Roles of Pyruvate, NADH, and Mitochondrial Complex i in Redox Balance and Imbalance in β Cell Function and Dysfunction

Journal of Diabetes Research

Volumn 2015, Issue , 2015, Pages

  Luo, Xiaoting ^a,b   Li, Rongrong ^a   Yan, Liang Jun ^a  

^a UNIVERSITY OF NORTH TEXAS HEALTH SCIENCE CENTER   (United States)

^b GANNAN MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

GLUCOSE; INSULIN; PYRUVIC ACID; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); GLUCOSE BLOOD LEVEL; NICOTINAMIDE ADENINE DINUCLEOTIDE;

DIABETES MELLITUS; GLUCOSE METABOLISM; HYPERGLYCEMIA; INSULIN RELEASE; METABOLIC DISORDER; OXIDATION; PANCREAS ISLET BETA CELL; PATHOGENESIS; PRIORITY JOURNAL; REVIEW; ANIMAL; GLUCOSE BLOOD LEVEL; HUMAN; METABOLISM; MITOCHONDRION; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; SECRETION (PROCESS);

ANIMALS; BLOOD GLUCOSE; DIABETES MELLITUS; ELECTRON TRANSPORT COMPLEX I; GLUCOSE; HUMANS; INSULIN; INSULIN-SECRETING CELLS; MITOCHONDRIA; NAD; OXIDATION-REDUCTION; OXIDATIVE STRESS; PYRUVIC ACID; REACTIVE OXYGEN SPECIES;

EID: 84936963852     PISSN: 23146745     EISSN: 23146753     Source Type: Journal    
DOI: 10.1155/2015/512618     Document Type: Review

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