Chromatin Modifications Associated with DNA Double-strand Breaks Repair as Potential Targets for Neurological Diseases

Neurotherapeutics

Volumn 10, Issue 4, 2013, Pages 817-830

  Brochier, Camille ^a,b   Langley, Brett ^a,b  

^a BURKE MEDICAL RESEARCH INSTITUTE   (United States)

^b WEILL CORNELL MEDICAL COLLEGE   (United States)

Author keywords

Chromatin modification; DNA damage; DNA repair; Epigenetics; Neurodegeneration

Indexed keywords

ADENOSINE DIPHOSPHATE RIBOSE; APOPTOSIS INDUCING FACTOR; ATM PROTEIN; BRCA1 PROTEIN; BRCA2 PROTEIN; HISTONE DEACETYLASE; HISTONE DEACETYLASE 1; HISTONE DEACETYLASE 2; HISTONE DEACETYLASE 3; HISTONE DEACETYLASE INHIBITOR; HISTONE H2AX; HISTONE H3; HISTONE H4; HISTONE METHYLTRANSFERASE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE 1; RAD51 PROTEIN; RAD54 PROTEIN; TRANSCRIPTION FACTOR E2F1; XRCC3 PROTEIN; XRCC4 PROTEIN;

ADENOSINE DIPHOSPHATE RIBOSYLATION; ALZHEIMER DISEASE; AMYOTROPHIC LATERAL SCLEROSIS; APOPTOSIS; CELL CYCLE PROGRESSION; CELL VIABILITY; CELLULAR DISTRIBUTION; CEREBROVASCULAR ACCIDENT; CHROMATIN; CHROMATIN ASSEMBLY AND DISASSEMBLY; DNA DAMAGE; DNA END JOINING REPAIR; DNA REPAIR; DOUBLE STRANDED DNA BREAK; EMBRYO DEVELOPMENT; ENVIRONMENTAL STRESS; EPIGENETICS; FIBROBLAST; HISTONE MODIFICATION; HISTONE PHOSPHORYLATION; HOMOLOGOUS RECOMBINATION; HUMAN; HUNTINGTON CHOREA; LEARNING; MEMORY; NERVE DEGENERATION; NEUROLOGIC DISEASE; NEUROPROTECTION; NONHUMAN; OXIDATIVE STRESS; PARKINSON DISEASE; PRIORITY JOURNAL; PROTEIN LOCALIZATION; PROTEIN PROCESSING; REVIEW; SPINAL CORD INJURY; X LINKED MENTAL RETARDATION; XERODERMA PIGMENTOSUM;

CHROMATIN ASSEMBLY AND DISASSEMBLY; DNA BREAKS, DOUBLE-STRANDED; DNA REPAIR; HUMANS; NEURODEGENERATIVE DISEASES;

EID: 84886233092     PISSN: 19337213     EISSN: 18787479     Source Type: Journal    
DOI: 10.1007/s13311-013-0210-9     Document Type: Review

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