Detrimental or beneficial: The role of TRPM2 in ischemia/reperfusion injury

Acta Pharmacologica Sinica

Volumn 37, Issue 1, 2016, Pages 4-12

  Zhan, Kai Yu ^a   Yu, Pei Lin ^b   Liu, Chun Hui ^a   Luo, Jian Hong ^a   Yang, Wei ^a  

^a ZHEJIANG UNIVERSITY SCHOOL OF MEDICINE   (China)

^b ZHEJIANG UNIVERSITY   (China)

Author keywords

Ca2+ overload; cardiac ischemia; cerebral ischemia; ischemia reperfusion injury; renal ischemia; ROS; TRPM2

Indexed keywords

2 AMINOETHOXYDIPHENYLBORANE; CLOTRIMAZOLE; ECONAZOLE; FLUFENAMIC ACID; TRANSIENT RECEPTOR POTENTIAL CHANNEL M2; TRANSIENT RECEPTOR POTENTIAL CHANNEL M;

BRAIN ISCHEMIA; CLINICAL TRIAL (TOPIC); HEART MUSCLE ISCHEMIA; KIDNEY ISCHEMIA; NONHUMAN; REPERFUSION INJURY; REVIEW; ANIMAL; HUMAN; KIDNEY; METABOLISM; MYOCARDIAL ISCHEMIA REPERFUSION INJURY; PHYSIOLOGY; VASCULARIZATION;

ANIMALS; BRAIN ISCHEMIA; HUMANS; KIDNEY; MYOCARDIAL REPERFUSION INJURY; REPERFUSION INJURY; TRPM CATION CHANNELS;

EID: 84953222286     PISSN: 16714083     EISSN: 17457254     Source Type: Journal    
DOI: 10.1038/aps.2015.141     Document Type: Review

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