Increased global transcription activity as a mechanism of replication stress in cancer

Nature Communications

Volumn 7, Issue , 2016, Pages

  Kotsantis, Panagiotis ^a   Silva, Lara Marques ^b   Irmscher, Sarah ^b   Jones, Rebecca M ^a   Folkes, Lisa ^c   Gromak, Natalia ^b   Petermann, Eva ^a  

^a UNIVERSITY OF BIRMINGHAM   (United Kingdom)

^b UNIVERSITY OF OXFORD   (United Kingdom)

^c UNIVERSITY OF OXFORD   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

BETA ACTIN; CHECKPOINT KINASE 1; CHECKPOINT KINASE 2; CYCLIN E; MESSENGER RNA; RIBONUCLEOSIDE; RNA; ROSCOVITINE; TATA BINDING PROTEIN; HRAS PROTEIN, HUMAN; PROTEIN P21; TBP PROTEIN, HUMAN; TP53BP1 PROTEIN, HUMAN; TUMOR SUPPRESSOR P53 BINDING PROTEIN 1;

CANCER; CYTOLOGY; DNA; GENE EXPRESSION; GENOME; PHYSIOLOGICAL RESPONSE; PROTEIN; RNA;

ACUTE MYELOBLASTIC LEUKEMIA; ARTICLE; BLADDER CARCINOMA; CELL CYCLE G1 PHASE; COLORECTAL CARCINOMA; CONTROLLED STUDY; CUTANEOUS MELANOMA; DNA DAMAGE; DNA RNA HYBRIDIZATION; FIBROBLAST; GENE OVEREXPRESSION; GENE REPLICATION; GENETIC TRANSCRIPTION; GENOMIC INSTABILITY; GLIOBLASTOMA; HUMAN; HUMAN CELL; LIVER CELL CARCINOMA; LUNG ADENOCARCINOMA; NEOPLASM; ONCOGENE C FOS; ONCOGENE MYC; ONCOGENE RAS; PARAGANGLIOMA; PHEOCHROMOCYTOMA; RNA SYNTHESIS; SENESCENCE; SQUAMOUS CELL LUNG CARCINOMA; STOMACH ADENOCARCINOMA; STRESS; TATA BOX; TRANSITIONAL CELL CARCINOMA; UPREGULATION; BIOSYNTHESIS; CELL AGING; CONFORMATION; DNA REPLICATION; GENE EXPRESSION REGULATION; GENETICS; METABOLISM; PATHOLOGY; PHYSIOLOGICAL STRESS; TUMOR CELL LINE;

CELL LINE, TUMOR; CELLULAR SENESCENCE; DNA REPLICATION; GENE EXPRESSION REGULATION, NEOPLASTIC; GENOMIC INSTABILITY; HUMANS; NEOPLASMS; NUCLEIC ACID CONFORMATION; PROTO-ONCOGENE PROTEINS P21(RAS); RNA, MESSENGER; RNA, NEOPLASM; STRESS, PHYSIOLOGICAL; TATA-BOX BINDING PROTEIN; TRANSCRIPTION, GENETIC; TUMOR SUPPRESSOR P53-BINDING PROTEIN 1;

EID: 84991510615     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms13087     Document Type: Article

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