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Proceedings of the National Academy of Sciences of the United States of America

Volumn 98, Issue 5, 2001, Pages 2882-2887

Defective trafficking and function of KATP channels caused by a sulfonylurea receptor 1 mutation associated with persistent hyperinsulinemic hypoglycemia of infancy

(4) Cartier, Etienne A a Conti, Lisa R b Vandenberg, Carol A b Shyng, Show Ling a

a OREGON HEALTH AND SCIENCE UNIVERSITY (United States)

b UNIVERSITY OF CALIFORNIA (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE DIPHOSPHATE; ADENOSINE TRIPHOSPHATE; DIAZOXIDE; INSULIN; PHENYLALANINE; POTASSIUM CHANNEL; SULFONYLUREA RECEPTOR; TRANSMEMBRANE CONDUCTANCE REGULATOR;

ANIMAL CELL; ARTICLE; ENDOPLASMIC RETICULUM; GENE DELETION; GENE EXPRESSION; GENE MUTATION; HYPERINSULINEMIA; HYPOGLYCEMIA; INFANCY; INSULIN RELEASE; INTRACELLULAR TRANSPORT; NONHUMAN; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

AMINO ACID MOTIFS; ANIMALS; ATP-BINDING CASSETTE TRANSPORTERS; COS CELLS; HYPERINSULINISM; HYPOGLYCEMIA; MEMBRANE POTENTIALS; MUTATION; POTASSIUM CHANNELS; POTASSIUM CHANNELS, INWARDLY RECTIFYING; PROTEIN TRANSPORT; RECEPTORS, DRUG;

ANIMALIA;

EID: 0035956875 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.051499698 Document Type: Article

Times cited : (149)

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