Presenilins regulate the cellular level of the tumor suppressor PTEN

Neurobiology of Aging

Volumn 29, Issue 5, 2008, Pages 653-660

  Zhang, Han ^a   Liu, Runzhong ^a   Wang, Ruishan ^a   Hong, Shuigen ^a   Xu, Huaxi ^a,b   Zhang, Yun wu ^a,b  

^a XIAMEN UNIVERSITY   (China)

^b BURNHAM INSTITUTE   (United States)

Author keywords

Akt; Alzheimer's Disease; Phosphatase and tensin homologue deleted on chromosome 10; Phosphoinositide 3 kinase; Presenilin

Indexed keywords

AMYLOID BETA PROTEIN; ANTERIOR PHARYNX DEFECTIVE 1 PROTEIN; BETA SECRETASE; GAMMA SECRETASE; NICASTRIN; PHOSPHATIDYLINOSITOL 3 KINASE; PHOSPHATIDYLINOSITOL 3,4,5 TRISPHOSPHATE 3 PHOSPHATASE; PRESENILIN 1; PRESENILIN 2; PROTEIN KINASE B;

ALZHEIMER DISEASE; ANIMAL CELL; ARTICLE; BRAIN CELL; CELL SURVIVAL; CONTROLLED STUDY; EMBRYO; IMMUNOHISTOCHEMISTRY; MOUSE; NONHUMAN; PATHOGENESIS; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION;

ANIMALS; CELL LINE; FIBROBLASTS; GENE EXPRESSION REGULATION; HUMANS; MICE; MICE, KNOCKOUT; NEURONS; PRESENILINS; PTEN PHOSPHOHYDROLASE;

EID: 40849115109     PISSN: 01974580     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neurobiolaging.2006.11.020     Document Type: Article

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