KATP channelopathies in the pancreas

Pflugers Archiv European Journal of Physiology

Volumn 460, Issue 2, 2010, Pages 307-320

  Remedi, Maria S ^a   Koster, Joseph C ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Diazoxide; Hyperinsulinism; Kir6.2; Neonatal diabetes; Sulfonylurea; SUR1

Indexed keywords

ADENOSINE TRIPHOSPHATE SENSITIVE POTASSIUM CHANNEL; CALCIUM CHANNEL L TYPE; CHLOROTHIAZIDE; DIAZOXIDE; GLIBENCLAMIDE; GLUCAGON LIKE PEPTIDE 1; GLUCOSE; INCRETIN; INSULIN; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR6.1; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR6.2; POTASSIUM ION; SULFONYLUREA DERIVATIVE; SULFONYLUREA RECEPTOR;

CHANNEL GATING; DISEASE MODEL; GENE MUTATION; GENETIC VARIABILITY; GLYCEMIC CONTROL; HUMAN; HYPERINSULINEMIA; INSULIN RELEASE; JUVENILE DIABETES MELLITUS; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; PANCREAS ISLET BETA CELL; PANCREAS ISLET TRANSPLANTATION; PANCREAS RESECTION; PATHOGENESIS; PERSISTENT HYPERINSULINEMIC HYPOGLYCEMIA OF INFANCY; POTASSIUM CHANNELOPATHY; POTASSIUM CONDUCTANCE; PRIORITY JOURNAL; REVIEW; RISK FACTOR;

ANIMALS; ATP-BINDING CASSETTE TRANSPORTERS; CHANNELOPATHIES; DIABETES MELLITUS, TYPE 2; HUMANS; INSULIN; INSULIN-SECRETING CELLS; PERSISTENT HYPERINSULINEMIA HYPOGLYCEMIA OF INFANCY; POTASSIUM CHANNELS, INWARDLY RECTIFYING; RECEPTORS, DRUG;

EID: 77955584690     PISSN: 00316768     EISSN: 14322013     Source Type: Journal    
DOI: 10.1007/s00424-009-0756-x     Document Type: Review

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