Mitochondrial Damage: A Target for New Therapeutic Horizons

Neurotherapeutics

Volumn 7, Issue 1, 2010, Pages 13-21

  Soustiel, Jean F ^a   Larisch, Sarit ^b  

^a TECHNION ISRAEL INSTITUTE OF TECHNOLOGY   (Israel)

^b UNIVERSITY OF HAIFA   (Israel)

Author keywords

apoptosis; cyclosporin A; mitochondria; mitochondrial permeability transition pore; Traumatic brain injury

Indexed keywords

4' CHLORODIAZEPAM; APOPTOSIS INDUCING FACTOR; BENZYLOXYCARBONYLVALYLALANYLASPARTYL FLUOROMETHYL KETONE; BONGKREKIC ACID; CASPASE INHIBITOR; CYCLOPHILIN D; CYCLOSPORIN A; ESTRADIOL; ESTROGEN; GLUCOSE; INHIBITOR OF APOPTOSIS PROTEIN; INHIBITOR OF APOPTOSIS PROTEIN ANTAGONIST; LACTIC ACID; NEUROPROTECTIVE AGENT; PIFITHRIN ALPHA; POLYPEPTIDE; PROTEIN P53; PYRUVIC ACID; UCF 101; UNCLASSIFIED DRUG; X LINKED INHIBITOR OF APOPTOSIS;

APOPTOSIS; ARTICLE; BRAIN CONTUSION; BRAIN INJURY; BRAIN ISCHEMIA; CEREBROVASCULAR ACCIDENT; CLINICAL TRIAL; DISORDERS OF MITOCHONDRIAL FUNCTIONS; HUMAN; MEMBRANE PERMEABILITY; MITOCHONDRIAL MEMBRANE; MITOCHONDRION SWELLING; NERVE CELL NECROSIS; NONHUMAN; NUCLEOTIDE SEQUENCE; OUTER MEMBRANE; PRIORITY JOURNAL; REGULATORY MECHANISM; REPERFUSION INJURY; STROKE; TRAUMATIC BRAIN INJURY;

EID: 72949097392     PISSN: 19337213     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.nurt.2009.11.001     Document Type: Article

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