Exposure to IL-15 and IL-21 enables autoreactive CD8 T cells to respond to weak antigens and cause disease in a mouse model of autoimmune diabetes

Journal of Immunology

Volumn 186, Issue 9, 2011, Pages 5131-5141

  Ramanathan, Sheela ^a,b   Dubois, Stephanie ^a,b   Chen, Xi Lin ^a,b   Leblanc, Chantal ^a,b   Ohashi, Pamela S ^c   Ilangumaran, Subburaj ^a,b  

^a UNIVERSITY OF SHERBROOKE   (Canada)

^b Centre Hospitalier Universitaire de Sherbrooke   (Canada)

^c PRINCESS MARGARET HOSPITAL   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIGEN; HERMES ANTIGEN; INTERLEUKIN 15; INTERLEUKIN 21; INTERLEUKIN 7; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1; T LYMPHOCYTE RECEPTOR;

ADOPTIVE TRANSFER; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANTIGEN SPECIFICITY; ARTICLE; AUTOIMMUNE DISEASE; CD8+ T LYMPHOCYTE; CELL ISOLATION; CELL SURFACE; CONTROLLED STUDY; CROSS PRESENTATION; CYTOKINE RELEASE; CYTOLYSIS; DIABETES MELLITUS; DIABETOGENESIS; DISSOCIATION; EFFECTOR CELL; HISTOPATHOLOGY; IN VIVO STUDY; LYMPHOCYTE PROLIFERATION; MOUSE; NONHUMAN; PRIORITY JOURNAL;

EID: 79955529069     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1001221     Document Type: Article

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