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Volumn 20, Issue 49 REV. IIS. 6, 2001, Pages 7257-7265

Pathways of retinoic acid- or arsenic trioxide-induced PML/RARα catabolism, role of oncogene degradation in disease remission

(3) Zhu, J a Lallemand Breitenbach, V a De The H a

a UNIVERSITÉ PARIS DESCARTES (France)

Author keywords

Apoptosis; Clinical trials; Differentiation; Nuclear bodies; Proteasome; SUMO

Indexed keywords

ARSENIC TRIOXIDE; CASPASE; HYBRID PROTEIN; PROTEASOME; RETINOIC ACID; RETINOIC ACID RECEPTOR; SUMO PROTEIN; UNCLASSIFIED DRUG;

ACUTE MYELOBLASTIC LEUKEMIA; APOPTOSIS; BINDING SITE; CANCER THERAPY; CELL DIFFERENTIATION; CELL NUCLEUS INCLUSION BODY; FEEDBACK SYSTEM; LEUKEMIA REMISSION; LEUKEMOGENESIS; MOLECULAR DYNAMICS; ONCOGENE; PRIORITY JOURNAL; PROTEIN DOMAIN; REVIEW;

ANIMALS; ARSENICALS; BINDING SITES; CASPASES; CELL DIFFERENTIATION; CYSTEINE ENDOPEPTIDASES; HUMANS; LEUKEMIA, PROMYELOCYTIC, ACUTE; MULTIENZYME COMPLEXES; NEOPLASM PROTEINS; ONCOGENE PROTEINS, FUSION; OXIDES; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTEIN STRUCTURE, TERTIARY; REMISSION INDUCTION; TRETINOIN;

EID: 0035969111 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204852 Document Type: Review

Times cited : (145)

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