Genomic amplification of MET with boundaries within fragile site FRA7G and upregulation of MET pathways in esophageal adenocarcinoma

Oncogene

Volumn 25, Issue 3, 2006, Pages 409-418

  Miller, C T ^a   Lin, L ^a   Casper, A M ^a   Lim, J ^a   Thomas, D G ^a   Orringer, M B ^a   Chang, A C ^a   Chambers, A F ^b   Giordano, T J ^a   Glover, T W ^a   Beer, D G ^a  

^a UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

^b LONDON REGIONAL CANCER PROGRAM   (Canada)

Author keywords

Esophageal adenocarcinoma; FRA7G; Gene amplification; MET oncogene

Indexed keywords

GELDANAMYCIN; HERMES ANTIGEN; MATRIX METALLOPROTEINASE; OSTEOPONTIN; PHOSPHOPROTEIN; PROTEIN ANTIBODY; PROTEIN TYROSINE KINASE INHIBITOR; SCATTER FACTOR; SCATTER FACTOR RECEPTOR;

AMPLICON; APOPTOSIS; ARTICLE; AUTOPHOSPHORYLATION; BARRETT ESOPHAGUS; CANCER GROWTH; CANCER INVASION; CANCER SURVIVAL; CELL ACTIVITY; CELL MIGRATION; CHROMOSOME 11P; CONTROLLED STUDY; DYSPLASIA; ESOPHAGEAL ADENOCARCINOMA; GENE ACTIVATION; GENE AMPLIFICATION; GENE EXPRESSION; GENE MAPPING; GENE OVEREXPRESSION; GENOME; HUMAN; HUMAN CELL; LYMPH NODE METASTASIS; METAPLASIA; MICROARRAY ANALYSIS; MUTATION; ONCOGENE; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION; TISSUE MICROARRAY; UPREGULATION;

EID: 30944452172     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/sj.onc.1209057     Document Type: Article

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