-
1
-
-
0032557425
-
Dramatic aggregation of Alzheimer Aβ by Cu(II) is induced by conditions representing physiological acidosis
-
Atwood CS, Moir RD, Huang X, et al. 1998. Dramatic aggregation of Alzheimer Aβ by Cu(II) is induced by conditions representing physiological acidosis. J Biol Chem 273:12817-12826.
-
(1998)
J Biol Chem
, vol.273
, pp. 12817-12826
-
-
Atwood, C.S.1
Moir, R.D.2
Huang, X.3
-
2
-
-
0032514165
-
Promotion of transition metal-induced reactive oxygen species formation by β-amyloid
-
Bondy SC, Guo-Ross SX, Truong AT. 1998. Promotion of transition metal-induced reactive oxygen species formation by β-amyloid. Brain Res 799:91-96.
-
(1998)
Brain Res
, vol.799
, pp. 91-96
-
-
Bondy, S.C.1
Guo-Ross, S.X.2
Truong, A.T.3
-
3
-
-
0031444294
-
The cellular prion protein binds copper in vivo
-
Brown DR, Qin K, Herms JW, et al. 1997. The cellular prion protein binds copper in vivo. Nature 390:684-687.
-
(1997)
Nature
, vol.390
, pp. 684-687
-
-
Brown, D.R.1
Qin, K.2
Herms, J.W.3
-
4
-
-
0032544674
-
Aggregation and motor neuron toxicity of an ALS-linked SOD1 mutant independent from wild-type SOD1
-
Bruijn LI, Houseweart MK, Kato S, et al. 1998. Aggregation and motor neuron toxicity of an ALS-linked SOD1 mutant independent from wild-type SOD1. Science 281:1851-1854.
-
(1998)
Science
, vol.281
, pp. 1851-1854
-
-
Bruijn, L.I.1
Houseweart, M.K.2
Kato, S.3
-
6
-
-
0033006576
-
Is increased redox-active iron in Alzheimer disease a failure of the copper-binding protein ceruloplasmin?
-
Castellani RJ, Smith MA, Nunomura A, et al. 1999. Is increased redox-active iron in Alzheimer disease a failure of the copper-binding protein ceruloplasmin? Free Radic Biol Med 26:1508-1512.
-
(1999)
Free Radic Biol Med
, vol.26
, pp. 1508-1512
-
-
Castellani, R.J.1
Smith, M.A.2
Nunomura, A.3
-
7
-
-
0034033764
-
Evidence for a novel heine-binding protein, HasAh, in Alzheimer disease
-
Castellani RJ, Harris PLR, Lecrosiey A, et al. 2000a. Evidence for a novel heine-binding protein, HasAh, in Alzheimer disease. Antiox Redox Signal 2:137-142.
-
(2000)
Antiox Redox Signal
, vol.2
, pp. 137-142
-
-
Castellani, R.J.1
Harris, P.L.R.2
Lecrosiey, A.3
-
9
-
-
0033551782
-
Aqueous dissolution of Alzheimer's disease Aβ amyloid deposits by biometal depletion
-
Cherny RA, Legg JT, McLean CA, et al. 1999. Aqueous dissolution of Alzheimer's disease Aβ amyloid deposits by biometal depletion. J Biol Chem 274:23223-23228.
-
(1999)
J Biol Chem
, vol.274
, pp. 23223-23228
-
-
Cherny, R.A.1
Legg, J.T.2
McLean, C.A.3
-
10
-
-
0030802648
-
The copper chaperone for superoxide dismutase
-
Cizewski Culotta V, Klomp LWJ, Strain J, Casareno RLB, Krems B, Gitlin JD. 1997. The copper chaperone for superoxide dismutase. J Biol Chem 272:23469-23472.
-
(1997)
J Biol Chem
, vol.272
, pp. 23469-23472
-
-
Cizewski Culotta, V.1
Klomp, L.W.J.2
Strain, J.3
Casareno, R.L.B.4
Krems, B.5
Gitlin, J.D.6
-
11
-
-
0033600849
-
Oxidative stress and iron are implicated in fragmenting vacuoles of Saccharomyces cerevisiae lacking Cu,Zn-superoxide dismutase
-
Corson LN, Folmer J, Strain JJ, et al. 1999. Oxidative stress and iron are implicated in fragmenting vacuoles of Saccharomyces cerevisiae lacking Cu,Zn-superoxide dismutase. J Biol Chem 274:27590-27596.
-
(1999)
J Biol Chem
, vol.274
, pp. 27590-27596
-
-
Corson, L.N.1
Folmer, J.2
Strain, J.J.3
-
12
-
-
0032504035
-
Oxidative stress and overexpression of manganese superoxide dismutase in patients with Alzheimer's disease
-
De Leo ME, Borrello S, Passantino M, et al. 1998. Oxidative stress and overexpression of manganese superoxide dismutase in patients with Alzheimer's disease. Neurosci Lett 250:173-176.
-
(1998)
Neurosci Lett
, vol.250
, pp. 173-176
-
-
De Leo, M.E.1
Borrello, S.2
Passantino, M.3
-
13
-
-
0033515564
-
Amyloid β peptides do not form peptide-derived free radicals spontaneously, but can enhance metal-catalyzed oxidation of hydroxylamines to nitroxides
-
Dikalov SI, Vitek MP, Maples KR, Mason RP. 1999. Amyloid β peptides do not form peptide-derived free radicals spontaneously, but can enhance metal-catalyzed oxidation of hydroxylamines to nitroxides. J Biol Chem 274:9392-9999.
-
(1999)
J Biol Chem
, vol.274
, pp. 9392-9999
-
-
Dikalov, S.I.1
Vitek, M.P.2
Maples, K.R.3
Mason, R.P.4
-
14
-
-
0032776668
-
Hindlimb motor neurons require Cu/Zn superoxide dismutase for maintenance of neuromuscular junctions
-
Flood DG, Reaume AG, Gruner JA, et al. 1999, Hindlimb motor neurons require Cu/Zn superoxide dismutase for maintenance of neuromuscular junctions. Am J Pathol 155:663-672.
-
(1999)
Am J Pathol
, vol.155
, pp. 663-672
-
-
Flood, D.G.1
Reaume, A.G.2
Gruner, J.A.3
-
15
-
-
18344414746
-
The Aβ peptide of Alzheimer's disease directly produces hydrogen peroxide through metal ion reduction
-
Huang X, Atwood CS, Hartshorn MA, et al. 1999a. The Aβ peptide of Alzheimer's disease directly produces hydrogen peroxide through metal ion reduction. Bioehemistry 38:7609-7616.
-
(1999)
Bioehemistry
, vol.38
, pp. 7609-7616
-
-
Huang, X.1
Atwood, C.S.2
Hartshorn, M.A.3
-
16
-
-
0033601338
-
Cu(II) potentiation of Alzheimer Aβ neurotoxicity
-
Huang X, Cuajungco MP, Atwood CS, et al. 1999b. Cu(II) potentiation of Alzheimer Aβ neurotoxicity. J Biol Chem 274:37111-37116.
-
(1999)
J Biol Chem
, vol.274
, pp. 37111-37116
-
-
Huang, X.1
Cuajungco, M.P.2
Atwood, C.S.3
-
17
-
-
0030612033
-
APPSw transgenic mice develop age-related A beta deposits and neuropil abnormalities, but no neuronal loss in CA1
-
Irizarry MC, McNamara M, Fedorchak K, et al. 1997a. APPSw transgenic mice develop age-related A beta deposits and neuropil abnormalities, but no neuronal loss in CA1. J Neuropathol Exp Neurol 56:965-973.
-
(1997)
J Neuropathol Exp Neurol
, vol.56
, pp. 965-973
-
-
Irizarry, M.C.1
McNamara, M.2
Fedorchak, K.3
-
18
-
-
0030611097
-
Aβ deposition is associated with neuropil changes, but not with overt neuronal loss in the human amyloid precursor protein V717F (PDAPP) transgenic mouse
-
Irizarry MC, Soriano F, McNamara M, et al. 1997b. Aβ deposition is associated with neuropil changes, but not with overt neuronal loss in the human amyloid precursor protein V717F (PDAPP) transgenic mouse. J Neurosci 17:7053-7059.
-
(1997)
J Neurosci
, vol.17
, pp. 7053-7059
-
-
Irizarry, M.C.1
Soriano, F.2
McNamara, M.3
-
19
-
-
0027723591
-
Lactotransferin immunocytochemistry in Alzheimer and normal human brain
-
Kawamata T, Tooyama I, Yamada T, et al. 1993. Lactotransferin immunocytochemistry in Alzheimer and normal human brain. Am J Pathol 142:1574-1585.
-
(1993)
Am J Pathol
, vol.142
, pp. 1574-1585
-
-
Kawamata, T.1
Tooyama, I.2
Yamada, T.3
-
21
-
-
0030569306
-
Increased regional brain concentrations of ceruloplasmin in neurodegenerative disorders
-
Loeffler DA, LeWitt PA, Juneau PL, et al. 1996. Increased regional brain concentrations of ceruloplasmin in neurodegenerative disorders. Brain Res 738:265-274.
-
(1996)
Brain Res
, vol.738
, pp. 265-274
-
-
Loeffler, D.A.1
LeWitt, P.A.2
Juneau, P.L.3
-
22
-
-
0032507975
-
Copper, iron and zinc in Alzheimer's disease senile plaques
-
Lovell MA, Robertson JD, Teesdale WJ, et al. 1998. Copper, iron and zinc in Alzheimer's disease senile plaques. J Neurol Sci 158:47-52.
-
(1998)
J Neurol Sci
, vol.158
, pp. 47-52
-
-
Lovell, M.A.1
Robertson, J.D.2
Teesdale, W.J.3
-
23
-
-
0027327488
-
Aluminum, iron, and zinc ions promote aggregation of physiological concentrations of beta-amyloid peptide
-
Mantyh PW, Ghilardi JR, Rogers S, et al. 1993. Aluminum, iron, and zinc ions promote aggregation of physiological concentrations of beta-amyloid peptide. J Neurochem 61:1171-1174.
-
(1993)
J Neurochem
, vol.61
, pp. 1171-1174
-
-
Mantyh, P.W.1
Ghilardi, J.R.2
Rogers, S.3
-
24
-
-
0022480081
-
Increased cerebral glucose-6-phosphate dehydrogenase activity in Alzheimer's disease may reflect oxidative stress
-
Martins RN, Harper CG, Stokes GB, Masters CL. 1986. Increased cerebral glucose-6-phosphate dehydrogenase activity in Alzheimer's disease may reflect oxidative stress. J Neurochem 46:1042-1045.
-
(1986)
J Neurochem
, vol.46
, pp. 1042-1045
-
-
Martins, R.N.1
Harper, C.G.2
Stokes, G.B.3
Masters, C.L.4
-
25
-
-
0031940986
-
A novel neurological phenotype in mice lacking mitochondrial manganese superoxide dismutase
-
Melov S, Schneider JA, Day BJ, et al. 1998. A novel neurological phenotype in mice lacking mitochondrial manganese superoxide dismutase. Nat Genet 18:159-163.
-
(1998)
Nat Genet
, vol.18
, pp. 159-163
-
-
Melov, S.1
Schneider, J.A.2
Day, B.J.3
-
26
-
-
0033528666
-
Differential effects of apolipoprotein E isoforms on metal-induced aggregation of Aβ using physiological concentrations
-
Moir RD, Atwood CS, Romano DM, et al. 1999. Differential effects of apolipoprotein E isoforms on metal-induced aggregation of Aβ using physiological concentrations. Biochemistry 38:4595-4603.
-
(1999)
Biochemistry
, vol.38
, pp. 4595-4603
-
-
Moir, R.D.1
Atwood, C.S.2
Romano, D.M.3
-
27
-
-
0032546577
-
Copper-binding amyloid precursor undergoes a site-specific fragmentation in the reduction of hydrogen peroxide
-
Multhaup G, Ruppert T, Schlicksupp A, et al. 1998, Copper-binding amyloid precursor undergoes a site-specific fragmentation in the reduction of hydrogen peroxide. Biochemistry 37:7224-7230.
-
(1998)
Biochemistry
, vol.37
, pp. 7224-7230
-
-
Multhaup, G.1
Ruppert, T.2
Schlicksupp, A.3
-
28
-
-
0033559462
-
RNA oxidation is a prominent feature of vulnerable neurons in Alzheimer disease
-
Nunomura A, Perry G, Pappolla MA, et al. 1999a. RNA oxidation is a prominent feature of vulnerable neurons in Alzheimer disease. J Neurosci 19:1959-1964.
-
(1999)
J Neurosci
, vol.19
, pp. 1959-1964
-
-
Nunomura, A.1
Perry, G.2
Pappolla, M.A.3
-
29
-
-
0001010343
-
Oxidation of nucleic acids in neurons precedes amyloid β deposition in Down syndrome
-
Nunomura A, Perry G, Smith MA. 1999b. Oxidation of nucleic acids in neurons precedes amyloid β deposition in Down syndrome. J Neuropathol Exp Neurol 58:511.
-
(1999)
J Neuropathol Exp Neurol
, vol.58
, pp. 511
-
-
Nunomura, A.1
Perry, G.2
Smith, M.A.3
-
30
-
-
0000410901
-
Oxidative damage and amyloid β deposition in Alzheimer disease with reference to apolipoprotein E4
-
Numomura A, Perry G, Smith MA, Chiba S. 2000a. Oxidative damage and amyloid β deposition in Alzheimer disease with reference to apolipoprotein E4 (Japanese). Dementia 14:232.
-
(2000)
Dementia
, vol.14
, pp. 232
-
-
Numomura, A.1
Perry, G.2
Smith, M.A.3
Chiba, S.4
-
31
-
-
0000225645
-
Increasing amyloid β 42 deposition is associated with decreasing neuronal RNA oxidation in Down's syndrome and familial Alzheimer's disease
-
Nunomura A, Chiba S, Lippa C, et al. 2000b. Increasing amyloid β 42 deposition is associated with decreasing neuronal RNA oxidation in Down's syndrome and familial Alzheimer's disease. Brain Pathol 10:783.
-
(2000)
Brain Pathol
, vol.10
, pp. 783
-
-
Nunomura, A.1
Chiba, S.2
Lippa, C.3
-
32
-
-
0033758324
-
Neuronal oxidative stress precedes amyloid-β deposition in Down's syndrome
-
Nunomura A, Perry G, Hirai K, et al. 2000c. Neuronal oxidative stress precedes amyloid-β deposition in Down's syndrome, J Neuropathol Exp Neurol 59:1011-1017.
-
(2000)
J Neuropathol Exp Neurol
, vol.59
, pp. 1011-1017
-
-
Nunomura, A.1
Perry, G.2
Hirai, K.3
-
33
-
-
0034923434
-
Oxidative damage is the earliest event in Alzheimer disease
-
Nunomura A, Perry G, Aliev G, et al. 2001. Oxidative damage is the earliest event in Alzheimer disease. J Neuropathol Exp Neurol 60:759-767.
-
(2001)
J Neuropathol Exp Neurol
, vol.60
, pp. 759-767
-
-
Nunomura, A.1
Perry, G.2
Aliev, G.3
-
34
-
-
0034098189
-
Lipoperoxidation is selectively involved in progressive supranuclear palsy
-
Odetti P, Garibaldi S, Norese R, et al. 2000. Lipoperoxidation is selectively involved in progressive supranuclear palsy. J Neuropathol Exp Neurol 59:393-397.
-
(2000)
J Neuropathol Exp Neurol
, vol.59
, pp. 393-397
-
-
Odetti, P.1
Garibaldi, S.2
Norese, R.3
-
35
-
-
0343336439
-
Ferritin is associated with the aberrant tau filaments present in progressive supranuclear palsy
-
Perez M, Valpuesta JM, de Gareini EM, et al. 1998. Ferritin is associated with the aberrant tau filaments present in progressive supranuclear palsy. Am J Pathol 152:1531-1539.
-
(1998)
Am J Pathol
, vol.152
, pp. 1531-1539
-
-
Perez, M.1
Valpuesta, J.M.2
De Gareini, E.M.3
-
37
-
-
0034026561
-
How important is oxidative damage? Lessons from Alzheimer disease
-
Perry G, Raina AK, Nunomura A, et al. 2000a. How important is oxidative damage? Lessons from Alzheimer disease. Free Radic Biol Med 28:831-834.
-
(2000)
Free Radic Biol Med
, vol.28
, pp. 831-834
-
-
Perry, G.1
Raina, A.K.2
Nunomura, A.3
-
38
-
-
0034716488
-
Amyloid-β junkies
-
Perry G, Nunomura A, Raina AK, Smith MA. 2000b, Amyloid-β junkies. Lancet 355:757.
-
(2000)
Lancet
, vol.355
, pp. 757
-
-
Perry, G.1
Nunomura, A.2
Raina, A.K.3
Smith, M.A.4
-
39
-
-
0010487727
-
Expression of iron transport proteins and excessive iron accumulation in the brain in neurodegenerative disorders
-
Qian ZM, Wang Q. 1998. Expression of iron transport proteins and excessive iron accumulation in the brain in neurodegenerative disorders. Brain Res Rev 27:257-267.
-
(1998)
Brain Res Rev
, vol.27
, pp. 257-267
-
-
Qian, Z.M.1
Wang, Q.2
-
40
-
-
0033036818
-
Quinone reductase (NQO1), a sensitive redox indicator, is increased in Alzheimer's disease
-
Raina AK, Templeton DJ, Deak JC, et al. 1999. Quinone reductase (NQO1), a sensitive redox indicator, is increased in Alzheimer's disease. Redox Report 4:23-27.
-
(1999)
Redox Report
, vol.4
, pp. 23-27
-
-
Raina, A.K.1
Templeton, D.J.2
Deak, J.C.3
-
42
-
-
0033569839
-
Increased neuronal glucose-6-phosphate dehydrogenase and sulfhydryl levels indicate reductive compensation to oxidative stress in Alzheimer disease
-
Russell RL, Siedlak SL, Raina AK, et al. 1999. Increased neuronal glucose-6-phosphate dehydrogenase and sulfhydryl levels indicate reductive compensation to oxidative stress in Alzheimer disease. Arch Biochem Biophys 370:236-239.
-
(1999)
Arch Biochem Biophys
, vol.370
, pp. 236-239
-
-
Russell, R.L.1
Siedlak, S.L.2
Raina, A.K.3
-
43
-
-
0032529412
-
Myeloperoxidase and horseradish peroxidase catalyze tyrosine nitration in proteins from nitrite and hydrogen peroxide
-
Sampson JB, Ye Y, Rosen H, Beckman JS. 1998. Myeloperoxidase and horseradish peroxidase catalyze tyrosine nitration in proteins from nitrite and hydrogen peroxide. Arch Biochem Biophys 356:207-213.
-
(1998)
Arch Biochem Biophys
, vol.356
, pp. 207-213
-
-
Sampson, J.B.1
Ye, Y.2
Rosen, H.3
Beckman, J.S.4
-
44
-
-
0030989545
-
4-Hydroxnonenal-derived advanced lipid peroxidation end products are increased in Alzheimer's disease
-
Sayre LM, Zelasko DA, Harris PLR, et al. 1997a. 4-Hydroxnonenal-derived advanced lipid peroxidation end products are increased in Alzheimer's disease. J Neurochem 68:2092-2097.
-
(1997)
J Neurochem
, vol.68
, pp. 2092-2097
-
-
Sayre, L.M.1
Zelasko, D.A.2
Harris, P.L.R.3
-
45
-
-
0030983405
-
Mechanisms of neurotoxicity associated with amyloid β deposition and the role of free radicals in the pathogenesis of Alzheimer's disease: A critical appraisal
-
Sayre LM, Zagorski MG, Surewicz WK, et al. 1997b. Mechanisms of neurotoxicity associated with amyloid β deposition and the role of free radicals in the pathogenesis of Alzheimer's disease: a critical appraisal. Chem Res Toxicol 10:518-526.
-
(1997)
Chem Res Toxicol
, vol.10
, pp. 518-526
-
-
Sayre, L.M.1
Zagorski, M.G.2
Surewicz, W.K.3
-
46
-
-
0033964859
-
In situ redox catalysis by neurofibrillary tangles and senile plaques in Alzheimer disease: A central role for bound transition metals
-
Sayre LM, Perry G, Harris PLR, et al. 2000. In situ redox catalysis by neurofibrillary tangles and senile plaques in Alzheimer disease: a central role for bound transition metals. J Neurochem 74:270-279.
-
(2000)
J Neurochem
, vol.74
, pp. 270-279
-
-
Sayre, L.M.1
Perry, G.2
Harris, P.L.R.3
-
47
-
-
0028233977
-
Advanced Maillard reaction end products are associated with Alzheimer disease pathology
-
Smith MA, Taneda S, Richey PL, et al. 1994. Advanced Maillard reaction end products are associated with Alzheimer disease pathology. Proc Natl Acad Sci USA 91:5710-5714.
-
(1994)
Proc Natl Acad Sci USA
, vol.91
, pp. 5710-5714
-
-
Smith, M.A.1
Taneda, S.2
Richey, P.L.3
-
48
-
-
0346386580
-
Widespread peroxynitrite-mediated damage in Alzheimer's disease
-
Smith MA, Harris PLR, Sayre LM, et al. 1997a. Widespread peroxynitrite-mediated damage in Alzheimer's disease. J Neurosci 17:2653-2657.
-
(1997)
J Neurosci
, vol.17
, pp. 2653-2657
-
-
Smith, M.A.1
Harris, P.L.R.2
Sayre, L.M.3
-
49
-
-
0030885482
-
Iron accumulation in Alzheimer disease is a source of redox-generated free radicals
-
Smith MA, Harris PLR, Sayre LM, Perry G. 1997b. Iron accumulation in Alzheimer disease is a source of redox-generated free radicals. Proc Nad Acad Sci USA 94:9866-9868.
-
(1997)
Proc Nad Acad Sci USA
, vol.94
, pp. 9866-9868
-
-
Smith, M.A.1
Harris, P.L.R.2
Sayre, L.M.3
Perry, G.4
-
50
-
-
0032579795
-
Abnormal localization of iron regulatory protein (IRP) in Alzheimer disease
-
Smith MA, Wehr K, Harris PLR, et al. 1998a. Abnormal localization of iron regulatory protein (IRP) in Alzheimer disease. Brain Res 788:232-236.
-
(1998)
Brain Res
, vol.788
, pp. 232-236
-
-
Smith, M.A.1
Wehr, K.2
Harris, P.L.R.3
-
51
-
-
0031776586
-
Cytochemical demonstration of oxidative damage in Alzheimer disease by imnmnochemical enhancement of the carbonyl reaction with dinitrophenylhydrazine
-
Smith MA, Sayre LM, Anderson VE, et al. 1998b. Cytochemical demonstration of oxidative damage in Alzheimer disease by imnmnochemical enhancement of the carbonyl reaction with dinitrophenylhydrazine. J Histochem Cytochem 46:731-735.
-
(1998)
J Histochem Cytochem
, vol.46
, pp. 731-735
-
-
Smith, M.A.1
Sayre, L.M.2
Anderson, V.E.3
-
52
-
-
0031981072
-
Amyloid-β deposition in Alzheimer transgenic mice is associated with oxidative stress
-
Smith MA, Hirai K, Hsiao K, et al. 1998c. Amyloid-β deposition in Alzheimer transgenic mice is associated with oxidative stress. J Neurochem 70:2212-2215.
-
(1998)
J Neurochem
, vol.70
, pp. 2212-2215
-
-
Smith, M.A.1
Hirai, K.2
Hsiao, K.3
-
53
-
-
0033836580
-
In Alzheimer disease, heme oxygenase is coincident with Alz50, an epitope of tau induced by 4-hydroxy-2-nonenal modification
-
Takeda A, Smith MA, Avila J, et al. 2000. In Alzheimer disease, heme oxygenase is coincident with Alz50, an epitope of tau induced by 4-hydroxy-2-nonenal modification. J Neurochem 75:1234-1241.
-
(2000)
J Neurochem
, vol.75
, pp. 1234-1241
-
-
Takeda, A.1
Smith, M.A.2
Avila, J.3
-
54
-
-
0032880901
-
Methionine residue 35 is important in amyloid β-peptide-associated free radical oxidative stress
-
Varadarajan S, Yatin S, Kanski J, et al. 1999, Methionine residue 35 is important in amyloid β-peptide-associated free radical oxidative stress. Brain Res Bull 50:133-141.
-
(1999)
Brain Res Bull
, vol.50
, pp. 133-141
-
-
Varadarajan, S.1
Yatin, S.2
Kanski, J.3
-
55
-
-
0032516036
-
Overexpression of human copper, zinc-superoxide dismutase (SOD1) prevents postischemic injury
-
Wang P, Chen H, Qin H, et al. 1998. Overexpression of human copper, zinc-superoxide dismutase (SOD1) prevents postischemic injury. Proe Natl Acad Sci USA 95:4556-4560.
-
(1998)
Proe Natl Acad Sci USA
, vol.95
, pp. 4556-4560
-
-
Wang, P.1
Chen, H.2
Qin, H.3
-
56
-
-
0033010755
-
Exacerbation of copper toxicity in primary neuronal cultures depleted of cellular glutathione
-
White AR, Bush AI, Beyreuther K, et al. 1999a. Exacerbation of copper toxicity in primary neuronal cultures depleted of cellular glutathione. J Neurochem 72:2092-2098.
-
(1999)
J Neurochem
, vol.72
, pp. 2092-2098
-
-
White, A.R.1
Bush, A.I.2
Beyreuther, K.3
-
57
-
-
0032830357
-
Copper levels are increased in the cerebral cortex and liver of APP and APLP2 knockout mice
-
White AR, Reyes R, Mercer JFB, et al. 1999b. Copper levels are increased in the cerebral cortex and liver of APP and APLP2 knockout mice. Brain Res 842:439-444.
-
(1999)
Brain Res
, vol.842
, pp. 439-444
-
-
White, A.R.1
Reyes, R.2
Mercer, J.F.B.3
-
58
-
-
0032561328
-
Increased oxidative damage is correlated to altered mitochondrial function in heterozygous manganese superoxide dismutase knockout mice
-
Williams MD, Van Remmen H, Conrad CC, et al. 1998. Increased oxidative damage is correlated to altered mitochondrial function in heterozygous manganese superoxide dismutase knockout mice. J Biol Chem 273:28510-28515.
-
(1998)
J Biol Chem
, vol.273
, pp. 28510-28515
-
-
Williams, M.D.1
Van Remmen, H.2
Conrad, C.C.3
-
59
-
-
0035145614
-
Increased levels of oxidative stress markers detected in the brains of mice devoid of prion protein
-
Wong B-S, Liu T, Li R, et al. 2001a. Increased levels of oxidative stress markers detected in the brains of mice devoid of prion protein. J Neurochem 76:565-572.
-
(2001)
J Neurochem
, vol.76
, pp. 565-572
-
-
Wong, B.-S.1
Liu, T.2
Li, R.3
-
60
-
-
0035025086
-
Induction of heme oxygenase-1 and neuronal nitric oxide synthase in Doppelexpressing mice devoid of prion protein: Implications for Doppel function
-
Wong B-S, Liu T, Paisley D, et al. 2001b. Induction of heme oxygenase-1 and neuronal nitric oxide synthase in Doppelexpressing mice devoid of prion protein: implications for Doppel function. Mol Cell Neurosci 17:768-775.
-
(2001)
Mol Cell Neurosci
, vol.17
, pp. 768-775
-
-
Wong, B.-S.1
Liu, T.2
Paisley, D.3
-
61
-
-
0033612959
-
The stabilization of ferrous iron by a toxic β-amyloid fragment and by an aluminum salt
-
Yang EY, Guo-Ross SX, Bondy SC. 1999. The stabilization of ferrous iron by a toxic β-amyloid fragment and by an aluminum salt. Brain Res 839:221-226.
-
(1999)
Brain Res
, vol.839
, pp. 221-226
-
-
Yang, E.Y.1
Guo-Ross, S.X.2
Bondy, S.C.3
-
62
-
-
0033133579
-
In vitro and in vivo oxidative stress associated with Alzheimer's amyloid β-peptide(1-42)
-
Yatin SM, Varadarajan S, Link CD, Butterfield DA. 1999. In vitro and in vivo oxidative stress associated with Alzheimer's amyloid β-peptide(1-42). Neurobiol Aging 20:325-330.
-
(1999)
Neurobiol Aging
, vol.20
, pp. 325-330
-
-
Yatin, S.M.1
Varadarajan, S.2
Link, C.D.3
Butterfield, D.A.4
|