Immunological mechanisms contributing to the double burden of diabetes and intracellular bacterial infections

Immunology

Volumn 144, Issue 2, 2015, Pages 171-185

  Hodgson, Kelly ^a   Morris, Jodie ^a   Bridson, Tahnee ^a   Govan, Brenda ^a   Rush, Catherine ^a   Ketheesan, Natkunam ^a  

^a JAMES COOK UNIVERSITY   (Australia)

Author keywords

Cell mediated immunity; Diabetes; Inflammation; Intracellular bacterial infections; Melioidosis; Tuberculosis

Indexed keywords

ADVANCED GLYCATION END PRODUCT; CYTOKINE; GAMMA INTERFERON; GLUCOSE;

ADAPTIVE IMMUNITY; ANTIBACTERIAL ACTIVITY; ANTIGEN PRESENTING CELL; BACTERIAL INFECTION; CHRONIC INFLAMMATION; DENDRITIC CELL; DIABETES MELLITUS; DISEASE EXACERBATION; GLUCOSE METABOLISM; HOST PATHOGEN INTERACTION; HUMAN; IMMUNE RESPONSE; IMMUNOREGULATION; INFLAMMATION; MACROPHAGE; NATURAL KILLER CELL; NATURAL KILLER T CELL; NEUTROPHIL; NONHUMAN; OXIDATION; OXIDATIVE STRESS; PHAGOCYTOSIS; PRIORITY JOURNAL; REVIEW; RISK FACTOR; T LYMPHOCYTE; ACTIVE IMMUNIZATION; BACTERIAL INFECTIONS; BIOSYNTHESIS; IMMUNOLOGY; METABOLISM;

BACTERIAL INFECTIONS; DENDRITIC CELLS; DIABETES MELLITUS; GLYCOSYLATION END PRODUCTS, ADVANCED; HUMANS; IMMUNITY, ACTIVE; INFLAMMATION; INTERFERON-GAMMA; KILLER CELLS, NATURAL; MACROPHAGES; NEUTROPHILS; OXIDATIVE STRESS; PHAGOCYTOSIS; RISK FACTORS; T-LYMPHOCYTES;

EID: 84922502607     PISSN: 00192805     EISSN: 13652567     Source Type: Journal    
DOI: 10.1111/imm.12394     Document Type: Review

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