A role for the TTX-resistant sodium channel Nav 1.8 in NGF-induced hyperalgesia, but not neuropathic pain

NeuroReport

Volumn 12, Issue 14, 2001, Pages 3077-3080

  Kerr, Bradley J ^a   Souslova, Veronika ^b   McMahon, Stephen B ^a   Wood, John N ^b  

^a KING'S COLLEGE LONDON   (United Kingdom)

^b UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

Inflammation; Nerve growth factor; Neuropathic pain; Sodium channels

Indexed keywords

GENE PRODUCT; NERVE GROWTH FACTOR; PROSTAGLANDIN E2; PROTEIN NAV1.8; SODIUM CHANNEL; SODIUM ION; TETRODOTOXIN; UNCLASSIFIED DRUG;

ANIMAL BEHAVIOR; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; FEMALE; GENE MUTATION; HYPERALGESIA; INFLAMMATION; MALE; MOUSE; NERVE CELL LESION; NEUROPATHY; NOCICEPTION; NONHUMAN; PAIN THRESHOLD; PERIPHERAL NERVE INJURY; PRIORITY JOURNAL; SENSITIZATION; SENSORY NERVE CELL; VOLTAGE CLAMP;

EID: 0035829033     PISSN: 09594965     EISSN: None     Source Type: Journal    
DOI: 10.1097/00001756-200110080-00019     Document Type: Article

References (26)