Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: Implications for mitochondrial dysfunction and neuronal damage

Human Molecular Genetics

Volumn 21, Issue 11, 2012, Pages 2538-2547

  Manczak, Maria ^a   Reddy, P Hemachandra ^a,b  

^a OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

^b OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; GUANOSINE TRIPHOSPHATASE; MITOCHONDRIAL FISSION PROTEIN DRP1; MITOCHONDRIAL PROTEIN; TAU PROTEIN; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; COGNITIVE DEFECT; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; FRONTAL CORTEX; HUMAN; HUMAN TISSUE; MOUSE; NERVE CELL LESION; NONHUMAN; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; BINDING SITES; DISEASE MODELS, ANIMAL; FLUORESCENT ANTIBODY TECHNIQUE; GTP PHOSPHOHYDROLASES; HUMANS; IMMUNOPRECIPITATION; MICE; MICE, TRANSGENIC; MICROTUBULE-ASSOCIATED PROTEINS; MITOCHONDRIA; MITOCHONDRIAL PROTEINS; NEURONS; PHOSPHORYLATION; TAU PROTEINS;

MUS;

EID: 84861130196     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/dds072     Document Type: Article

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