-
1
-
-
33746088001
-
Trends in prevalence and outcome of heart failure with preserved ejection fraction
-
PID: 16855265, COI: 1:CAS:528:DC%2BD28XntVWktr0%3D
-
Owan TE, Hodge DO, Herges RM, et al. Trends in prevalence and outcome of heart failure with preserved ejection fraction. N Engl J Med. 2006;355:251–9.
-
(2006)
N Engl J Med
, vol.355
, pp. 251-259
-
-
Owan, T.E.1
Hodge, D.O.2
Herges, R.M.3
-
2
-
-
79952829685
-
Heart failure with preserved ejection fraction: pathophysiology, diagnosis, and treatment
-
PID: 21138935
-
Borlaug BA, Paulus WJ. Heart failure with preserved ejection fraction: pathophysiology, diagnosis, and treatment. Eur Heart J. 2011;32:670–9.
-
(2011)
Eur Heart J
, vol.32
, pp. 670-679
-
-
Borlaug, B.A.1
Paulus, W.J.2
-
3
-
-
0031752685
-
Global burden of diabetes, 1995–2025: prevalence, numerical estimates, and projections
-
PID: 9727886, COI: 1:STN:280:DyaK1czpsVyluw%3D%3D
-
King H, Aubert RE, Herman WH. Global burden of diabetes, 1995–2025: prevalence, numerical estimates, and projections. Diabetes Care. 1998;21:1414–31.
-
(1998)
Diabetes Care
, vol.21
, pp. 1414-1431
-
-
King, H.1
Aubert, R.E.2
Herman, W.H.3
-
4
-
-
84856487711
-
Prevalence of obesity and trends in the distribution of body mass index among US adults, 1999–2010
-
PID: 22253363
-
Flegal KM, Carroll MD, Kit BK, Ogden CL. Prevalence of obesity and trends in the distribution of body mass index among US adults, 1999–2010. JAMA. 2012;307:491–7.
-
(2012)
JAMA
, vol.307
, pp. 491-497
-
-
Flegal, K.M.1
Carroll, M.D.2
Kit, B.K.3
Ogden, C.L.4
-
5
-
-
84879756170
-
Myocardial titin and collagen in cardiac diastolic dysfunction: partners in crime
-
PID: 23709670
-
Hamdani N, Paulus WJ. Myocardial titin and collagen in cardiac diastolic dysfunction: partners in crime. Circulation. 2013;128:5–8.
-
(2013)
Circulation
, vol.128
, pp. 5-8
-
-
Hamdani, N.1
Paulus, W.J.2
-
6
-
-
84879753113
-
Shortening of the elastic tandem immunoglobulin segment of titin leads to diastolic dysfunction
-
PID: 23709671, COI: 1:CAS:528:DC%2BC3sXhtVKqu7%2FN, This article provides first evidence that titin stiffness, in the absence of any involvement of the extracellular matrix, is sufficient to induce HFpEF
-
Chung CS, Hutchinson KR, Methawasin M, et al. Shortening of the elastic tandem immunoglobulin segment of titin leads to diastolic dysfunction. Circulation. 2013;128:19–28. This article provides first evidence that titin stiffness, in the absence of any involvement of the extracellular matrix, is sufficient to induce HFpEF.
-
(2013)
Circulation
, vol.128
, pp. 19-28
-
-
Chung, C.S.1
Hutchinson, K.R.2
Methawasin, M.3
-
7
-
-
84879748718
-
A novel paradigm for heart failure with preserved ejection fraction: comorbidities drive myocardial dysfunction and remodeling through coronary microvascular endothelial inflammation
-
PID: 23684677, This review postulates a new paradigm for the development of HFpEF, which identifies a systemic proinflammatory state induced by comorbidities as the origin of microvascular endothelial cell inflammation and subsequent concentric cardiac remodeling and dysfunction
-
Paulus WJ, Tschope C. A novel paradigm for heart failure with preserved ejection fraction: comorbidities drive myocardial dysfunction and remodeling through coronary microvascular endothelial inflammation. J Am Coll Cardiol. 2013;62:263–71. This review postulates a new paradigm for the development of HFpEF, which identifies a systemic proinflammatory state induced by comorbidities as the origin of microvascular endothelial cell inflammation and subsequent concentric cardiac remodeling and dysfunction.
-
(2013)
J Am Coll Cardiol
, vol.62
, pp. 263-271
-
-
Paulus, W.J.1
Tschope, C.2
-
8
-
-
0037207468
-
Cardiac endothelial-myocardial signaling: its role in cardiac growth, contractile performance, and rhythmicity
-
PID: 12506127, COI: 1:CAS:528:DC%2BD3sXntlGgsw%3D%3D
-
Brutsaert DL. Cardiac endothelial-myocardial signaling: its role in cardiac growth, contractile performance, and rhythmicity. Physiol Rev. 2003;83:59–115.
-
(2003)
Physiol Rev
, vol.83
, pp. 59-115
-
-
Brutsaert, D.L.1
-
9
-
-
0029047323
-
Nitric oxide modulates the expression of monocyte chemoattractant protein 1 in cultured human endothelial cells
-
PID: 7758169, COI: 1:CAS:528:DyaK2MXlvVShtrw%3D
-
Zeiher AM, Fisslthaler B, Schray-Utz B, Busse R. Nitric oxide modulates the expression of monocyte chemoattractant protein 1 in cultured human endothelial cells. Circ Res. 1995;76:980–6.
-
(1995)
Circ Res
, vol.76
, pp. 980-986
-
-
Zeiher, A.M.1
Fisslthaler, B.2
Schray-Utz, B.3
Busse, R.4
-
10
-
-
0029786764
-
Nitric oxide regulates vascular cell adhesion molecule 1 gene expression and redox-sensitive transcriptional events in human vascular endothelial cells
-
PID: 8799163, COI: 1:CAS:528:DyaK28Xlt1Chtrw%3D
-
Khan BV, Harrison DG, Olbrych MT, et al. Nitric oxide regulates vascular cell adhesion molecule 1 gene expression and redox-sensitive transcriptional events in human vascular endothelial cells. Proc Natl Acad Sci U S A. 1996;93:9114–9.
-
(1996)
Proc Natl Acad Sci U S A
, vol.93
, pp. 9114-9119
-
-
Khan, B.V.1
Harrison, D.G.2
Olbrych, M.T.3
-
11
-
-
0028304674
-
Intracellular oxidative stress induced by nitric oxide synthesis inhibition increases endothelial cell adhesion to neutrophils
-
PID: 7910528, COI: 1:CAS:528:DyaK2cXkvVSrtLo%3D
-
Niu XF, Smith CW, Kubes P. Intracellular oxidative stress induced by nitric oxide synthesis inhibition increases endothelial cell adhesion to neutrophils. Circ Res. 1994;74:1133–40.
-
(1994)
Circ Res
, vol.74
, pp. 1133-1140
-
-
Niu, X.F.1
Smith, C.W.2
Kubes, P.3
-
12
-
-
55649114859
-
Enhancement of endothelial nitric oxide synthase production reverses vascular dysfunction and inflammation in the hindlimbs of a rat model of diabetes
-
PID: 18825362, COI: 1:CAS:528:DC%2BD1cXhtlCjs7%2FK
-
Riad A, Westermann D, Van Linthout S, et al. Enhancement of endothelial nitric oxide synthase production reverses vascular dysfunction and inflammation in the hindlimbs of a rat model of diabetes. Diabetologia. 2008;51:2325–32.
-
(2008)
Diabetologia
, vol.51
, pp. 2325-2332
-
-
Riad, A.1
Westermann, D.2
Van Linthout, S.3
-
13
-
-
0031037180
-
Suppression of apoptosis by nitric oxide via inhibition of interleukin-1beta-converting enzyme (ICE)-like and cysteine protease protein (CPP)-32-like proteases
-
PID: 9034139, COI: 1:CAS:528:DyaK2sXhtlWnurk%3D
-
Dimmeler S, Haendeler J, Nehls M, Zeiher AM. Suppression of apoptosis by nitric oxide via inhibition of interleukin-1beta-converting enzyme (ICE)-like and cysteine protease protein (CPP)-32-like proteases. J Exp Med. 1997;185:601–7.
-
(1997)
J Exp Med
, vol.185
, pp. 601-607
-
-
Dimmeler, S.1
Haendeler, J.2
Nehls, M.3
Zeiher, A.M.4
-
14
-
-
8344222641
-
Nitric oxide-releasing aspirin derivative, NCX 4016, promotes reparative angiogenesis and prevents apoptosis and oxidative stress in a mouse model of peripheral ischemia
-
PID: 15345513, COI: 1:CAS:528:DC%2BD2cXptV2ju7Y%3D
-
Emanueli C, Van Linthout S, Salis MB, et al. Nitric oxide-releasing aspirin derivative, NCX 4016, promotes reparative angiogenesis and prevents apoptosis and oxidative stress in a mouse model of peripheral ischemia. Arterioscler Thromb Vasc Biol. 2004;24:2082–7.
-
(2004)
Arterioscler Thromb Vasc Biol
, vol.24
, pp. 2082-2087
-
-
Emanueli, C.1
Van Linthout, S.2
Salis, M.B.3
-
15
-
-
20044373069
-
Endothelial function and dysfunction. Part II: association with cardiovascular risk factors and diseases. A statement by the Working Group on Endothelins and Endothelial Factors of the European Society of Hypertension
-
PID: 15662207, COI: 1:CAS:528:DC%2BD2MXmtFCisg%3D%3D
-
Brunner H, Cockcroft JR, Deanfield J, et al. Endothelial function and dysfunction. Part II: association with cardiovascular risk factors and diseases. A statement by the Working Group on Endothelins and Endothelial Factors of the European Society of Hypertension. J Hypertens. 2005;23:233–46.
-
(2005)
J Hypertens
, vol.23
, pp. 233-246
-
-
Brunner, H.1
Cockcroft, J.R.2
Deanfield, J.3
-
16
-
-
34547912897
-
Tumor necrosis factor-alpha reduces argininosuccinate synthase expression and nitric oxide production in aortic endothelial cells
-
PID: 17496212, COI: 1:CAS:528:DC%2BD2sXptlyku7c%3D
-
Goodwin BL, Pendleton LC, Levy MM, et al. Tumor necrosis factor-alpha reduces argininosuccinate synthase expression and nitric oxide production in aortic endothelial cells. Am J Physiol Heart Circ Physiol. 2007;293:H1115–21.
-
(2007)
Am J Physiol Heart Circ Physiol
, vol.293
, pp. H1115-H1121
-
-
Goodwin, B.L.1
Pendleton, L.C.2
Levy, M.M.3
-
17
-
-
34249319470
-
TNF-alpha contributes to endothelial dysfunction by upregulating arginase in ischemia/reperfusion injury
-
PID: 17413034, COI: 1:CAS:528:DC%2BD2sXltl2iur8%3D
-
Gao X, Xu X, Belmadani S, et al. TNF-alpha contributes to endothelial dysfunction by upregulating arginase in ischemia/reperfusion injury. Arterioscler Thromb Vasc Biol. 2007;27:1269–75.
-
(2007)
Arterioscler Thromb Vasc Biol
, vol.27
, pp. 1269-1275
-
-
Gao, X.1
Xu, X.2
Belmadani, S.3
-
18
-
-
84891600238
-
LXR agonism improves TNF-alpha-induced endothelial dysfunction in the absence of its cholesterol-modulating effects
-
PID: 24401210, COI: 1:CAS:528:DC%2BC2cXksV2qtQ%3D%3D
-
Spillmann F, Van Linthout S, Miteva K, et al. LXR agonism improves TNF-alpha-induced endothelial dysfunction in the absence of its cholesterol-modulating effects. Atherosclerosis. 2014;232:1–9.
-
(2014)
Atherosclerosis
, vol.232
, pp. 1-9
-
-
Spillmann, F.1
Van Linthout, S.2
Miteva, K.3
-
19
-
-
64849104331
-
Vascular-protective effects of high-density lipoprotein include the downregulation of the angiotensin II type 1 receptor
-
PID: 19273745, COI: 1:CAS:528:DC%2BD1MXivVemt78%3D
-
Van Linthout S, Spillmann F, Lorenz M, et al. Vascular-protective effects of high-density lipoprotein include the downregulation of the angiotensin II type 1 receptor. Hypertension. 2009;53:682–7.
-
(2009)
Hypertension
, vol.53
, pp. 682-687
-
-
Van Linthout, S.1
Spillmann, F.2
Lorenz, M.3
-
20
-
-
84884586939
-
Role of tetrahydrobiopterin in pulmonary vascular remodelling associated with pulmonary fibrosis
-
PID: 23739137
-
Almudever P, Milara J, De Diego A, et al. Role of tetrahydrobiopterin in pulmonary vascular remodelling associated with pulmonary fibrosis. Thorax. 2013;68:938–48.
-
(2013)
Thorax
, vol.68
, pp. 938-948
-
-
Almudever, P.1
Milara, J.2
De Diego, A.3
-
21
-
-
24944458112
-
Endothelial nitric oxide synthase dysfunction in diabetic mice: importance of tetrahydrobiopterin in eNOS dimerisation
-
PID: 16034613, COI: 1:CAS:528:DC%2BD2MXhtVSqtL7K
-
Cai S, Khoo J, Mussa S, et al. Endothelial nitric oxide synthase dysfunction in diabetic mice: importance of tetrahydrobiopterin in eNOS dimerisation. Diabetologia. 2005;48:1933–40.
-
(2005)
Diabetologia
, vol.48
, pp. 1933-1940
-
-
Cai, S.1
Khoo, J.2
Mussa, S.3
-
22
-
-
57349197115
-
Role of TNF-alpha-induced reactive oxygen species in endothelial dysfunction during reperfusion injury
-
PID: 18849334, COI: 1:CAS:528:DC%2BD1cXhsFSntb3N
-
Gao X, Zhang H, Belmadani S, et al. Role of TNF-alpha-induced reactive oxygen species in endothelial dysfunction during reperfusion injury. Am J Physiol Heart Circ Physiol. 2008;295:H2242–9.
-
(2008)
Am J Physiol Heart Circ Physiol
, vol.295
, pp. H2242-H2249
-
-
Gao, X.1
Zhang, H.2
Belmadani, S.3
-
23
-
-
70349675632
-
Molecular insights and therapeutic targets for diabetic endothelial dysfunction
-
PID: 19786641
-
Xu J, Zou MH. Molecular insights and therapeutic targets for diabetic endothelial dysfunction. Circulation. 2009;120:1266–86.
-
(2009)
Circulation
, vol.120
, pp. 1266-1286
-
-
Xu, J.1
Zou, M.H.2
-
24
-
-
33646013920
-
Endothelial-cardiomyocyte interactions in cardiac development and repair
-
PID: 16460266, COI: 1:CAS:528:DC%2BD28XksFWksr8%3D
-
Hsieh PC, Davis ME, Lisowski LK, Lee RT. Endothelial-cardiomyocyte interactions in cardiac development and repair. Annu Rev Physiol. 2006;68:51–66.
-
(2006)
Annu Rev Physiol
, vol.68
, pp. 51-66
-
-
Hsieh, P.C.1
Davis, M.E.2
Lisowski, L.K.3
Lee, R.T.4
-
25
-
-
0029940216
-
Paracrine modulation of heart cell function by endothelial cells
-
PID: 8759239, COI: 1:STN:280:DyaK28zjs1Slsg%3D%3D
-
Shah AM. Paracrine modulation of heart cell function by endothelial cells. Cardiovasc Res. 1996;31:847–67.
-
(1996)
Cardiovasc Res
, vol.31
, pp. 847-867
-
-
Shah, A.M.1
-
26
-
-
84899126216
-
ROS signalling between endothelial cells and cardiac cells
-
PID: 24591150, COI: 1:CAS:528:DC%2BC2cXmsVKhsb0%3D
-
Zhang M, Shah AM. ROS signalling between endothelial cells and cardiac cells. Cardiovasc Res. 2014;102:249–57.
-
(2014)
Cardiovasc Res
, vol.102
, pp. 249-257
-
-
Zhang, M.1
Shah, A.M.2
-
27
-
-
0023905907
-
Effects of damaging the endocardial surface on the mechanical performance of isolated cardiac muscle
-
PID: 3338120, COI: 1:STN:280:DyaL1c7gvFyluw%3D%3D
-
Brutsaert DL, Meulemans AL, Sipido KR, Sys SU. Effects of damaging the endocardial surface on the mechanical performance of isolated cardiac muscle. Circ Res. 1988;62:358–66.
-
(1988)
Circ Res
, vol.62
, pp. 358-366
-
-
Brutsaert, D.L.1
Meulemans, A.L.2
Sipido, K.R.3
Sys, S.U.4
-
28
-
-
0028823029
-
Paracrine coronary endothelial control of left ventricular function in humans
-
PID: 7554191, COI: 1:STN:280:DyaK28%2FhslartQ%3D%3D
-
Paulus WJ, Vantrimpont PJ, Shah AM. Paracrine coronary endothelial control of left ventricular function in humans. Circulation. 1995;92:2119–26.
-
(1995)
Circulation
, vol.92
, pp. 2119-2126
-
-
Paulus, W.J.1
Vantrimpont, P.J.2
Shah, A.M.3
-
29
-
-
20044374676
-
High prevalence of cardiac parvovirus B19 infection in patients with isolated left ventricular diastolic dysfunction
-
PID: 15710767, COI: 1:STN:280:DC%2BD2M%2FptlGjsA%3D%3D
-
Tschope C, Bock CT, Kasner M, et al. High prevalence of cardiac parvovirus B19 infection in patients with isolated left ventricular diastolic dysfunction. Circulation. 2005;111:879–86.
-
(2005)
Circulation
, vol.111
, pp. 879-886
-
-
Tschope, C.1
Bock, C.T.2
Kasner, M.3
-
30
-
-
84867883843
-
Endothelial dysfunction: a pathophysiologic factor in heart failure with preserved ejection fraction
-
PID: 23040569
-
Lam CS, Brutsaert DL. Endothelial dysfunction: a pathophysiologic factor in heart failure with preserved ejection fraction. J Am Coll Cardiol. 2012;60:1787–9.
-
(2012)
J Am Coll Cardiol
, vol.60
, pp. 1787-1789
-
-
Lam, C.S.1
Brutsaert, D.L.2
-
31
-
-
84899129310
-
Endothelial NADPH oxidase-2 promotes interstitial cardiac fibrosis and diastolic dysfunction through pro-inflammatory effects and endothelial-mesenchymal transition
-
Murdoch CE, Chaubey S, Zeng L, et al. Endothelial NADPH oxidase-2 promotes interstitial cardiac fibrosis and diastolic dysfunction through pro-inflammatory effects and endothelial-mesenchymal transition. J Am Coll Cardiol. 2014;63:2734–41.
-
(2014)
J Am Coll Cardiol
, vol.63
, pp. 2734-2741
-
-
Murdoch, C.E.1
Chaubey, S.2
Zeng, L.3
-
32
-
-
84899048801
-
Crosstalk between fibroblasts and inflammatory cells
-
PID: 24728497, COI: 1:CAS:528:DC%2BC2cXmsVKhsLo%3D, This review gives an extensive overview about how inflammatory cells contribute to (cardiac) fibroblast activation and how in turn fibroblasts contribute to the inflammatory process leading to chronic inflammation
-
Van Linthout S, Miteva K, Tschope C. Crosstalk between fibroblasts and inflammatory cells. Cardiovasc Res. 2014;102:258–69. This review gives an extensive overview about how inflammatory cells contribute to (cardiac) fibroblast activation and how in turn fibroblasts contribute to the inflammatory process leading to chronic inflammation.
-
(2014)
Cardiovasc Res
, vol.102
, pp. 258-269
-
-
Van Linthout, S.1
Miteva, K.2
Tschope, C.3
-
33
-
-
84897109919
-
Remodeling of the mononuclear phagocyte network underlies chronic inflammation and disease progression in heart failure: critical importance of the cardiosplenic axis
-
PID: 24186967, COI: 1:CAS:528:DC%2BC2cXhvVSktLg%3D
-
Ismahil MA, Hamid T, Bansal SS, et al. Remodeling of the mononuclear phagocyte network underlies chronic inflammation and disease progression in heart failure: critical importance of the cardiosplenic axis. Circ Res. 2014;114:266–82.
-
(2014)
Circ Res
, vol.114
, pp. 266-282
-
-
Ismahil, M.A.1
Hamid, T.2
Bansal, S.S.3
-
34
-
-
84863992805
-
Mesenchymal stromal cells but not cardiac fibroblasts exert beneficial systemic immunomodulatory effects in experimental myocarditis
-
PID: 22815907, COI: 1:CAS:528:DC%2BC38XhtVyrsrjO
-
Savvatis K, van Linthout S, Miteva K, et al. Mesenchymal stromal cells but not cardiac fibroblasts exert beneficial systemic immunomodulatory effects in experimental myocarditis. PLoS One. 2012;7:e41047.
-
(2012)
PLoS One
, vol.7
, pp. e41047
-
-
Savvatis, K.1
van Linthout, S.2
Miteva, K.3
-
35
-
-
84894288342
-
Monocyte subset accumulation in the human heart following acute myocardial infarction and the role of the spleen as monocyte reservoir
-
PID: 23966310, COI: 1:CAS:528:DC%2BC2cXit12itbY%3D
-
van der Laan AM, Ter Horst EN, Delewi R, et al. Monocyte subset accumulation in the human heart following acute myocardial infarction and the role of the spleen as monocyte reservoir. Eur Heart J. 2014;35:376–85.
-
(2014)
Eur Heart J
, vol.35
, pp. 376-385
-
-
van der Laan, A.M.1
Ter Horst, E.N.2
Delewi, R.3
-
36
-
-
79952260400
-
Cardiac inflammation contributes to changes in the extracellular matrix in patients with heart failure and normal ejection fraction
-
PID: 21075869
-
Westermann D, Lindner D, Kasner M, et al. Cardiac inflammation contributes to changes in the extracellular matrix in patients with heart failure and normal ejection fraction. Circ Heart Fail. 2011;4:44–52.
-
(2011)
Circ Heart Fail
, vol.4
, pp. 44-52
-
-
Westermann, D.1
Lindner, D.2
Kasner, M.3
-
37
-
-
79952262784
-
Transforming growth factor-beta: governing the transition from inflammation to fibrosis in heart failure with preserved left ventricular function
-
PID: 21245455
-
Kapur NK. Transforming growth factor-beta: governing the transition from inflammation to fibrosis in heart failure with preserved left ventricular function. Circ Heart Fail. 2011;4:5–7.
-
(2011)
Circ Heart Fail
, vol.4
, pp. 5-7
-
-
Kapur, N.K.1
-
38
-
-
0036645337
-
Transforming growth factor-beta function blocking prevents myocardial fibrosis and diastolic dysfunction in pressure-overloaded rats
-
PID: 12093782, COI: 1:CAS:528:DC%2BD38XlvVWit7w%3D
-
Kuwahara F, Kai H, Tokuda K, et al. Transforming growth factor-beta function blocking prevents myocardial fibrosis and diastolic dysfunction in pressure-overloaded rats. Circulation. 2002;106:130–5.
-
(2002)
Circulation
, vol.106
, pp. 130-135
-
-
Kuwahara, F.1
Kai, H.2
Tokuda, K.3
-
39
-
-
79957624520
-
Fibrocytes: emerging effector cells in chronic inflammation
-
PID: 21597472, COI: 1:CAS:528:DC%2BC3MXmtlemtLk%3D
-
Reilkoff RA, Bucala R, Herzog EL. Fibrocytes: emerging effector cells in chronic inflammation. Nat Rev Immunol. 2011;11:427–35.
-
(2011)
Nat Rev Immunol
, vol.11
, pp. 427-435
-
-
Reilkoff, R.A.1
Bucala, R.2
Herzog, E.L.3
-
40
-
-
84892850549
-
Class I HDACs regulate angiotensin II-dependent cardiac fibrosis via fibroblasts and circulating fibrocytes
-
PID: 24374140, COI: 1:CAS:528:DC%2BC2cXht1KitLw%3D
-
Williams SM, Golden-Mason L, Ferguson BS, et al. Class I HDACs regulate angiotensin II-dependent cardiac fibrosis via fibroblasts and circulating fibrocytes. J Mol Cell Cardiol. 2014;67:112–25.
-
(2014)
J Mol Cell Cardiol
, vol.67
, pp. 112-125
-
-
Williams, S.M.1
Golden-Mason, L.2
Ferguson, B.S.3
-
41
-
-
41149122223
-
Atrial natriuretic peptide inhibits transforming growth factor beta-induced Smad signaling and myofibroblast transformation in mouse cardiac fibroblasts
-
PID: 17991884, COI: 1:CAS:528:DC%2BD1cXhtVGktL0%3D
-
Li P, Wang D, Lucas J, et al. Atrial natriuretic peptide inhibits transforming growth factor beta-induced Smad signaling and myofibroblast transformation in mouse cardiac fibroblasts. Circ Res. 2008;102:185–92.
-
(2008)
Circ Res
, vol.102
, pp. 185-192
-
-
Li, P.1
Wang, D.2
Lucas, J.3
-
42
-
-
84900817740
-
PDE2-mediated cAMP hydrolysis accelerates cardiac fibroblast to myofibroblast conversion and is antagonized by exogenous activation of cGMP signaling pathways
-
PID: 24531807, COI: 1:CAS:528:DC%2BC2cXns1Grsbg%3D
-
Vettel C, Lammle S, Ewens S, et al. PDE2-mediated cAMP hydrolysis accelerates cardiac fibroblast to myofibroblast conversion and is antagonized by exogenous activation of cGMP signaling pathways. Am J Physiol Heart Circ Physiol. 2014;306:H1246–52.
-
(2014)
Am J Physiol Heart Circ Physiol
, vol.306
, pp. H1246-H1252
-
-
Vettel, C.1
Lammle, S.2
Ewens, S.3
-
43
-
-
84886059882
-
Phosphodiesterase-2 is up-regulated in human failing hearts and blunts beta-adrenergic responses in cardiomyocytes
-
PID: 23810893, COI: 1:CAS:528:DC%2BC3sXhvVagsrvP
-
Mehel H, Emons J, Vettel C, et al. Phosphodiesterase-2 is up-regulated in human failing hearts and blunts beta-adrenergic responses in cardiomyocytes. J Am Coll Cardiol. 2013;62:1596–606.
-
(2013)
J Am Coll Cardiol
, vol.62
, pp. 1596-1606
-
-
Mehel, H.1
Emons, J.2
Vettel, C.3
-
44
-
-
84868219699
-
Selective PDE5A inhibition with sildenafil rescues left ventricular dysfunction, inflammatory immune response and cardiac remodeling in angiotensin II-induced heart failure in vivo
-
PID: 23117837, COI: 1:CAS:528:DC%2BC38XhslOhsLjJ
-
Westermann D, Becher PM, Lindner D, et al. Selective PDE5A inhibition with sildenafil rescues left ventricular dysfunction, inflammatory immune response and cardiac remodeling in angiotensin II-induced heart failure in vivo. Basic Res Cardiol. 2012;107:308.
-
(2012)
Basic Res Cardiol
, vol.107
, pp. 308
-
-
Westermann, D.1
Becher, P.M.2
Lindner, D.3
-
45
-
-
77950909714
-
Reduced NO-cGMP signaling contributes to vascular inflammation and insulin resistance induced by high-fat feeding
-
PID: 20093624, COI: 1:CAS:528:DC%2BC3cXktFOmu7s%3D
-
Rizzo NO, Maloney E, Pham M, et al. Reduced NO-cGMP signaling contributes to vascular inflammation and insulin resistance induced by high-fat feeding. Arterioscler Thromb Vasc Biol. 2010;30:758–65.
-
(2010)
Arterioscler Thromb Vasc Biol
, vol.30
, pp. 758-765
-
-
Rizzo, N.O.1
Maloney, E.2
Pham, M.3
-
46
-
-
34548498378
-
Inhibition of TGF-beta induced lung fibroblast to myofibroblast conversion by phosphodiesterase inhibiting drugs and activators of soluble guanylyl cyclase
-
PID: 17659276, COI: 1:CAS:528:DC%2BD2sXhtVCitrbK
-
Dunkern TR, Feurstein D, Rossi GA, et al. Inhibition of TGF-beta induced lung fibroblast to myofibroblast conversion by phosphodiesterase inhibiting drugs and activators of soluble guanylyl cyclase. Eur J Pharmacol. 2007;572:12–22.
-
(2007)
Eur J Pharmacol
, vol.572
, pp. 12-22
-
-
Dunkern, T.R.1
Feurstein, D.2
Rossi, G.A.3
-
47
-
-
84875531214
-
Effect of phosphodiesterase-5 inhibition on exercise capacity and clinical status in heart failure with preserved ejection fraction: a randomized clinical trial
-
PID: 23478662, COI: 1:CAS:528:DC%2BC3sXlvFSgs7Y%3D
-
Redfield MM, Chen HH, Borlaug BA, et al. Effect of phosphodiesterase-5 inhibition on exercise capacity and clinical status in heart failure with preserved ejection fraction: a randomized clinical trial. JAMA. 2013;309:1268–77.
-
(2013)
JAMA
, vol.309
, pp. 1268-1277
-
-
Redfield, M.M.1
Chen, H.H.2
Borlaug, B.A.3
-
48
-
-
33846205660
-
Chronic NOS inhibition actuates endothelial-mesenchymal transformation
-
PID: 16963618, COI: 1:CAS:528:DC%2BD2sXisFGisro%3D
-
O’Riordan E, Mendelev N, Patschan S, et al. Chronic NOS inhibition actuates endothelial-mesenchymal transformation. Am J Physiol Heart Circ Physiol. 2007;292:H285–94.
-
(2007)
Am J Physiol Heart Circ Physiol
, vol.292
, pp. H285-H294
-
-
O’Riordan, E.1
Mendelev, N.2
Patschan, S.3
-
49
-
-
34547676391
-
Endothelial-to-mesenchymal transition contributes to cardiac fibrosis
-
PID: 17660828, COI: 1:CAS:528:DC%2BD2sXos1Cjsbw%3D
-
Zeisberg EM, Tarnavski O, Zeisberg M, et al. Endothelial-to-mesenchymal transition contributes to cardiac fibrosis. Nat Med. 2007;13:952–61.
-
(2007)
Nat Med
, vol.13
, pp. 952-961
-
-
Zeisberg, E.M.1
Tarnavski, O.2
Zeisberg, M.3
-
50
-
-
80053462373
-
Replicative aging induces endothelial to mesenchymal transition in human aortic endothelial cells: potential role of inflammation
-
PID: 21985896
-
Fleenor BS, Marshall KD, Rippe C, Seals DR. Replicative aging induces endothelial to mesenchymal transition in human aortic endothelial cells: potential role of inflammation. J Vasc Res. 2012;49:59–64.
-
(2012)
J Vasc Res
, vol.49
, pp. 59-64
-
-
Fleenor, B.S.1
Marshall, K.D.2
Rippe, C.3
Seals, D.R.4
-
51
-
-
84877307533
-
The effect of oxidized low-density lipoprotein (ox-LDL) on radiation-induced endothelial-to-mesenchymal transition
-
PID: 23289363, COI: 1:CAS:528:DC%2BC3sXmvVSmu7k%3D
-
Kim M, Choi SH, Jin YB, et al. The effect of oxidized low-density lipoprotein (ox-LDL) on radiation-induced endothelial-to-mesenchymal transition. Int J Radiat Biol. 2013;89:356–63.
-
(2013)
Int J Radiat Biol
, vol.89
, pp. 356-363
-
-
Kim, M.1
Choi, S.H.2
Jin, Y.B.3
-
52
-
-
77953364227
-
Endothelial cell-derived endothelin-1 promotes cardiac fibrosis in diabetic hearts through stimulation of endothelial-to-mesenchymal transition
-
PID: 20497976, COI: 1:CAS:528:DC%2BC3cXntF2itr4%3D
-
Widyantoro B, Emoto N, Nakayama K, et al. Endothelial cell-derived endothelin-1 promotes cardiac fibrosis in diabetic hearts through stimulation of endothelial-to-mesenchymal transition. Circulation. 2010;121:2407–18.
-
(2010)
Circulation
, vol.121
, pp. 2407-2418
-
-
Widyantoro, B.1
Emoto, N.2
Nakayama, K.3
-
53
-
-
84905292022
-
Cardiac fibroblasts support cardiac inflammation in heart failure
-
PID: 25086637
-
Lindner D, Zietsch C, Tank J, et al. Cardiac fibroblasts support cardiac inflammation in heart failure. Basic Res Cardiol. 2014;109:428.
-
(2014)
Basic Res Cardiol
, vol.109
, pp. 428
-
-
Lindner, D.1
Zietsch, C.2
Tank, J.3
-
54
-
-
84876795148
-
Therapeutic potential of tumour necrosis factor-alpha antagonists in patients with chronic heart failure
-
PID: 23337264
-
Javed Q, Murtaza I. Therapeutic potential of tumour necrosis factor-alpha antagonists in patients with chronic heart failure. Heart Lung Circ. 2013;22:323–7.
-
(2013)
Heart Lung Circ
, vol.22
, pp. 323-327
-
-
Javed, Q.1
Murtaza, I.2
-
55
-
-
10944236872
-
Cardiac endothelial cells regulate reactive oxygen species-induced cardiomyocyte apoptosis through neuregulin-1beta/erbB4 signaling
-
PID: 15385548, COI: 1:CAS:528:DC%2BD2cXhtVaqsrvN
-
Kuramochi Y, Cote GM, Guo X, et al. Cardiac endothelial cells regulate reactive oxygen species-induced cardiomyocyte apoptosis through neuregulin-1beta/erbB4 signaling. J Biol Chem. 2004;279:51141–7.
-
(2004)
J Biol Chem
, vol.279
, pp. 51141-51147
-
-
Kuramochi, Y.1
Cote, G.M.2
Guo, X.3
-
56
-
-
4344563450
-
Endothelial cells promote cardiac myocyte survival and spatial reorganization: implications for cardiac regeneration
-
PID: 15302801
-
Narmoneva DA, Vukmirovic R, Davis ME, et al. Endothelial cells promote cardiac myocyte survival and spatial reorganization: implications for cardiac regeneration. Circulation. 2004;110:962–8.
-
(2004)
Circulation
, vol.110
, pp. 962-968
-
-
Narmoneva, D.A.1
Vukmirovic, R.2
Davis, M.E.3
-
57
-
-
0037325194
-
Intracellular glutathione and lipid peroxide availability and the secretion of vasoactive substances by human umbilical vein endothelial cells after incubation with TNF-alpha
-
COI: 1:CAS:528:DC%2BD3sXit1ymu74%3D
-
Scalera F. Intracellular glutathione and lipid peroxide availability and the secretion of vasoactive substances by human umbilical vein endothelial cells after incubation with TNF-alpha. Eur J Clin Investig. 2003;33:176–82.
-
(2003)
Eur J Clin Investig
, vol.33
, pp. 176-182
-
-
Scalera, F.1
-
58
-
-
0036322768
-
Stretch-induced paracrine hypertrophic stimuli increase TGF-beta1 expression in cardiomyocytes
-
PID: 12190114
-
van Wamel AJ, Ruwhof C, van der Valk-Kokshoorn LJ, et al. Stretch-induced paracrine hypertrophic stimuli increase TGF-beta1 expression in cardiomyocytes. Mol Cell Biochem. 2002;236:147–53.
-
(2002)
Mol Cell Biochem
, vol.236
, pp. 147-153
-
-
van Wamel, A.J.1
Ruwhof, C.2
van der Valk-Kokshoorn, L.J.3
-
59
-
-
0035072921
-
The role of angiotensin II, endothelin-1 and transforming growth factor-beta as autocrine/paracrine mediators of stretch-induced cardiomyocyte hypertrophy
-
PID: 11330825
-
van Wamel AJ, Ruwhof C, van der Valk-Kokshoom LE, et al. The role of angiotensin II, endothelin-1 and transforming growth factor-beta as autocrine/paracrine mediators of stretch-induced cardiomyocyte hypertrophy. Mol Cell Biochem. 2001;218:113–24.
-
(2001)
Mol Cell Biochem
, vol.218
, pp. 113-124
-
-
van Wamel, A.J.1
Ruwhof, C.2
van der Valk-Kokshoom, L.E.3
-
61
-
-
0031930918
-
Fibroblast growth factor 2 control of vascular tone
-
PID: 9461194, COI: 1:CAS:528:DyaK1cXpvFegtQ%3D%3D
-
Zhou M, Sutliff RL, Paul RJ, et al. Fibroblast growth factor 2 control of vascular tone. Nat Med. 1998;4:201–7.
-
(1998)
Nat Med
, vol.4
, pp. 201-207
-
-
Zhou, M.1
Sutliff, R.L.2
Paul, R.J.3
-
62
-
-
14344279280
-
A cardiac myocyte vascular endothelial growth factor paracrine pathway is required to maintain cardiac function
-
PID: 11331753, COI: 1:CAS:528:DC%2BD3MXjs1Wntr4%3D
-
Giordano FJ, Gerber HP, Williams SP, et al. A cardiac myocyte vascular endothelial growth factor paracrine pathway is required to maintain cardiac function. Proc Natl Acad Sci U S A. 2001;98:5780–5.
-
(2001)
Proc Natl Acad Sci U S A
, vol.98
, pp. 5780-5785
-
-
Giordano, F.J.1
Gerber, H.P.2
Williams, S.P.3
-
63
-
-
39749137483
-
Sense and stretchability: the role of titin and titin-associated proteins in myocardial stress-sensing and mechanical dysfunction
-
PID: 17475230, COI: 1:CAS:528:DC%2BD1cXjvVaju7Y%3D
-
Linke WA. Sense and stretchability: the role of titin and titin-associated proteins in myocardial stress-sensing and mechanical dysfunction. Cardiovasc Res. 2008;77:637–48.
-
(2008)
Cardiovasc Res
, vol.77
, pp. 637-648
-
-
Linke, W.A.1
-
64
-
-
77952481131
-
Cardiac titin: a multifunctional giant
-
PID: 20479164
-
LeWinter MM, Granzier H. Cardiac titin: a multifunctional giant. Circulation. 2010;121:2137–45.
-
(2010)
Circulation
, vol.121
, pp. 2137-2145
-
-
LeWinter, M.M.1
Granzier, H.2
-
65
-
-
84892633453
-
Myocardial titin hypophosphorylation importantly contributes to heart failure with preserved ejection fraction in a rat metabolic risk model
-
PID: 24014826, COI: 1:CAS:528:DC%2BC3sXhvVahtbfL
-
Hamdani N, Franssen C, Lourenco A, et al. Myocardial titin hypophosphorylation importantly contributes to heart failure with preserved ejection fraction in a rat metabolic risk model. Circ Heart Fail. 2013;6:1239–49.
-
(2013)
Circ Heart Fail
, vol.6
, pp. 1239-1249
-
-
Hamdani, N.1
Franssen, C.2
Lourenco, A.3
-
66
-
-
68849089514
-
Modulation of titin-based stiffness by disulfide bonding in the cardiac titin N2-B unique sequence
-
PID: 19651040, COI: 1:CAS:528:DC%2BD1MXhsVChsrnJ
-
Grutzner A, Garcia-Manyes S, Kotter S, et al. Modulation of titin-based stiffness by disulfide bonding in the cardiac titin N2-B unique sequence. Biophys J. 2009;97:825–34.
-
(2009)
Biophys J
, vol.97
, pp. 825-834
-
-
Grutzner, A.1
Garcia-Manyes, S.2
Kotter, S.3
-
67
-
-
70349668973
-
PKC phosphorylation of titin’s PEVK element: a novel and conserved pathway for modulating myocardial stiffness
-
PID: 19679839, COI: 1:CAS:528:DC%2BD1MXhtFelsrzE, 17 p following 638
-
Hidalgo C, Hudson B, Bogomolovas J, et al. PKC phosphorylation of titin’s PEVK element: a novel and conserved pathway for modulating myocardial stiffness. Circ Res. 2009;105:631–8. 17 p following 638.
-
(2009)
Circ Res
, vol.105
, pp. 631-638
-
-
Hidalgo, C.1
Hudson, B.2
Bogomolovas, J.3
-
68
-
-
84874264782
-
Deranged myofilament phosphorylation and function in experimental heart failure with preserved ejection fraction
-
PID: 23213108, COI: 1:CAS:528:DC%2BC3sXis1Cqsrg%3D
-
Hamdani N, Bishu KG, von Frieling-Salewsky M, et al. Deranged myofilament phosphorylation and function in experimental heart failure with preserved ejection fraction. Cardiovasc Res. 2013;97:464–71.
-
(2013)
Cardiovasc Res
, vol.97
, pp. 464-471
-
-
Hamdani, N.1
Bishu, K.G.2
von Frieling-Salewsky, M.3
-
69
-
-
84865495526
-
A novel mechanism involving four-and-a-half LIM domain protein-1 and extracellular signal-regulated kinase-2 regulates titin phosphorylation and mechanics
-
PID: 22778266, COI: 1:CAS:528:DC%2BC38Xht1Cgtr7M
-
Raskin A, Lange S, Banares K, et al. A novel mechanism involving four-and-a-half LIM domain protein-1 and extracellular signal-regulated kinase-2 regulates titin phosphorylation and mechanics. J Biol Chem. 2012;287:29273–84.
-
(2012)
J Biol Chem
, vol.287
, pp. 29273-29284
-
-
Raskin, A.1
Lange, S.2
Banares, K.3
-
70
-
-
84874256463
-
Crucial role for Ca2(+)/calmodulin-dependent protein kinase-II in regulating diastolic stress of normal and failing hearts via titin phosphorylation
-
PID: 23283722, COI: 1:CAS:528:DC%2BC3sXisVOjt7Y%3D
-
Hamdani N, Krysiak J, Kreusser MM, et al. Crucial role for Ca2(+)/calmodulin-dependent protein kinase-II in regulating diastolic stress of normal and failing hearts via titin phosphorylation. Circ Res. 2013;112:664–74.
-
(2013)
Circ Res
, vol.112
, pp. 664-674
-
-
Hamdani, N.1
Krysiak, J.2
Kreusser, M.M.3
-
71
-
-
64249155200
-
Hypophosphorylation of the Stiff N2B titin isoform raises cardiomyocyte resting tension in failing human myocardium
-
PID: 19179657, COI: 1:CAS:528:DC%2BD1MXjt1Khtb0%3D
-
Borbely A, Falcao-Pires I, van Heerebeek L, et al. Hypophosphorylation of the Stiff N2B titin isoform raises cardiomyocyte resting tension in failing human myocardium. Circ Res. 2009;104:780–6.
-
(2009)
Circ Res
, vol.104
, pp. 780-786
-
-
Borbely, A.1
Falcao-Pires, I.2
van Heerebeek, L.3
-
72
-
-
15244348075
-
Phosphorylation of titin modulates passive stiffness of cardiac muscle in a titin isoform-dependent manner
-
PID: 15738048, COI: 1:CAS:528:DC%2BD2MXivVCmt7Y%3D
-
Fukuda N, Wu Y, Nair P, Granzier HL. Phosphorylation of titin modulates passive stiffness of cardiac muscle in a titin isoform-dependent manner. J Gen Physiol. 2005;125:257–71.
-
(2005)
J Gen Physiol
, vol.125
, pp. 257-271
-
-
Fukuda, N.1
Wu, Y.2
Nair, P.3
Granzier, H.L.4
-
73
-
-
59649084739
-
Protein kinase G modulates human myocardial passive stiffness by phosphorylation of the titin springs
-
PID: 19023132, COI: 1:CAS:528:DC%2BD1cXhsFSmt77J
-
Kruger M, Kotter S, Grutzner A, et al. Protein kinase G modulates human myocardial passive stiffness by phosphorylation of the titin springs. Circ Res. 2009;104:87–94.
-
(2009)
Circ Res
, vol.104
, pp. 87-94
-
-
Kruger, M.1
Kotter, S.2
Grutzner, A.3
-
74
-
-
84865209443
-
Low myocardial protein kinase G activity in heart failure with preserved ejection fraction
-
PID: 22806632, COI: 1:CAS:528:DC%2BC38Xht1ajt77E, This article demonstrated reduced myocardial cGMP concentration and protein kinase G activity in HFpEF compared to HFREF patients
-
van Heerebeek L, Hamdani N, Falcao-Pires I, et al. Low myocardial protein kinase G activity in heart failure with preserved ejection fraction. Circulation. 2012;126:830–9. This article demonstrated reduced myocardial cGMP concentration and protein kinase G activity in HFpEF compared to HFREF patients.
-
(2012)
Circulation
, vol.126
, pp. 830-839
-
-
van Heerebeek, L.1
Hamdani, N.2
Falcao-Pires, I.3
-
75
-
-
0038203256
-
Involvement of Ca2+/calmodulin-dependent protein kinase II in endothelial NO production and endothelium-dependent relaxation
-
PID: 12560211, COI: 1:CAS:528:DC%2BD3sXltVehsbo%3D
-
Schneider JC, El Kebir D, Chereau C, et al. Involvement of Ca2+/calmodulin-dependent protein kinase II in endothelial NO production and endothelium-dependent relaxation. Am J Physiol Heart Circ Physiol. 2003;284:H2311–9.
-
(2003)
Am J Physiol Heart Circ Physiol
, vol.284
, pp. H2311-H2319
-
-
Schneider, J.C.1
El Kebir, D.2
Chereau, C.3
-
76
-
-
26944446917
-
Impact of body mass and body composition on circulating levels of natriuretic peptides: results from the Dallas Heart Study
-
PID: 16203929, COI: 1:CAS:528:DC%2BD2MXhtVCltrfE
-
Das SR, Drazner MH, Dries DL, et al. Impact of body mass and body composition on circulating levels of natriuretic peptides: results from the Dallas Heart Study. Circulation. 2005;112:2163–8.
-
(2005)
Circulation
, vol.112
, pp. 2163-2168
-
-
Das, S.R.1
Drazner, M.H.2
Dries, D.L.3
-
77
-
-
0027354169
-
Cytokines increase neutral endopeptidase activity in lung fibroblasts
-
PID: 8380249, COI: 1:CAS:528:DyaK3sXhs1Ojur4%3D
-
Kondepudi A, Johnson A. Cytokines increase neutral endopeptidase activity in lung fibroblasts. Am J Respir Cell Mol Biol. 1993;8:43–9.
-
(1993)
Am J Respir Cell Mol Biol
, vol.8
, pp. 43-49
-
-
Kondepudi, A.1
Johnson, A.2
-
78
-
-
84920282718
-
Obesity and diastolic heart failure: is inflammation the link?
-
Carbone S, Shah KB, Van Tassell BW, et al. Obesity and diastolic heart failure: is inflammation the link? Transl Med. 2013;3:e124.
-
(2013)
Transl Med
, vol.3
, pp. e124
-
-
Carbone, S.1
Shah, K.B.2
Van Tassell, B.W.3
-
79
-
-
37349080234
-
Diastolic stiffness of the failing diabetic heart: importance of fibrosis, advanced glycation end products, and myocyte resting tension
-
PID: 18071071
-
van Heerebeek L, Hamdani N, Handoko ML, et al. Diastolic stiffness of the failing diabetic heart: importance of fibrosis, advanced glycation end products, and myocyte resting tension. Circulation. 2008;117:43–51.
-
(2008)
Circulation
, vol.117
, pp. 43-51
-
-
van Heerebeek, L.1
Hamdani, N.2
Handoko, M.L.3
-
80
-
-
26444478269
-
Advanced glycation end products and RAGE: a common thread in aging, diabetes, neurodegeneration, and inflammation
-
PID: 15764591, COI: 1:CAS:528:DC%2BD2MXlvFSrtrk%3D
-
Ramasamy R, Vannucci SJ, Yan SS, et al. Advanced glycation end products and RAGE: a common thread in aging, diabetes, neurodegeneration, and inflammation. Glycobiology. 2005;15:16R–28R.
-
(2005)
Glycobiology
, vol.15
, pp. 16R-28R
-
-
Ramasamy, R.1
Vannucci, S.J.2
Yan, S.S.3
-
81
-
-
84939600425
-
Oxidative stress predicts progression of peripheral and cardiac autonomic nerve dysfunction over 6 years in diabetic patients
-
Ziegler D, Buchholz S, Sohr C, et al. Oxidative stress predicts progression of peripheral and cardiac autonomic nerve dysfunction over 6 years in diabetic patients. Acta Diabetol. 2014. doi:10.1007/s00592-014-0601-3.
-
(2014)
Acta Diabetol
-
-
Ziegler, D.1
Buchholz, S.2
Sohr, C.3
-
82
-
-
33750300252
-
Mechanisms of disease: mitochondria as new therapeutic targets in diabetic neuropathy
-
PID: 17057749, COI: 1:CAS:528:DC%2BD28Xht1WqtrbI
-
Leinninger GM, Edwards JL, Lipshaw MJ, Feldman EL. Mechanisms of disease: mitochondria as new therapeutic targets in diabetic neuropathy. Nat Clin Pract Neurol. 2006;2:620–8.
-
(2006)
Nat Clin Pract Neurol
, vol.2
, pp. 620-628
-
-
Leinninger, G.M.1
Edwards, J.L.2
Lipshaw, M.J.3
Feldman, E.L.4
-
83
-
-
12944272082
-
Serum levels of substance P are decreased in patients with type 1 diabetes
-
PID: 10926310, COI: 1:CAS:528:DC%2BD3cXosVWisro%3D
-
Kunt T, Forst T, Schmidt S, et al. Serum levels of substance P are decreased in patients with type 1 diabetes. Exp Clin Endocrinol Diabetes. 2000;108:164–7.
-
(2000)
Exp Clin Endocrinol Diabetes
, vol.108
, pp. 164-167
-
-
Kunt, T.1
Forst, T.2
Schmidt, S.3
-
84
-
-
0033848381
-
Acute hyperglycaemia causes elevation in plasma atrial natriuretic peptide concentrations in type 1 diabetes mellitus
-
PID: 10972580, COI: 1:STN:280:DC%2BD3M%2FmsF2msQ%3D%3D
-
McKenna K, Smith D, Tormey W, Thompson CJ. Acute hyperglycaemia causes elevation in plasma atrial natriuretic peptide concentrations in type 1 diabetes mellitus. Diabet Med. 2000;17:512–7.
-
(2000)
Diabet Med
, vol.17
, pp. 512-517
-
-
McKenna, K.1
Smith, D.2
Tormey, W.3
Thompson, C.J.4
-
85
-
-
39149109157
-
Expression of neuropeptide Y and its receptors Y1 and Y2 in the rat heart and its supplying autonomic and spinal sensory ganglia in experimentally induced diabetes
-
PID: 18201831, COI: 1:CAS:528:DC%2BD1cXitVKgtr4%3D
-
Chottova Dvorakova M, Wiegand S, Pesta M, et al. Expression of neuropeptide Y and its receptors Y1 and Y2 in the rat heart and its supplying autonomic and spinal sensory ganglia in experimentally induced diabetes. Neuroscience. 2008;151:1016–28.
-
(2008)
Neuroscience
, vol.151
, pp. 1016-1028
-
-
Chottova Dvorakova, M.1
Wiegand, S.2
Pesta, M.3
-
86
-
-
77956330748
-
Global cardiovascular reserve dysfunction in heart failure with preserved ejection fraction
-
PID: 20813282
-
Borlaug BA, Olson TP, Lam CS, et al. Global cardiovascular reserve dysfunction in heart failure with preserved ejection fraction. J Am Coll Cardiol. 2010;56:845–54.
-
(2010)
J Am Coll Cardiol
, vol.56
, pp. 845-854
-
-
Borlaug, B.A.1
Olson, T.P.2
Lam, C.S.3
-
87
-
-
12144290445
-
B-type natriuretic peptide exerts broad functional opposition to transforming growth factor-beta in primary human cardiac fibroblasts: fibrosis, myofibroblast conversion, proliferation, and inflammation
-
PID: 14726474, COI: 1:CAS:528:DC%2BD2cXhsFOgurg%3D
-
Kapoun AM, Liang F, O’Young G, et al. B-type natriuretic peptide exerts broad functional opposition to transforming growth factor-beta in primary human cardiac fibroblasts: fibrosis, myofibroblast conversion, proliferation, and inflammation. Circ Res. 2004;94:453–61.
-
(2004)
Circ Res
, vol.94
, pp. 453-461
-
-
Kapoun, A.M.1
Liang, F.2
O’Young, G.3
-
88
-
-
0028318312
-
Ten years of natriuretic peptide research: a new dawn for their diagnostic and therapeutic use?
-
PID: 8025431, COI: 1:CAS:528:DyaK2cXlt1Ggtr4%3D
-
Struthers AD. Ten years of natriuretic peptide research: a new dawn for their diagnostic and therapeutic use? BMJ. 1994;308:1615–9.
-
(1994)
BMJ
, vol.308
, pp. 1615-1619
-
-
Struthers, A.D.1
-
89
-
-
2342443071
-
Obesity and suppressed B-type natriuretic peptide levels in heart failure
-
PID: 15120816, COI: 1:CAS:528:DC%2BD2cXjs1ylu7s%3D
-
Mehra MR, Uber PA, Park MH, et al. Obesity and suppressed B-type natriuretic peptide levels in heart failure. J Am Coll Cardiol. 2004;43:1590–5.
-
(2004)
J Am Coll Cardiol
, vol.43
, pp. 1590-1595
-
-
Mehra, M.R.1
Uber, P.A.2
Park, M.H.3
-
90
-
-
2942610467
-
Left ventricular dysfunction in normotensive type 1 diabetic patients: the impact of autonomic neuropathy
-
PID: 15154934, COI: 1:STN:280:DC%2BD2c3ls1yqsA%3D%3D
-
Taskiran M, Rasmussen V, Rasmussen B, et al. Left ventricular dysfunction in normotensive type 1 diabetic patients: the impact of autonomic neuropathy. Diabet Med. 2004;21:524–30.
-
(2004)
Diabet Med
, vol.21
, pp. 524-530
-
-
Taskiran, M.1
Rasmussen, V.2
Rasmussen, B.3
-
91
-
-
0037180778
-
The inflammatory reflex
-
PID: 12490958, COI: 1:CAS:528:DC%2BD38XpsFygtLc%3D
-
Tracey KJ. The inflammatory reflex. Nature. 2002;420:853–9.
-
(2002)
Nature
, vol.420
, pp. 853-859
-
-
Tracey, K.J.1
-
92
-
-
84867892494
-
Incremental prognostic significance of peripheral endothelial dysfunction in patients with heart failure with normal left ventricular ejection fraction
-
PID: 23040568, This article outlines the prognostic significance of peripheral endothelial dysfunction in patients with HFpEF
-
Akiyama E, Sugiyama S, Matsuzawa Y, et al. Incremental prognostic significance of peripheral endothelial dysfunction in patients with heart failure with normal left ventricular ejection fraction. J Am Coll Cardiol. 2012;60:1778–86. This article outlines the prognostic significance of peripheral endothelial dysfunction in patients with HFpEF.
-
(2012)
J Am Coll Cardiol
, vol.60
, pp. 1778-1786
-
-
Akiyama, E.1
Sugiyama, S.2
Matsuzawa, Y.3
-
93
-
-
43449083224
-
Prevention of heart failure: a scientific statement from the American Heart Association Councils on Epidemiology and Prevention, Clinical Cardiology, Cardiovascular Nursing, and High Blood Pressure Research; Quality of Care and Outcomes Research Interdisciplinary Working Group; and Functional Genomics and Translational Biology Interdisciplinary Working Group
-
PID: 18391114
-
Schocken DD, Benjamin EJ, Fonarow GC, et al. Prevention of heart failure: a scientific statement from the American Heart Association Councils on Epidemiology and Prevention, Clinical Cardiology, Cardiovascular Nursing, and High Blood Pressure Research; Quality of Care and Outcomes Research Interdisciplinary Working Group; and Functional Genomics and Translational Biology Interdisciplinary Working Group. Circulation. 2008;117:2544–65.
-
(2008)
Circulation
, vol.117
, pp. 2544-2565
-
-
Schocken, D.D.1
Benjamin, E.J.2
Fonarow, G.C.3
-
94
-
-
74949083441
-
Endomyocardial biopsy derived adherent proliferating cells—a potential cell source for cardiac tissue engineering
-
PID: 20013794, COI: 1:CAS:528:DC%2BC3cXhtVSntbg%3D
-
Haag M, Van Linthout S, Schroder SE, et al. Endomyocardial biopsy derived adherent proliferating cells—a potential cell source for cardiac tissue engineering. J Cell Biochem. 2010;109:564–75.
-
(2010)
J Cell Biochem
, vol.109
, pp. 564-575
-
-
Haag, M.1
Van Linthout, S.2
Schroder, S.E.3
-
95
-
-
38649105374
-
Mesenchymal stem cell-mediated immunosuppression occurs via concerted action of chemokines and nitric oxide
-
PID: 18371435, COI: 1:CAS:528:DC%2BD1cXitlygtb8%3D
-
Ren G, Zhang L, Zhao X, et al. Mesenchymal stem cell-mediated immunosuppression occurs via concerted action of chemokines and nitric oxide. Cell Stem Cell. 2008;2:141–50.
-
(2008)
Cell Stem Cell
, vol.2
, pp. 141-150
-
-
Ren, G.1
Zhang, L.2
Zhao, X.3
-
96
-
-
80052503522
-
Mesenchymal stem cells improve murine acute coxsackievirus B3-induced myocarditis
-
PID: 21183501, COI: 1:CAS:528:DC%2BC3MXhtFCisr7K
-
Van Linthout S, Savvatis K, Miteva K, et al. Mesenchymal stem cells improve murine acute coxsackievirus B3-induced myocarditis. Eur Heart J. 2011;32:2168–78.
-
(2011)
Eur Heart J
, vol.32
, pp. 2168-2178
-
-
Van Linthout, S.1
Savvatis, K.2
Miteva, K.3
-
97
-
-
84877117402
-
Immune attributes of cardiac-derived adherent proliferating (CAP) cells in cardiac therapy
-
PID: 22389069, COI: 1:CAS:528:DC%2BC3sXntFyhu7s%3D
-
Haag M, Stolk M, Ringe J, et al. Immune attributes of cardiac-derived adherent proliferating (CAP) cells in cardiac therapy. J Tissue Eng Regen Med. 2013;7:362–70.
-
(2013)
J Tissue Eng Regen Med
, vol.7
, pp. 362-370
-
-
Haag, M.1
Stolk, M.2
Ringe, J.3
-
98
-
-
67549126317
-
The role of placental-derived adherent stromal cell (PLX-PAD) in the treatment of critical limb ischemia
-
PID: 19526389, COI: 1:CAS:528:DC%2BD1MXosVanu7o%3D
-
Prather WR, Toren A, Meiron M, et al. The role of placental-derived adherent stromal cell (PLX-PAD) in the treatment of critical limb ischemia. Cytotherapy. 2009;11:427–34.
-
(2009)
Cytotherapy
, vol.11
, pp. 427-434
-
-
Prather, W.R.1
Toren, A.2
Meiron, M.3
-
99
-
-
72849145234
-
Mesenchymal stem cells promote matrix metalloproteinase secretion by cardiac fibroblasts and reduce cardiac ventricular fibrosis after myocardial infarction
-
PID: 19591227, COI: 1:CAS:528:DC%2BC3cXlsFyluw%3D%3D
-
Mias C, Lairez O, Trouche E, et al. Mesenchymal stem cells promote matrix metalloproteinase secretion by cardiac fibroblasts and reduce cardiac ventricular fibrosis after myocardial infarction. Stem Cells. 2009;27:2734–43.
-
(2009)
Stem Cells
, vol.27
, pp. 2734-2743
-
-
Mias, C.1
Lairez, O.2
Trouche, E.3
-
100
-
-
83055164357
-
Human cardiac-derived adherent proliferating cells reduce murine acute coxsackievirus B3-induced myocarditis
-
PID: 22174827, COI: 1:CAS:528:DC%2BC38Xht1Sl
-
Miteva K, Haag M, Peng J, et al. Human cardiac-derived adherent proliferating cells reduce murine acute coxsackievirus B3-induced myocarditis. PLoS One. 2011;6:e28513.
-
(2011)
PLoS One
, vol.6
, pp. e28513
-
-
Miteva, K.1
Haag, M.2
Peng, J.3
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