Pten inactivation alters peripheral B lymphocyte fate and reconstitutes CD19 function

Nature Immunology

Volumn 4, Issue 3, 2003, Pages 287-294

  Anzelon, Amy N ^a,b   Wu, Hong ^c   Rickert, Robert C ^a,b  

^a UNIVERSITY OF CALIFORNIA SAN DIEGO   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CD19 ANTIGEN; PHOSPHATASE; PHOSPHATIDYLINOSITOL 3 KINASE; PHOSPHATIDYLINOSITOL 3,4,5 TRISPHOSPHATE; PHOSPHATIDYLINOSITOL 3,4,5 TRISPHOSPHATE 3 PHOSPHATASE;

ANIMAL CELL; ANIMAL TISSUE; ANTIGEN FUNCTION; ARTICLE; B LYMPHOCYTE; B LYMPHOCYTE DIFFERENTIATION; CELL FATE; CELL GROWTH; CELL SURVIVAL; CELLULAR DISTRIBUTION; CONTROLLED STUDY; GENE DELETION; GENE INACTIVATION; GERMINAL CENTER; GROWTH REGULATION; IMMUNOSTIMULATION; LYMPHOCYTE SUBPOPULATION; MITOGENESIS; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN LOCALIZATION; REGULATORY MECHANISM; SEQUENCE HOMOLOGY;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANIMALS; ANTIGENS, CD19; B-LYMPHOCYTES; CELL DIFFERENTIATION; MICE; PHOSPHORIC MONOESTER HYDROLASES; PTEN PHOSPHOHYDROLASE; SPLEEN; TUMOR SUPPRESSOR PROTEINS;

EID: 0037342166     PISSN: 15292908     EISSN: None     Source Type: Journal    
DOI: 10.1038/ni892     Document Type: Article

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