Mitochondria in autoinflammation: Cause, mediator or bystander?

Trends in Endocrinology and Metabolism

Volumn 26, Issue 5, 2015, Pages 263-271

  van der Burgh, Robert ^a   Boes, Marianne ^a  

^a UNIVERSITY MEDICAL CENTER UTRECHT   (Netherlands)

Author keywords

Autoinflammation; Inflammasome; Interleukin 1 ; Mitochondria

Indexed keywords

CALCIUM; GUANOSINE TRIPHOSPHATASE; INFLAMMASOME; MITOCHONDRIAL DNA; REACTIVE OXYGEN METABOLITE; DNA;

ACQUIRED IMMUNE DEFICIENCY SYNDROME; AUTOINFLAMMATION; AUTOINFLAMMATORY DISEASE; CELL DAMAGE; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ENDOPLASMIC RETICULUM STRESS; FAMILIAL MEDITERRANEAN FEVER; GENETIC ASSOCIATION; HUMAN; INFLAMMATION; INNATE IMMUNITY; MITOCHONDRIAL DYNAMICS; MITOCHONDRION; PRIORITY JOURNAL; REVIEW; AUTOIMMUNE DISEASE; AUTOPHAGY; GENETICS; HEREDITARY AUTOINFLAMMATORY DISEASES; IMMUNE SYSTEM; IMMUNOLOGY; MACROPHAGE; MITOCHONDRIAL MEMBRANE; PHYSIOLOGY; SIGNAL TRANSDUCTION;

AUTOIMMUNE DISEASES; AUTOPHAGY; DNA; DNA, MITOCHONDRIAL; HEREDITARY AUTOINFLAMMATORY DISEASES; HUMANS; IMMUNE SYSTEM; INFLAMMASOMES; MACROPHAGES; MITOCHONDRIA; MITOCHONDRIAL MEMBRANES; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION;

EID: 84928703375     PISSN: 10432760     EISSN: 18793061     Source Type: Journal    
DOI: 10.1016/j.tem.2015.03.004     Document Type: Review

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