ATR preferentially interacts with common fragile site FRA3B and the binding requires its kinase activity in response to aphidicolin treatment

Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis

Volumn 686, Issue 1-2, 2010, Pages 39-46

  Wan, Cheng ^a   Kulkarni, Atul ^b   Wang, Yuh Hwa ^a  

^a WAKE FOREST SCHOOL OF MEDICINE   (United States)

^b ROBERT WOOD JOHNSON MEDICAL SCHOOL   (United States)

Author keywords

ATR; FRA3B; Fragile site

Indexed keywords

2 AMINOPURINE; APHIDICOLIN; ATR PROTEIN; DNA; PROTEIN KINASE;

ARTICLE; CHROMATIN IMMUNOPRECIPITATION; CHROMOSOME FRAGILE SITE; COMMON FRAGILE SITE; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME INHIBITION; GENETIC TRANSFECTION; HELA CELL; HUMAN; HUMAN CELL; HUMAN CELL CULTURE; NUCLEOTIDE SEQUENCE; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROTEIN BINDING; STRESS; WESTERN BLOTTING;

ACID ANHYDRIDE HYDROLASES; APHIDICOLIN; CELL CYCLE PROTEINS; DNA DAMAGE; DNA REPLICATION; ENZYME INHIBITORS; GENOMIC INSTABILITY; HUMANS; NEOPLASM PROTEINS; PROTEIN KINASES; PROTEIN-SERINE-THREONINE KINASES;

EID: 77649183106     PISSN: 00275107     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mrfmmm.2009.12.012     Document Type: Article

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