Animal toxins can alter the function of Nav1.8 and Nav1.9

Toxins

Volumn 4, Issue 8, 2012, Pages 620-632

  Gilchrist, John ^a   bosmans, Frank ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Animal toxins; Nav1.8; Nav1.9; Pain; Voltage sensor; Voltage activated sodium channel

Indexed keywords

ANIMAL TOXIN; BATRACHOTOXIN; CONOTOXIN; SAXITOXIN; SODIUM CHANNEL NAV1.8; SODIUM CHANNEL NAV1.9; SPIDER VENOM; TETRODOTOXIN; TOXIN; UNCLASSIFIED DRUG;

ALLODYNIA; AMINO ACID SEQUENCE; CHANNEL GATING; DRUG DESIGN; DRUG RESEARCH; ELECTRIC CONDUCTIVITY; ELECTROPHYSIOLOGY; HUMAN; HYPERALGESIA; INFLAMMATION; INFLAMMATORY PAIN; NEUROMODULATION; NEUROPATHIC PAIN; NOCICEPTION; NONHUMAN; PAIN THRESHOLD; PROTEIN DETERMINATION; PROTEIN DOMAIN; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN MODIFICATION; PROTEIN MOTIF; PROTEIN STRUCTURE; PROTEIN TARGETING; REVIEW; SENSORY NERVE CELL; SIGNAL TRANSDUCTION; SODIUM CURRENT; SPINAL GANGLION;

EID: 84865402118     PISSN: 20726651     EISSN: None     Source Type: Journal    
DOI: 10.3390/toxins4080620     Document Type: Review

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