Redox imbalance and mitochondrial abnormalities in the diabetic lung

Redox Biology

Volumn 11, Issue , 2017, Pages 51-59

  Wu, Jinzi ^a   Jin, Zhen ^a   Yan, Liang Jun ^a  

^a UNIVERSITY OF NORTH TEXAS HEALTH SCIENCE CENTER   (United States)

Author keywords

Diabetes; Lung injury; Mitochondrial abnormalities; Oxidative stress; Redox imbalance

Indexed keywords

ADENOSINE TRIPHOSPHATE; ALDEHYDE REDUCTASE; CYTOCHROME C OXIDASE; DIHYDROLIPOAMIDE DEHYDROGENASE; GLUCOSE 6 PHOSPHATE DEHYDROGENASE; GLUTATHIONE; HYDROGEN PEROXIDE; NICOTINAMIDE ADENINE DINUCLEOTIDE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE 1; NICOTINAMIDE METHYLTRANSFERASE; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE (PHOSPHATE) DEHYDROGENASE (QUINONE); REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE; SIRTUIN 3; SUCCINATE DEHYDROGENASE (UBIQUINONE); UBIQUINOL CYTOCHROME C REDUCTASE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE; NQO1 PROTEIN, RAT; SIRT-3 PROTEIN, RAT; SIRTUIN;

ADULT; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL DEATH; CONTROLLED STUDY; DIABETES MELLITUS; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ENZYME ACTIVITY; LUNG INJURY; MALE; MITOCHONDRIAL MEMBRANE POTENTIAL; NONHUMAN; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN CONTENT; PROTEIN EXPRESSION; RAT; STREPTOZOTOCIN-INDUCED DIABETES MELLITUS; UPREGULATION; YOUNG ADULT; ANIMAL; GENETICS; HUMAN; LUNG; METABOLISM; MITOCHONDRION; OXIDATION REDUCTION REACTION; PATHOLOGY;

ANIMALS; DIABETES MELLITUS; DIHYDROLIPOAMIDE DEHYDROGENASE; ELECTRON TRANSPORT COMPLEX I; GLUCOSEPHOSPHATE DEHYDROGENASE; GLUTATHIONE; HUMANS; LUNG; MITOCHONDRIA; NAD; NAD(P)H DEHYDROGENASE (QUINONE); NADP; OXIDATION-REDUCTION; OXIDATIVE STRESS; POLY(ADP-RIBOSE) POLYMERASES; RATS; SIRTUINS;

EID: 84997109064     PISSN: 22132317     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.redox.2016.11.003     Document Type: Article

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