Genetic and Dietary Iron Overload Differentially Affect the Course of Salmonella Typhimurium Infection

Frontiers in Cellular and Infection Microbiology

Volumn 7, Issue APR, 2017, Pages

  Nairz, Manfred ^a   Schroll, Andrea ^a   Haschka, David ^a   Dichtl, Stefanie ^a   Tymoszuk, Piotr ^a   Demetz, Egon ^a   Moser, Patrizia ^b   Haas, Hubertus ^c   Fang, Ferric C ^d   Theurl, Igor ^a   Weiss, Günter ^a  

^a INNSBRUCK MEDICAL UNIVERSITY   (Austria)

^b MEDICAL UNIVERSITY OF INNSBRUCK   (Austria)

^c INNSBRUCK MEDICAL UNIVERSITY   (Austria)

^d UNIVERSITY OF WASHINGTON   (United States)

Author keywords

Hepcidin; Infection; Iron; Lipocalin; Macrophage; SALMONELLA; Siderophore

Indexed keywords

FERRITIN; IRON; HEMOCHROMATOSIS PROTEIN; HFE PROTEIN, MOUSE; IRON INTAKE;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; BACTERIAL GENE; BLOOD CELL COUNT; CIRA GENE; DIETARY INTAKE; DISEASE COURSE; ENZYME LINKED IMMUNOSORBENT ASSAY; FEOB GENE; FEPA GENE; GENE EXPRESSION; HEMOCHROMATOSIS; HEREDITY; HISTOLOGY; HOST PATHOGEN INTERACTION; INFECTION CONTROL; INNATE IMMUNITY; IROC GENE; IRON BLOOD LEVEL; IRON METABOLISM; IRON OVERLOAD; MOUSE; NONHUMAN; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SALMONELLA ENTERICA SEROVAR TYPHIMURIUM; SALMONELLOSIS; SITB GENE; SPLEEN; SURVIVAL RATE; TISSUE INJURY; ADMINISTRATION AND DOSAGE; ANIMAL; COMPLICATION; DEFICIENCY; DISEASE MODEL; IRON INTAKE; KNOCKOUT MOUSE; MICROBIOLOGY; PATHOGENICITY; PATHOLOGY;

ANIMALS; DISEASE MODELS, ANIMAL; HEMOCHROMATOSIS PROTEIN; HOST-PATHOGEN INTERACTIONS; IRON OVERLOAD; IRON, DIETARY; MICE; MICE, KNOCKOUT; SALMONELLA INFECTIONS; SALMONELLA TYPHIMURIUM;

EID: 85027470040     PISSN: None     EISSN: 22352988     Source Type: Journal    
DOI: 10.3389/fcimb.2017.00110     Document Type: Article

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