Exacerbation of copper toxicity in primary neuronal cultures depleted of cellular glutathione

Journal of Neurochemistry

Volumn 72, Issue 5, 1999, Pages 2092-2098

  White, Anthony R ^c   Bush, Ashley I ^a   Beyreuther, Konrad ^b   Masters, Colin L ^d   Cappai, Roberto ^d  

^a HARVARD MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF HEIDELBERG   (Germany)

^c UNIVERSITY OF MELBOURNE   (Australia)

^d NONE

Author keywords

Alzheimer's disease; Amyloid Neurodegeneration; Buthionine sulfoximine; Neurons; Toxicity

Indexed keywords

AMYLOID BETA PROTEIN; CARMUSTINE; COPPER; GLUTATHIONE; GLUTATHIONE REDUCTASE; IRON;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; COPPER METABOLISM; EMBRYO; ENZYME INHIBITION; GLUTATHIONE METABOLISM; MOLECULAR INTERACTION; MOUSE; NERVE CELL CULTURE; NERVE DEGENERATION; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL;

AMYLOID BETA-PROTEIN; ANIMALS; BUTHIONINE SULFOXIMINE; CELLS, CULTURED; COPPER; DRUG COMBINATIONS; DRUG SYNERGISM; FREE RADICAL SCAVENGERS; GLUTATHIONE; GLUTATHIONE REDUCTASE; METALLOPORPHYRINS; MICE; MICE, INBRED C57BL; NEURONS; PEPTIDE FRAGMENTS;

ANIMALIA; MURINAE;

EID: 0033010755     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.1999.0722092.x     Document Type: Article

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