
Receptor interacting protein kinase mediates necrotic cone but not rod cell death in a mouse model of inherited degeneration
-
Murakami Y et al. Receptor interacting protein kinase mediates necrotic cone but not rod cell death in a mouse model of inherited degeneration. Proc Natl Acad Sci U S A. 2012;109(36):14598-603.
-
(2012)
Proc Natl Acad Sci U S A.
, vol.109
, Issue.36
, pp. 14598-14603
-
-
Murakami, Y.1
-
66
-
-
0032033132
-
The death domain kinase RIP mediates the TNF-induced NF-kappaB signal
-
Kelliher MA et al. The death domain kinase RIP mediates the TNF-induced NF-kappaB signal. Immunity. 1998;8(3):297-303.
-
(1998)
Immunity.
, vol.8
, Issue.3
, pp. 297-303
-
-
Kelliher, M.A.1
-
67
-
-
0037114185
-
Cutting edge: A novel Toll/IL-1 receptor domain-containing adapter that preferentially activates the IFN-beta promoter in the Toll-like receptor signaling
-
Yamamoto M et al. Cutting edge: a novel Toll/IL-1 receptor domain-containing adapter that preferentially activates the IFN-beta promoter in the Toll-like receptor signaling. J Immunol. 2002;169(12):6668-72.
-
(2002)
J Immunol.
, vol.169
, Issue.12
, pp. 6668-6672
-
-
Yamamoto, M.1
-
68
-
-
0037320451
-
TICAM-1, an adaptor molecule that participates in Toll-like receptor 3-mediated interferon-beta induction
-
Oshiumi H et al. TICAM-1, an adaptor molecule that participates in Toll-like receptor 3-mediated interferon-beta induction. Nat Immunol. 2003;4(2):161-7.
-
(2003)
Nat Immunol.
, vol.4
, Issue.2
, pp. 161-167
-
-
Oshiumi, H.1
-
69
-
-
17144413785
-
Apoptosis induced by the toll-like receptor adaptor TRIF is dependent on its receptor interacting protein homotypic interaction motif
-
Kaiser WJ, Offermann MK. Apoptosis induced by the toll-like receptor adaptor TRIF is dependent on its receptor interacting protein homotypic interaction motif. J Immunol. 2005;174(8):4942-52.
-
(2005)
J Immunol.
, vol.174
, Issue.8
, pp. 4942-4952
-
-
Kaiser, W.J.1
Offermann, M.K.2
-
70
-
-
77952241062
-
Proapoptotic signalling through Toll-like receptor-3 involves TRIF-dependent activation of caspase-8 and is under the control of inhibitor of apoptosis proteins in melanoma cells
-
Weber A et al. Proapoptotic signalling through Toll-like receptor-3 involves TRIF-dependent activation of caspase-8 and is under the control of inhibitor of apoptosis proteins in melanoma cells. Cell Death Differ. 2010;17(6):942-51.
-
(2010)
Cell Death Differ.
, vol.17
, Issue.6
, pp. 942-951
-
-
Weber, A.1
-
71
-
-
78650718758
-
Receptor interacting protein kinases mediate retinal detachment-induced photoreceptor necrosis and compensate for inhibition of apoptosis
-
Trichonas G et al. Receptor interacting protein kinases mediate retinal detachment-induced photoreceptor necrosis and compensate for inhibition of apoptosis. Proc Natl Acad Sci U S A. 2010;107(50):21695-700.
-
(2010)
Proc Natl Acad Sci U S A.
, vol.107
, Issue.50
, pp. 21695-21700
-
-
Trichonas, G.1
-
72
-
-
34548564715
-
Necrostatin: A potentially novel cardioprotective agent?
-
Smith CC et al. Necrostatin: a potentially novel cardioprotective agent? Cardiovasc Drugs Ther. 2007;21(4):227-33.
-
(2007)
Cardiovasc Drugs Ther.
, vol.21
, Issue.4
, pp. 227-233
-
-
Smith, C.C.1
-
73
-
-
78650911946
-
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI
-
Northington FJ et al. Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI. J Cereb Blood Flow Metab. 2011;31(1):178-89.
-
(2011)
J Cereb Blood Flow Metab.
, vol.31
, Issue.1
, pp. 178-189
-
-
Northington, F.J.1
-
74
-
-
36849056027
-
The cardioprotective effect of necrostatin requires the cyclophilin-D component of the mitochondrial permeability transition pore
-
Lim SY et al. The cardioprotective effect of necrostatin requires the cyclophilin-D component of the mitochondrial permeability transition pore. Cardiovasc Drugs Ther. 2007;21(6):467-9.
-
(2007)
Cardiovasc Drugs Ther.
, vol.21
, Issue.6
, pp. 467-469
-
-
Lim, S.Y.1
-
75
-
-
79960890578
-
A role for tumor necrosis factor-alpha in ischemia and ischemic preconditioning
-
Watters O, O'Connor JJ. A role for tumor necrosis factor-alpha in ischemia and ischemic preconditioning. J Neuroinflammation. 2011;8:87.
-
(2011)
J Neuroinflammation.
, vol.8
, pp. 87
-
-
Watters, O.1
O'connor, J.J.2
-
77
-
-
0032546387
-
Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1
-
Zhang J et al. Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1. Nature. 1998;392(6673):296-300.
-
(1998)
Nature.
, vol.392
, Issue.6673
, pp. 296-300
-
-
Zhang, J.1
-
78
-
-
7144263731
-
FADD: Essential for embryo development and signaling from some, but not all, inducers of apoptosis
-
Yeh WC et al. FADD: essential for embryo development and signaling from some, but not all, inducers of apoptosis. Science. 1998;279(5358):1954-8.
-
(1998)
Science.
, vol.279
, Issue.5358
, pp. 1954-1958
-
-
Yeh, W.C.1
-
79
-
-
0032143986
-
Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally
-
Varfolomeev EE et al. Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally. Immunity. 1998;9(2):267-76.
-
(1998)
Immunity.
, vol.9
, Issue.2
, pp. 267-276
-
-
Varfolomeev, E.E.1
-
80
-
-
79952780505
-
Functional complementation between FADD and RIP1 in embryos and lymphocytes
-
Zhang H et al. Functional complementation between FADD and RIP1 in embryos and lymphocytes. Nature. 2011;471(7338):373-6.
-
(2011)
Nature.
, vol.471
, Issue.7338
, pp. 373-376
-
-
Zhang, H.1
-
81
-
-
79952811804
-
RIP3 mediates the embryonic lethality of caspase-8-deficient mice
-
Kaiser WJ et al. RIP3 mediates the embryonic lethality of caspase-8-deficient mice. Nature. 2011;471(7338):368-72.
-
(2011)
Nature.
, vol.471
, Issue.7338
, pp. 368-372
-
-
Kaiser, W.J.1
-
82
-
-
79952810024
-
Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis
-
Oberst A et al. Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis. Nature. 2011;471(7338):363-7.
-
(2011)
Nature.
, vol.471
, Issue.7338
, pp. 363-367
-
-
Oberst, A.1
-
83
-
-
84861712290
-
Survival function of the FADD-CASPASE-8-cFLIP(L) complex
-
Dillon CP et al. Survival function of the FADD-CASPASE-8-cFLIP(L) complex. Cell Rep. 2012;1(5):401-7.
-
(2012)
Cell Rep.
, vol.1
, Issue.5
, pp. 401-407
-
-
Dillon, C.P.1
-
84
-
-
70350484946
-
Caspase-8 deficiency in epidermal keratinocytes triggers an inflammatory skin disease
-
Kovalenko A et al. Caspase-8 deficiency in epidermal keratinocytes triggers an inflammatory skin disease. J Exp Med. 2009;206(10):2161-77.
-
(2009)
J Exp Med.
, vol.206
, Issue.10
, pp. 2161-2177
-
-
Kovalenko, A.1
-
85
-
-
80052850704
-
FADD prevents RIP3-mediated epithelial cell necrosis and chronic intestinal inflammation
-
Welz PS et al. FADD prevents RIP3-mediated epithelial cell necrosis and chronic intestinal inflammation. Nature. 2011;477(7364):330-4.
-
(2011)
Nature.
, vol.477
, Issue.7364
, pp. 330-334
-
-
Welz, P.S.1
-
86
-
-
80755132824
-
The adaptor protein FADD protects epidermal keratinocytes from necroptosis in vivo and prevents skin inflammation
-
Bonnet MC et al. The adaptor protein FADD protects epidermal keratinocytes from necroptosis in vivo and prevents skin inflammation. Immunity. 2011;35(4):572-82.
-
(2011)
Immunity.
, vol.35
, Issue.4
, pp. 572-582
-
-
Bonnet, M.C.1
-
87
-
-
84863000898
-
The ubiquitin ligase XIAP recruits LUBAC for NOD2 signaling in inflammation and innate immunity
-
Damgaard RB et al. The ubiquitin ligase XIAP recruits LUBAC for NOD2 signaling in inflammation and innate immunity. Mol Cell. 2012;46(6):746-58.
-
(2012)
Mol Cell.
, vol.46
, Issue.6
, pp. 746-758
-
-
Damgaard, R.B.1
-
88
-
-
79952770123
-
RIG-I RNA helicase activation of IRF3 transcription factor is negatively regulated by caspase-8-mediated cleavage of the RIP1 protein
-
Rajput A et al. RIG-I RNA helicase activation of IRF3 transcription factor is negatively regulated by caspase-8-mediated cleavage of the RIP1 protein. Immunity. 2011;34(3):340-51.
-
(2011)
Immunity.
, vol.34
, Issue.3
, pp. 340-351
-
-
Rajput, A.1
-
89
-
-
82455212812
-
'Necrosome'-induced inflammation: Must cells die for it?
-
Wallach D, Kovalenko A, Kang TB. 'Necrosome'-induced inflammation: must cells die for it? Trends Immunol. 2011;32(11):505-9.
-
(2011)
Trends Immunol.
, vol.32
, Issue.11
, pp. 505-509
-
-
Wallach, D.1
Kovalenko, A.2
Kang, T.B.3
-
90
-
-
2442642691
-
RIP1 is an essential mediator of Toll-like receptor 3-induced NF-kappa B activation
-
Meylan E et al. RIP1 is an essential mediator of Toll-like receptor 3-induced NF-kappa B activation. Nat Immunol. 2004;5(5):503-7.
-
(2004)
Nat Immunol.
, vol.5
, Issue.5
, pp. 503-507
-
-
Meylan, E.1
-
91
-
-
27744561393
-
Rip1 mediates the Trif-dependent toll-like receptor 3-and 4-induced NF-{kappa}B activation but does not contribute to interferon regulatory factor 3 activation
-
Cusson-Hermance N et al. Rip1 mediates the Trif-dependent toll-like receptor 3-and 4-induced NF-{kappa}B activation but does not contribute to interferon regulatory factor 3 activation. J Biol Chem. 2005;280(44):36560-6.
-
(2005)
J Biol Chem.
, vol.280
, Issue.44
, pp. 36560-36566
-
-
Cusson-Hermance, N.1
-
92
-
-
50049125047
-
Function of TRADD in tumor necrosis factor receptor 1 signaling and in TRIF-dependent inflammatory responses
-
Ermolaeva MA et al. Function of TRADD in tumor necrosis factor receptor 1 signaling and in TRIF-dependent inflammatory responses. Nat Immunol. 2008;9(9):1037-46.
-
(2008)
Nat Immunol.
, vol.9
, Issue.9
, pp. 1037-1046
-
-
Ermolaeva, M.A.1
-
93
-
-
43049174990
-
TRADD protein is an essential component of the RIG-like helicase antiviral pathway
-
Michallet MC et al. TRADD protein is an essential component of the RIG-like helicase antiviral pathway. Immunity. 2008;28(5):651-61.
-
(2008)
Immunity.
, vol.28
, Issue.5
, pp. 651-661
-
-
Michallet, M.C.1
-
94
-
-
0842304221
-
Kinase RIP3 is dispensable for normal NF-kappa Bs, signaling by the B-cell and T-cell receptors, tumor necrosis factor receptor 1, and Toll-like receptors 2 and 4
-
Newton K, Sun X, Dixit VM. Kinase RIP3 is dispensable for normal NF-kappa Bs, signaling by the B-cell and T-cell receptors, tumor necrosis factor receptor 1, and Toll-like receptors 2 and 4. Mol Cell Biol. 2004;24(4):1464-9.
-
(2004)
Mol Cell Biol.
, vol.24
, Issue.4
, pp. 1464-1469
-
-
Newton, K.1
Sun, X.2
Dixit, V.M.3
-
95
-
-
0034685982
-
The RIP-like kinase, RIP3, induces apoptosis and NF-kappaB nuclear translocation and localizes to mitochondria
-
Kasof GM et al. The RIP-like kinase, RIP3, induces apoptosis and NF-kappaB nuclear translocation and localizes to mitochondria. FEBS Lett. 2000;473(3):285-91.
-
(2000)
FEBS Lett.
, vol.473
, Issue.3
, pp. 285-291
-
-
Kasof, G.M.1
-
96
-
-
0033546276
-
RIP3, a novel apoptosis-inducing kinase
-
Sun X et al. RIP3, a novel apoptosis-inducing kinase. J Biol Chem. 1999;274(24):16871-5.
-
(1999)
J Biol Chem.
, vol.274
, Issue.24
, pp. 16871-16875
-
-
Sun, X.1
-
97
-
-
84857404572
-
Inhibitor of apoptosis proteins limit RIP3 kinase-dependent interleukin-1 activation
-
Vince JE et al. Inhibitor of apoptosis proteins limit RIP3 kinase-dependent interleukin-1 activation. Immunity. 2012;36(2):215-27.
-
(2012)
Immunity.
, vol.36
, Issue.2
, pp. 215-227
-
-
Vince, J.E.1
-
98
-
-
84857175933
-
Dectin-1 is an extracellular pathogen sensor for the induction and processing of IL-1beta via a noncanonical caspase-8 inflammasome
-
Gringhuis SI et al. Dectin-1 is an extracellular pathogen sensor for the induction and processing of IL-1beta via a noncanonical caspase-8 inflammasome. Nat Immunol. 2012;13(3):246-54.
-
(2012)
Nat Immunol.
, vol.13
, Issue.3
, pp. 246-254
-
-
Gringhuis, S.I.1
-
99
-
-
84866087868
-
AIM2/ASC triggers caspase-8-dependent apoptosis in Francisella-infected caspase-1-deficient macrophages
-
Pierini R et al. AIM2/ASC triggers caspase-8-dependent apoptosis in Francisella-infected caspase-1-deficient macrophages. Cell Death Differ. 2012;19(10):1709-21.
-
(2012)
Cell Death Differ.
, vol.19
, Issue.10
, pp. 1709-1721
-
-
Pierini, R.1
-
100
-
-
80455176839
-
Non-canonical inflammasome activation targets caspase-11
-
Kayagaki N et al. Non-canonical inflammasome activation targets caspase-11. Nature. 2011;479(7371):117-21.
-
(2011)
Nature.
, vol.479
, Issue.7371
, pp. 117-121
-
-
Kayagaki, N.1
-
101
-
-
84864600268
-
TRIF licenses caspase-11-dependent NLRP3 inflammasome activation by gram-negative bacteria
-
Rathinam VA et al. TRIF licenses caspase-11-dependent NLRP3 inflammasome activation by gram-negative bacteria. Cell. 2012;150(3):606-19.
-
(2012)
Cell.
, vol.150
, Issue.3
, pp. 606-619
-
-
Rathinam, V.A.1
-
102
-
-
0029112253
-
Induction of apoptosis in mature T cells by tumour necrosis factor
-
Zheng L et al. Induction of apoptosis in mature T cells by tumour necrosis factor. Nature. 1995;377(6547):348-51.
-
(1995)
Nature.
, vol.377
, Issue.6547
, pp. 348-351
-
-
Zheng, L.1
-
103
-
-
0033007321
-
Mature T lymphocyte apoptosis-immune regulation in a dynamic and unpredictable antigenic environment
-
Lenardo M et al. Mature T lymphocyte apoptosis-immune regulation in a dynamic and unpredictable antigenic environment. Annu Rev Immunol. 1999;17:221-53.
-
(1999)
Annu Rev Immunol.
, vol.17
, pp. 221-253
-
-
Lenardo, M.1
-
104
-
-
0031975910
-
T cell growth cytokines cause the superinduction of molecules mediating antigen-induced T lymphocyte death
-
Zheng L et al. T cell growth cytokines cause the superinduction of molecules mediating antigen-induced T lymphocyte death. J Immunol. 1998;160(2):763-9.
-
(1998)
J Immunol.
, vol.160
, Issue.2
, pp. 763-769
-
-
Zheng, L.1
-
105
-
-
0026568919
-
Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis
-
Watanabe-Fukunaga R et al. Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis. Nature. 1992;356(6367):314-7.
-
(1992)
Nature.
, vol.356
, Issue.6367
, pp. 314-317
-
-
Watanabe-Fukunaga, R.1
-
106
-
-
0028429962
-
The mouse Fas-ligand gene is mutated in gld mice and is part of a TNF family gene cluster
-
Lynch DH et al. The mouse Fas-ligand gene is mutated in gld mice and is part of a TNF family gene cluster. Immunity. 1994;1(2):131-6.
-
(1994)
Immunity.
, vol.1
, Issue.2
, pp. 131-136
-
-
Lynch, D.H.1
-
107
-
-
0031080703
-
ALPS: An autoimmune human lymphoproliferative syndrome associated with abnormal lymphocyte apoptosis
-
Puck JM, Sneller MC. ALPS: an autoimmune human lymphoproliferative syndrome associated with abnormal lymphocyte apoptosis. Semin Immunol. 1997;9(1):77-84.
-
(1997)
Semin Immunol.
, vol.9
, Issue.1
, pp. 77-84
-
-
Puck, J.M.1
Sneller, M.C.2
-
108
-
-
23844548539
-
Conditional Fas-associated death domain protein (FADD): GFP knockout mice reveal FADD is dispensable in thymic development but essential in peripheral T cell homeostasis
-
Zhang Y et al. Conditional Fas-associated death domain protein (FADD): GFP knockout mice reveal FADD is dispensable in thymic development but essential in peripheral T cell homeostasis. J Immunol. 2005;175(5):3033-44.
-
(2005)
J Immunol.
, vol.175
, Issue.5
, pp. 3033-3044
-
-
Zhang, Y.1
-
109
-
-
18544383460
-
Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency
-
Chun HJ et al. Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency. Nature. 2002;419(6905):395-9.
-
(2002)
Nature.
, vol.419
, Issue.6905
, pp. 395-399
-
-
Chun, H.J.1
-
110
-
-
20044368626
-
Requirement for caspase-8 in NF-kappaB activation by antigen receptor
-
Su H et al. Requirement for caspase-8 in NF-kappaB activation by antigen receptor. Science. 2005;307(5714):1465-8.
-
(2005)
Science.
, vol.307
, Issue.5714
, pp. 1465-1468
-
-
Su, H.1
-
111
-
-
77955609495
-
Fas-associated death domain (FADD) is a negative regulator of T-cell receptor-mediated necroptosis
-
Osborn SL et al. Fas-associated death domain (FADD) is a negative regulator of T-cell receptor-mediated necroptosis. Proc Natl Acad Sci U S A. 2010;107(29):13034-9.
-
(2010)
Proc Natl Acad Sci U S A.
, vol.107
, Issue.29
, pp. 13034-13039
-
-
Osborn, S.L.1
-
112
-
-
80053087284
-
Complementary roles of Fas-associated death domain (FADD) and receptor interacting protein kinase-3 (RIPK3) in T-cell homeostasis and antiviral immunity
-
Lu JV et al. Complementary roles of Fas-associated death domain (FADD) and receptor interacting protein kinase-3 (RIPK3) in T-cell homeostasis and antiviral immunity. Proc Natl Acad Sci U S A. 2011;108(37):15312-7.
-
(2011)
Proc Natl Acad Sci U S A.
, vol.108
, Issue.37
, pp. 15312-15317
-
-
Lu, J.V.1
-
113
-
-
24744459035
-
Cutting edge: Innate immunity conferred by B cells is regulated by caspase-8
-
Beisner DR et al. Cutting edge: innate immunity conferred by B cells is regulated by caspase-8. J Immunol. 2005;175(6):3469-73.
-
(2005)
J Immunol.
, vol.175
, Issue.6
, pp. 3469-3473
-
-
Beisner, D.R.1
-
114
-
-
33646879040
-
The Fas-associated death domain protein is required in apoptosis and TLR induced proliferative responses in B cells
-
Imtiyaz HZ et al. The Fas-associated death domain protein is required in apoptosis and TLR induced proliferative responses in B cells. J Immunol. 2006;176(11):6852-61.
-
(2006)
J Immunol.
, vol.176
, Issue.11
, pp. 6852-6861
-
-
Imtiyaz, H.Z.1
-
115
-
-
3543097542
-
RIP links TLR4 to Akt and is essential for cell survival in response to LPS stimulation
-
Vivarelli MS et al. RIP links TLR4 to Akt and is essential for cell survival in response to LPS stimulation. J Exp Med. 2004;200(3):399-404.
-
(2004)
J Exp Med.
, vol.200
, Issue.3
, pp. 399-404
-
-
Vivarelli, M.S.1
|