Degradation of misfolded proteins by autophagy: Is it a strategy for huntington’s disease treatment?

Journal of Huntington's Disease

Volumn 2, Issue 2, 2013, Pages 149-157

  Lin, Fang ^a   Qin, Zheng Hong ^a  

^a MEDICAL COLLEGE OF SOOCHOW UNIVERSITY   (China)

Author keywords

Autophagy; Huntingtin; Huntington s disease; Polyglutamine expansion

Indexed keywords

CARBAMAZEPINE; DRUG DERIVATIVE; HTT PROTEIN, HUMAN; HUNTINGTIN; LITHIUM; MUTANT PROTEIN; NERVE PROTEIN; OXAZOLE DERIVATIVE; PEPTIDE; POLYGLUTAMINE; RAPAMYCIN; RILMENIDINE; TEMSIROLIMUS; TREHALOSE; VALPROIC ACID;

AUTOPHAGY; GENETICS; HUMAN; HUNTINGTON CHOREA; METABOLISM; PHYSIOLOGY; REVIEW; TRINUCLEOTIDE REPEAT;

AUTOPHAGY; HUNTINGTIN; HUNTINGTON'S DISEASE; POLYGLUTAMINE EXPANSION;

AUTOPHAGY; CARBAMAZEPINE; HUMANS; HUNTINGTON DISEASE; LITHIUM; MUTANT PROTEINS; NERVE TISSUE PROTEINS; OXAZOLES; PEPTIDES; SIROLIMUS; TREHALOSE; TRINUCLEOTIDE REPEAT EXPANSION; VALPROIC ACID;

EID: 84908025737     PISSN: 18796397     EISSN: 18796400     Source Type: Journal    
DOI: 10.3233/JHD-130052     Document Type: Article

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