Divergent functions of the Rho GTPases Rac1 and Cdc42 in podocyte injury

Kidney International

Volumn 84, Issue 5, 2013, Pages 920-930

  Blattner, Simone M ^a   Hodgin, Jeffrey B ^a   Nishio, Masashi ^a   Wylie, Stephanie A ^a   Saha, Jharna ^a   Soofi, Abdul A ^a   Vining, Courtenay ^a   Randolph, Ann ^a   Herbach, Nadja ^b   Wanke, Ruediger ^b   Atkins, Kevin B ^a   Gyung Kang, Hee ^c   Henger, Anna ^a   Brakebusch, Cord ^d   Holzman, Lawrence B ^e   Kretzler, Matthias ^a  

^a UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF MUNICH   (Germany)

^c Seoul National University   (South Korea)

^d UNIVERSITY OF COPENHAGEN   (Denmark)

^e UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

cytoskeleton; glomerular disease; glomerular filtration barrier; podocyte; transgenic mouse

Indexed keywords

COFILIN; NEPHRIN; PODOCIN; PROTEIN CDC42; RAC1 PROTEIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL DAMAGE; CELL DISRUPTION; CELL FUNCTION; CELL STRUCTURE; CONTROLLED STUDY; DISEASE EXACERBATION; DISEASE SEVERITY; GLOMERULOSCLEROSIS; MALE; MOUSE; NONHUMAN; PODOCYTE; PRIORITY JOURNAL; PROTEIN DEPHOSPHORYLATION; PROTEINURIA; STEADY STATE;

ACTIN DEPOLYMERIZING FACTORS; ACUTE KIDNEY INJURY; ALBUMINURIA; ANIMALS; CDC42 GTP-BINDING PROTEIN; CELL SHAPE; DESOXYCORTICOSTERONE ACETATE; DISEASE MODELS, ANIMAL; GENOTYPE; HYPERTENSION; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MALE; MEMBRANE PROTEINS; MICE; MICE, 129 STRAIN; MICE, INBRED C57BL; MICE, KNOCKOUT; NEPHRECTOMY; NEUROPEPTIDES; PHENOTYPE; PHOSPHORYLATION; PODOCYTES; PROTAMINES; RAC1 GTP-BINDING PROTEIN; RENAL INSUFFICIENCY; SIGNAL TRANSDUCTION; TIME FACTORS;

EID: 84888382854     PISSN: 00852538     EISSN: 15231755     Source Type: Journal    
DOI: 10.1038/ki.2013.175     Document Type: Article

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