Kir6.2 mutations causing neonatal diabetes provide new insights into Kir6.2-SUR1 interactions

EMBO Journal

Volumn 24, Issue 13, 2005, Pages 2318-2330

  Tammaro, Paolo ^a   Girard, Christophe ^a   Molnes, Janne ^b   Njølstad, Pal R ^b,c   Ashcroft, Frances M ^a  

^a UNIVERSITY OF OXFORD   (United Kingdom)

^b UNIVERSITY OF BERGEN   (Norway)

^c HAUKELAND UNIVERSITY HOSPITAL   (Norway)

Author keywords

Diabetes; KATP channel; Kir6.2; SUR1

Indexed keywords

ADENOSINE TRIPHOSPHATE; ADENOSINE TRIPHOSPHATE MAGNESIUM; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR6.2; SULFONYLUREA RECEPTOR; SULFONYLUREA RECEPTOR 1; T LYMPHOCYTE RECEPTOR; UNCLASSIFIED DRUG;

ANIMAL CELL; ARTICLE; CONTROLLED STUDY; DIABETES MELLITUS; GENE MUTATION; HETEROLOGOUS EXPRESSION; HYDROLYSIS; IC 50; INSULIN RELEASE; MUTANT; NONHUMAN; NUCLEOTIDE SEQUENCE; OOCYTE; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; WILD TYPE; XENOPUS LAEVIS;

ADENOSINE TRIPHOSPHATE; ANIMALS; ATP-BINDING CASSETTE TRANSPORTERS; DIABETES MELLITUS; FEMALE; HUMANS; INFANT, NEWBORN; ION CHANNEL GATING; MODELS, MOLECULAR; MULTIDRUG RESISTANCE-ASSOCIATED PROTEINS; MUTAGENESIS, SITE-DIRECTED; MUTATION; OOCYTES; PATCH-CLAMP TECHNIQUES; POTASSIUM CHANNELS, INWARDLY RECTIFYING; PROTEIN BINDING; RATS; XENOPUS LAEVIS;

EID: 22744438517     PISSN: 02614189     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.emboj.7600715     Document Type: Article

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