The function of classical and alternative non-homologous end-joining pathways in the fusion of dysfunctional telomeres

EMBO Journal

Volumn 29, Issue 15, 2010, Pages 2598-2610

  Rai, Rekha ^a,c   Zheng, Hong ^a   He, Hua ^a   Luo, Ying ^a   Multani, Asha ^a   Carpenter, Phillip B ^b   Chang, Sandy ^a,c  

^a UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER   (United States)

^b UNIVERSITY OF TEXAS MEDICAL SCHOOL   (United States)

^c YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

A NHEJ; C NHEJ; DNA damage; telomere

Indexed keywords

DNA; TELOMERASE; TELOMERE BINDING PROTEIN;

ARTICLE; CHROMOSOME; DNA REPAIR; DOUBLE STRANDED DNA BREAK; EMBRYO; GENOMIC INSTABILITY; HOMOLOGOUS RECOMBINATION; HUMAN; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN BINDING; TELOMERE;

ANIMALS; ANTIGENS, NUCLEAR; CELLS, CULTURED; DNA REPAIR; DNA-BINDING PROTEINS; HUMANS; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MICE; MICE, KNOCKOUT; TELOMERE; TELOMERIC REPEAT BINDING PROTEIN 2;

EID: 77955419733     PISSN: 02614189     EISSN: 14602075     Source Type: Journal    
DOI: 10.1038/emboj.2010.142     Document Type: Article

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