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Volumn 93, Issue 1, 2011, Pages 23-58

The environment and susceptibility to schizophrenia

Author keywords

Animal models; Attributable proportion; Birth cohort; Cannabis; CNV; Cohort; Copy number variants; Cytokines; Determinants; Developmental trajectories; Dopamine; Ecologic; Environmental; Epidemiology; Epigenetics; Exposures; Famine; Fetal; Fetus; Folate; Folic acid; GABA; Gene environment interaction; Genetics; Glutamate; Herpes simples; Homocysteine; HSV2; Hypoxia; Incidence; Infection; Influenza; Iron; Lead; LPS; Malnutrition; Maternal; Maternal fetal; Maternal immune activation; Microarray; Micronutrient; Migrant; Neurodevelopment; NMDA; Nutrition; Obstetric complications; Pathogenic models; Poly I:C; Pregnancy; Prenatal; Prevalence; Prevention; Protein deprivation; Psychosocial; Rh incompatibility; Rhesus incompatibility; Risk factors; Rubella; Schizophrenia; Season of birth; Seasonality; Socioeconomic status; Stress; T. gondii; Toxoplasma gondii; Translational; Trauma; Urban; Urban rural; Urbanicity; Vitamin D

Indexed keywords

CANNABIS; LEAD;

EID: 78249256348     PISSN: 03010082     EISSN: 18735118     Source Type: Journal    
DOI: 10.1016/j.pneurobio.2010.09.003     Document Type: Review
Times cited : (526)

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