Abnormal interaction of VDAC1 with amyloid beta and phosphorylated tau causes mitochondrial dysfunction in Alzheimer's disease

Human Molecular Genetics

Volumn 21, Issue 23, 2012, Pages 5131-5146

  Manczak, Maria ^a   Reddy, P Hemachandra ^a,b  

^a OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

^b OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; MUTANT PROTEIN; TAU PROTEIN; VOLTAGE DEPENDENT ANION CHANNEL 1;

ALZHEIMER DISEASE; ARTICLE; BRAIN; CONTROLLED STUDY; FRONTAL CORTEX; HUMAN; HUMAN TISSUE; IMMUNOBLOTTING; MOUSE; NEUROLOGIC DISEASE; NONHUMAN; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION;

AGE FACTORS; ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; BRAIN; DISEASE MODELS, ANIMAL; HUMANS; MICE; MICE, TRANSGENIC; MITOCHONDRIA; PHOSPHORYLATION; PROTEIN BINDING; TAU PROTEINS; VOLTAGE-DEPENDENT ANION CHANNEL 1;

MUS; MUS MUSCULUS;

EID: 84869012777     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/dds360     Document Type: Article

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