What renders TAU toxic

Frontiers in Neurology

Volumn 4 JUN, Issue , 2013, Pages

  Götz, Jürgen ^a,b   Xia, Di ^a   Leinenga, Gerhard ^a   Chew, Yee Lian ^c   Nicholas, Hannah R ^c  

^a UNIVERSITY OF QUEENSLAND   (Australia)

^b UNIVERSITY OF SYDNEY   (Australia)

^c UNIVERSITY OF SYDNEY   (Australia)

Author keywords

Alzheimer's disease; C. elegans; Frontotemporal dementia; Knock out; PP2A; PTL 1; TAU; Transgenic

Indexed keywords

AMYLOID BETA PROTEIN; MICROTUBULE ASSOCIATED PROTEIN 2; MICROTUBULE PROTEIN; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; PROTEIN PTL 1; TAU PROTEIN; TYROSINE 3 MONOOXYGENASE; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; BIOACCUMULATION; CELLULAR DISTRIBUTION; COGNITIVE DEFECT; GENE; GENE INACTIVATION; GENE OVEREXPRESSION; HUMAN; NERVE CELL LESION; NEUROPATHOLOGY; NEUROTOXICITY; NONHUMAN; PROTEIN AGGREGATION; PROTEIN LOCALIZATION; PROTEIN PROTEIN INTERACTION; PTL 1 GENE; REVIEW; TAU GENE;

EID: 84883536024     PISSN: None     EISSN: 16642295     Source Type: Journal    
DOI: 10.3389/fneur.2013.00072     Document Type: Review

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