Potential biochemical mechanisms of lung injury in diabetes

Aging and Disease

Volumn 8, Issue 1, 2017, Pages 7-16

  Zheng, Hong ^a,b   Wu, Jinzi ^a   Jin, Zhen ^a   Yan, Liang Jun ^a  

^a UNIVERSITY OF NORTH TEXAS HEALTH SCIENCE CENTER   (United States)

^b SHANDONG UNIVERSITY OF TRADITIONAL CHINESE MEDICINE   (China)

Author keywords

Diabetes mellitus; Diabetic hyperglycemia; Diabetic lung injury; Mitochondria; Oxidative stress

Indexed keywords

GLUTATHIONE PEROXIDASE; GLUTATHIONE REDUCTASE; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; MALONALDEHYDE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; POLYOL; PROTEIN KINASE C; REACTIVE OXYGEN METABOLITE; SUPEROXIDE DISMUTASE;

AIRWAY RESISTANCE; AUTONOMIC NEUROPATHY; BRONCHUS REACTIVITY; DIABETES MELLITUS; DIABETIC MICROANGIOPATHY; EXPIRATORY FLOW; FORCED EXPIRATORY VOLUME; HEART OUTPUT; HUMAN; HYPERGLYCEMIA; LIVER CELL DAMAGE; LUNG GAS EXCHANGE; LUNG INJURY; LUNG VENTILATION; OXIDATIVE STRESS; PNEUMONIA; PROTEIN GLYCOSYLATION; RESIDUAL VOLUME; REVIEW; TISSUE INJURY; TOTAL LUNG CAPACITY;

EID: 85014253481     PISSN: None     EISSN: 21525250     Source Type: Journal    
DOI: 10.14336/AD.2016.0627     Document Type: Review

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